Biblio
Export 1991 results:
Author Title Type [ Year
Filters: First Letter Of Last Name is B [Clear All Filters]
“Cognitive Event-Related Potential, an Early Diagnosis Biomarker in Frail Elderly Subjects: The ERP-MAPT-PLUS Ancillary Study.”, J Alzheimers Dis, vol. 58, no. 1, pp. 87-97, 2017.
, “Cognitive Rehabilitation in Alzheimer's Disease: A Controlled Intervention Trial.”, J Alzheimers Dis, vol. 57, no. 4, pp. 1315-1324, 2017.
, “Cognitive Status, Gray Matter Atrophy, and Lower Orthostatic Blood Pressure in Older Adults.”, J Alzheimers Dis, vol. 57, no. 4, pp. 1239-1250, 2017.
, “Cognitive-Enhancing Effects of a Polyphenols-Rich Extract from Fruits without Changes in Neuropathology in an Animal Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 1, pp. 115-135, 2017.
, “Cognitive-Enhancing Effects of a Polyphenols-Rich Extract from Fruits without Changes in Neuropathology in an Animal Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 1, pp. 115-135, 2017.
, “Combinatorial Treatment Effects in a Cell Culture Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1155-1166, 2017.
, “Combinatorial Treatment Effects in a Cell Culture Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1155-1166, 2017.
, “Combined Creutzfeldt-Jakob/ Alzheimer's Disease Cases are Important in Search for Microbes in Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 3, pp. 867-873, 2017.
, “A Common Variant in the MC1R Gene (p.V92M) is associated with Alzheimer's Disease Risk.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1065-1074, 2017.
, “A Common Variant of IL-6R is Associated with Elevated IL-6 Pathway Activity in Alzheimer's Disease Brains.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1037-1054, 2017.
, “A Common Variant of IL-6R is Associated with Elevated IL-6 Pathway Activity in Alzheimer's Disease Brains.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1037-1054, 2017.
, “Comparison of Gait Parameters for Predicting Cognitive Decline: The Mayo Clinic Study of Aging.”, J Alzheimers Dis, vol. 55, no. 2, pp. 559-567, 2017.
, “Comprehensive Quantitative Profiling of Tau and Phosphorylated Tau Peptides in Cerebrospinal Fluid by Mass Spectrometry Provides New Biomarker Candidates.”, J Alzheimers Dis, vol. 55, no. 1, pp. 303-313, 2017.
, “Cortical Cerebral Microinfarcts on 3 Tesla MRI in Patients with Vascular Cognitive Impairment.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1443-1450, 2017.
, “Cortical Iron Reflects Severity of Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1533-1545, 2017.
, “Cross-Sectional and Longitudinal Cognitive Correlates of FDDNP PET and CSF Amyloid-β and Tau in Parkinson's Disease1.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1261-1272, 2017.
, “Cross-Sectional and Longitudinal Cognitive Correlates of FDDNP PET and CSF Amyloid-β and Tau in Parkinson's Disease1.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1261-1272, 2017.
, “Cross-Sectional and Longitudinal Cognitive Correlates of FDDNP PET and CSF Amyloid-β and Tau in Parkinson's Disease1.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1261-1272, 2017.
, “A Cross-Validation of FDG- and Amyloid-PET Biomarkers in Mild Cognitive Impairment for the Risk Prediction to Dementia due to Alzheimer's Disease in a Clinical Setting.”, J Alzheimers Dis, vol. 59, no. 2, pp. 603-614, 2017.
, “Curcumin Ameliorates Neuroinflammation, Neurodegeneration, and Memory Deficits in p25 Transgenic Mouse Model that Bears Hallmarks of Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1429-1442, 2017.
, “Curcumin Inhibits Tau Aggregation and Disintegrates Preformed Tau Filaments in vitro.”, J Alzheimers Dis, vol. 60, no. 3, pp. 999-1014, 2017.
, “Current Approaches and Clinician Attitudes to the Use of Cerebrospinal Fluid Biomarkers in Diagnostic Evaluation of Dementia in Europe.”, J Alzheimers Dis, vol. 60, no. 1, pp. 201-210, 2017.
, “Current Approaches and Clinician Attitudes to the Use of Cerebrospinal Fluid Biomarkers in Diagnostic Evaluation of Dementia in Europe.”, J Alzheimers Dis, vol. 60, no. 1, pp. 201-210, 2017.
, “Damage to the Frontal Aslant Tract Accounts for Visuo-Constructive Deficits in Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 3, pp. 1015-1024, 2017.
, “Decreased Glucose Metabolism in Medial Prefrontal Areas is Associated with Nutritional Status in Patients with Prodromal and Early Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 1, pp. 225-233, 2017.
,