Biblio
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“Different Clinical and Neuroimaging Characteristics in Early Stage Parkinson's Disease with Dementia and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 52, no. 1, pp. 205-11, 2016.
, “Differential Membrane Toxicity of Amyloid-β Fragments by Pore Forming Mechanisms.”, J Alzheimers Dis, vol. 51, no. 3, pp. 689-99, 2016.
, “Differential Regulation of N-Methyl-D-Aspartate Receptor Subunits is an Early Event in the Actions of Soluble Amyloid-β(1-40) Oligomers on Hippocampal Neurons.”, J Alzheimers Dis, vol. 51, no. 1, pp. 197-212, 2016.
, “Differential Risk of Incident Alzheimer's Disease Dementia in Stable Versus Unstable Patterns of Subjective Cognitive Decline.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1135-1146, 2016.
, “Direct Evidence of Internalization of Tau by Microglia In Vitro and In Vivo.”, J Alzheimers Dis, vol. 50, no. 1, pp. 77-87, 2016.
, “The Disconnection Hypothesis in Alzheimer's Disease Studied Through Multimodal Magnetic Resonance Imaging: Structural, Perfusion, and Diffusion Tensor Imaging.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1051-64, 2016.
, “Discovery and Subsequent Confirmation of Novel Serum Biomarkers Diagnosing Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 2, pp. 317-27, 2016.
, “Discovery and Subsequent Confirmation of Novel Serum Biomarkers Diagnosing Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 2, pp. 317-27, 2016.
, “Disrupted Brain Network in Progressive Mild Cognitive Impairment Measured by Eigenvector Centrality Mapping is Linked to Cognition and Cerebrospinal Fluid Biomarkers.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1483-1493, 2016.
, “Disruption of White Matter Integrity by Chronic Cerebral Hypoperfusion in Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1311-9, 2016.
, “Diversity of Cognitive Phenotypes Associated with C9ORF72 Hexanucleotide Expansion.”, J Alzheimers Dis, vol. 52, no. 1, pp. 25-31, 2016.
, “Does Microglial Activation Influence Hippocampal Volume and Neuronal Function in Alzheimer's Disease and Parkinson's Disease Dementia?”, J Alzheimers Dis, vol. 51, no. 4, pp. 1275-89, 2016.
, “Drivers: A Biologically Contextualized, Cross-Inferential View of the Epidemiology of Neurodegenerative Disorders.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1003-22, 2016.
, “Drivers: A Biologically Contextualized, Cross-Inferential View of the Epidemiology of Neurodegenerative Disorders.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1003-22, 2016.
, “Drivers: A Biologically Contextualized, Cross-Inferential View of the Epidemiology of Neurodegenerative Disorders.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1003-22, 2016.
, “Dual Kinase Inhibition Affords Extended in vitro Neuroprotection in Amyloid-β Toxicity.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1659-1670, 2016.
, “EEG Markers of Dementia with Lewy Bodies: A Multicenter Cohort Study.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1649-1657, 2016.
, “Effect of Tween-20 on Core Biomarkers Measured in Cerebrospinal Fluid from Patients with Alzheimer's Disease, Mild Cognitive Impairment, or Healthy Control Individuals.”, J Alzheimers Dis, vol. 49, no. 2, pp. 493-502, 2016.
, “Effectiveness of Supporting Informal Caregivers of People with Dementia: A Systematic Review of Randomized and Non-Randomized Controlled Trials.”, J Alzheimers Dis, vol. 52, no. 3, pp. 929-65, 2016.
, “Effects of Hypertension and Anti-Hypertensive Treatment on Amyloid-β (Aβ) Plaque Load and Aβ-Synthesizing and Aβ-Degrading Enzymes in Frontal Cortex.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1191-203, 2016.
, “Efficacy and Safety of Crocus sativus L. in Patients with Mild Cognitive Impairment: One Year Single-Blind Randomized, with Parallel Groups, Clinical Trial.”, J Alzheimers Dis, vol. 54, no. 1, pp. 129-33, 2016.
, “Efficacy and Safety of Sustained Release Donepezil High Dose versus Immediate Release Donepezil Standard Dose in Japanese Patients with Severe Alzheimer's Disease: A Randomized, Double-Blind Trial.”, J Alzheimers Dis, vol. 52, no. 1, pp. 345-57, 2016.
, “Enalapril Alone or Co-Administered with Losartan Rescues Cerebrovascular Dysfunction, but not Mnemonic Deficits or Amyloidosis in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1183-95, 2016.
, “Evaluation of a Genetic Risk Score to Improve Risk Prediction for Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 3, pp. 921-32, 2016.
, “Evaluation of a Genetic Risk Score to Improve Risk Prediction for Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 3, pp. 921-32, 2016.
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