Biblio
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“Insulin Resistance is Associated with Increased Levels of Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Reduced Memory Function in At-Risk Healthy Middle-Aged Adults.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1373-83, 2016.
, “Interaction Between Midlife Blood Glucose and APOE Genotype Predicts Later Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1553-62, 2016.
, “Iron Regulates Apolipoprotein E Expression and Secretion in Neurons and Astrocytes.”, J Alzheimers Dis, vol. 51, no. 2, pp. 471-87, 2016.
, “Lack of evidence for a role of HHV-6 in the pathogenesis of Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 229-35, 2016.
, “Late-Life Depressive Symptoms and Lifetime History of Major Depression: Cognitive Deficits are Largely Due to Incipient Dementia rather than Depression.”, J Alzheimers Dis, vol. 54, no. 1, pp. 185-99, 2016.
, “Less Daily Computer Use is Related to Smaller Hippocampal Volumes in Cognitively Intact Elderly.”, J Alzheimers Dis, vol. 52, no. 2, pp. 713-7, 2016.
, “The Level of NMDA Receptor in the Membrane Modulates Amyloid-β Association and Perforation.”, J Alzheimers Dis, vol. 53, no. 1, pp. 197-207, 2016.
, “Lifelong Reading Disorder and Mild Cognitive Impairment: Implications for Diagnosis.”, J Alzheimers Dis, vol. 50, no. 1, pp. 41-5, 2016.
, “Lifelong Reading Disorder and Mild Cognitive Impairment: Implications for Diagnosis.”, J Alzheimers Dis, vol. 50, no. 1, pp. 41-5, 2016.
, “Longitudinal Alteration of Intrinsic Brain Activity in the Striatum in Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 54, no. 1, pp. 69-78, 2016.
, “Longitudinal Study of Impaired Intra- and Inter-Network Brain Connectivity in Subjects at High Risk for Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 3, pp. 913-27, 2016.
, “Longitudinal Study of Impaired Intra- and Inter-Network Brain Connectivity in Subjects at High Risk for Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 3, pp. 913-27, 2016.
, “Looking for Neuroimaging Markers in Frontotemporal Lobar Degeneration Clinical Trials: A Multi-Voxel Pattern Analysis Study in Granulin Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 249-62, 2016.
, “Looking for Neuroimaging Markers in Frontotemporal Lobar Degeneration Clinical Trials: A Multi-Voxel Pattern Analysis Study in Granulin Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 249-62, 2016.
, “Lower Late-Life Body-Mass Index is Associated with Higher Cortical Amyloid Burden in Clinically Normal Elderly.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1097-105, 2016.
, “MAPT H1 Haplotype is Associated with Late-Onset Alzheimer's Disease Risk in APOEɛ4 Noncarriers: Results from the Dementia Genetics Spanish Consortium.”, J Alzheimers Dis, vol. 49, no. 2, pp. 343-52, 2016.
, “Memory Impairment at Initial Clinical Presentation in Posterior Cortical Atrophy.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1245-50, 2016.
, “Meta-Analysis of Transcriptome Data Related to Hippocampus Biopsies and iPSC-Derived Neuronal Cells from Alzheimer's Disease Patients Reveals an Association with FOXA1 and FOXA2 Gene Regulatory Networks.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1065-82, 2016.
, “Metallothioneins in Prion- and Amyloid-Related Diseases.”, J Alzheimers Dis, vol. 51, no. 3, pp. 637-56, 2016.
, “Metallothioneins in Prion- and Amyloid-Related Diseases.”, J Alzheimers Dis, vol. 51, no. 3, pp. 637-56, 2016.
, “"Missed" Mild Cognitive Impairment: High False-Negative Error Rate Based on Conventional Diagnostic Criteria.”, J Alzheimers Dis, vol. 52, no. 2, pp. 685-91, 2016.
, “Mitochondrial Regulatory Pathways in the Pathogenesis of Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1257-70, 2016.
, “Mitochondrial Regulatory Pathways in the Pathogenesis of Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1257-70, 2016.
, “A Mitochondrial Role of SV2a Protein in Aging and Alzheimer's Disease: Studies with Levetiracetam.”, J Alzheimers Dis, vol. 50, no. 1, pp. 201-15, 2016.
, “Moderate-to-High Intensity Physical Exercise in Patients with Alzheimer's Disease: A Randomized Controlled Trial.”, J Alzheimers Dis, vol. 50, no. 2, pp. 443-53, 2016.
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