Biblio
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“Cerebral Amyloid Angiopathy-Related Microbleeds and Cerebrospinal Fluid Biomarkers in Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 3, pp. 905-913, 2017.
, “Cerebral Amyloid Angiopathy-Related Microbleeds and Cerebrospinal Fluid Biomarkers in Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 3, pp. 905-913, 2017.
, “The Cerebrospinal Fluid Aβ1-42/Aβ1-40 Ratio Improves Concordance with Amyloid-PET for Diagnosing Alzheimer's Disease in a Clinical Setting.”, J Alzheimers Dis, vol. 60, no. 2, pp. 561-576, 2017.
, “Cerebrospinal Fluid Neurogranin as a Biomarker of Neurodegenerative Diseases: A Cross-Sectional Study.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1327-1334, 2017.
, “Challenges and Considerations Related to Studying Dementia in Blacks/African Americans.”, J Alzheimers Dis, vol. 60, no. 1, pp. 1-10, 2017.
, “Changes of Cerebrospinal Fluid Peptides due to Tauopathy.”, J Alzheimers Dis, vol. 58, no. 2, pp. 507-520, 2017.
, “Chronic Lithium Treatment in a Rat Model of Basal Forebrain Cholinergic Depletion: Effects on Memory Impairment and Neurodegeneration.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1505-1518, 2017.
, “Classification of Depression, Cognitive Disorders, and Co-Morbid Depression and Cognitive Disorders with Perfusion SPECT Neuroimaging.”, J Alzheimers Dis, vol. 57, no. 1, pp. 253-266, 2017.
, “Cognitive Status, Gray Matter Atrophy, and Lower Orthostatic Blood Pressure in Older Adults.”, J Alzheimers Dis, vol. 57, no. 4, pp. 1239-1250, 2017.
, “Cognitive-Enhancing Effects of a Polyphenols-Rich Extract from Fruits without Changes in Neuropathology in an Animal Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 1, pp. 115-135, 2017.
, “Comparative Analysis of Cortical Microinfarcts and Microbleeds using 3.0-Tesla Postmortem Magnetic Resonance Images and Histopathology.”, J Alzheimers Dis, vol. 59, no. 3, pp. 951-959, 2017.
, “Comprehensive Screening for Disease Risk Variants in Early-Onset Alzheimer's Disease Genes in African Americans Identifies Novel PSEN Variants.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1215-1222, 2017.
, “Comprehensive Screening for Disease Risk Variants in Early-Onset Alzheimer's Disease Genes in African Americans Identifies Novel PSEN Variants.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1215-1222, 2017.
, “Consecutive Analysis of BACE1 Function on Developing and Developed Neuronal Cells.”, J Alzheimers Dis, vol. 56, no. 2, pp. 641-653, 2017.
, “Cortical Iron Reflects Severity of Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1533-1545, 2017.
, “Cortical Iron Reflects Severity of Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1533-1545, 2017.
, “A Cross-Validation of FDG- and Amyloid-PET Biomarkers in Mild Cognitive Impairment for the Risk Prediction to Dementia due to Alzheimer's Disease in a Clinical Setting.”, J Alzheimers Dis, vol. 59, no. 2, pp. 603-614, 2017.
, “Cyclic cis-Locked Phospho-Dipeptides Reduce Entry of AβPP into Amyloidogenic Processing Pathway.”, J Alzheimers Dis, vol. 55, no. 1, pp. 391-410, 2017.
, “Death of Dementia Patients in Psychiatric Hospitals and Regional Supply of Psychiatric Services: Study of the National Data from 1996 to 2014 in Japan.”, J Alzheimers Dis, vol. 56, no. 2, pp. 817-824, 2017.
, “Death of Dementia Patients in Psychiatric Hospitals and Regional Supply of Psychiatric Services: Study of the National Data from 1996 to 2014 in Japan.”, J Alzheimers Dis, vol. 56, no. 2, pp. 817-824, 2017.
, “Death of Dementia Patients in Psychiatric Hospitals and Regional Supply of Psychiatric Services: Study of the National Data from 1996 to 2014 in Japan.”, J Alzheimers Dis, vol. 56, no. 2, pp. 817-824, 2017.
, “Decrease of Amyloid-β Levels by Curcumin Derivative via Modulation of Amyloid-β Protein Precursor Trafficking.”, J Alzheimers Dis, vol. 56, no. 2, pp. 529-542, 2017.
, “Decreased Expression of hsa-miR-4274 in Cerebrospinal Fluid of Normal Pressure Hydrocephalus Mimics with Parkinsonian Syndromes.”, J Alzheimers Dis, vol. 56, no. 1, pp. 317-325, 2017.
, “Decreased Glucose Metabolism in Medial Prefrontal Areas is Associated with Nutritional Status in Patients with Prodromal and Early Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 1, pp. 225-233, 2017.
, “Decreased Glucose Metabolism in Medial Prefrontal Areas is Associated with Nutritional Status in Patients with Prodromal and Early Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 1, pp. 225-233, 2017.
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