Biblio
“Expression of the Alzheimer's Disease Mutations AβPP695sw and PSEN1M146I in Double-Transgenic Göttingen Minipigs.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1617-30, 2016.
, “Expression Pattern of Scavenger Receptors and Amyloid-β Phagocytosis of Astrocytes and Microglia in Culture are Modified by Acidosis: Implications for Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 3, pp. 857-73, 2016.
, “Extracellular Tau Paired Helical Filaments Differentially Affect Tau Pathogenic Mechanisms in Mitotic and Post-Mitotic Cells: Implications for Mechanisms of Tau Propagation in the Brain.”, J Alzheimers Dis, vol. 54, no. 2, pp. 477-96, 2016.
, “Extracellular Vesicles Containing P301L Mutant Tau Accelerate Pathological Tau Phosphorylation and Oligomer Formation but Do Not Seed Mature Neurofibrillary Tangles in ALZ17 Mice.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1207-1217, 2016.
, “Familial Presenilin Mutations and Sporadic Alzheimer's Disease Pathology: Is the Assumption of Biochemical Equivalence Justified?”, J Alzheimers Dis, vol. 50, no. 3, pp. 645-58, 2016.
, “FDG-PET and Neuropsychiatric Symptoms among Cognitively Normal Elderly Persons: The Mayo Clinic Study of Aging.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1609-16, 2016.
, “Feasibility of a Memory Clinic-Based Physical Activity Prescription Program.”, J Alzheimers Dis, vol. 53, no. 1, pp. 161-70, 2016.
, “Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse.”, J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
, “Fine Particulate Matter, Residential Proximity to Major Roads, and Markers of Small Vessel Disease in a Memory Study Population.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1315-23, 2016.
, “First Symptoms and Neurocognitive Correlates of Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 54, no. 3, pp. 957-970, 2016.
, “Fluoxetine Treatment Induces Seizure Behavior and Premature Death in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 51, no. 3, pp. 677-82, 2016.
, “The Folate-Vitamin B12 Interaction, Low Hemoglobin, and the Mortality Risk from Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 2, pp. 705-12, 2016.
, “Formal Psychiatric Disorders are not Overrepresented in Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1249-56, 2016.
, “Formulaic Language in People with Probable Alzheimer's Disease: A Frequency-Based Approach.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1145-60, 2016.
, “FOXP2 Expression in Frontotemporal Lobar Degeneration-Tau.”, J Alzheimers Dis, vol. 54, no. 2, pp. 471-5, 2016.
, “FTIR and Raman Spectroscopy Applied to Dementia Diagnosis Through Analysis of Biological Fluids.”, J Alzheimers Dis, vol. 52, no. 3, pp. 801-12, 2016.
, “Functional Connectivity of Ventral and Dorsal Visual Streams in Posterior Cortical Atrophy.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1119-30, 2016.
, “Future Dementia Severity is Almost Entirely Explained by the Latent Variable δ's Intercept and Slope.”, J Alzheimers Dis, vol. 49, no. 2, pp. 521-9, 2016.
, “Gene Expression of Quaking in Sporadic Alzheimer's Disease Patients is Both Upregulated and Related to Expression Levels of Genes Involved in Amyloid Plaque and Neurofibrillary Tangle Formation.”, J Alzheimers Dis, vol. 53, no. 1, pp. 209-19, 2016.
, “Gene Expression Profiling in the APP/PS1KI Mouse Model of Familial Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 397-409, 2016.
, “Genetic Counseling and Testing for Alzheimer's Disease and Frontotemporal Lobar Degeneration: An Italian Consensus Protocol.”, J Alzheimers Dis, vol. 51, no. 1, pp. 277-91, 2016.
, “Genetic Influences on Plasma Homocysteine Levels in African Americans and Yoruba Nigerians.”, J Alzheimers Dis, vol. 49, no. 4, pp. 991-1003, 2016.
, “The Genetic Variability of UCP4 Affects the Individual Susceptibility to Late-Onset Alzheimer's Disease and Modifies the Disease's Risk in APOE-ɛ4 Carriers.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1265-74, 2016.
, “A Genetic Variant of the Sortilin 1 Gene is Associated with Reduced Risk of Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1353-63, 2016.
, “Genetic Variations in ABCA7 Can Increase Secreted Levels of Amyloid-β40 and Amyloid-β42 Peptides and ABCA7 Transcription in Cell Culture Models.”, J Alzheimers Dis, vol. 53, no. 3, pp. 875-92, 2016.
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