Biblio
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“Increased Electron Paramagnetic Resonance Signal Correlates with Mitochondrial Dysfunction and Oxidative Stress in an Alzheimer's disease Mouse Brain.”, J Alzheimers Dis, vol. 51, no. 2, pp. 571-80, 2016.
, “Increased Electron Paramagnetic Resonance Signal Correlates with Mitochondrial Dysfunction and Oxidative Stress in an Alzheimer's disease Mouse Brain.”, J Alzheimers Dis, vol. 51, no. 2, pp. 571-80, 2016.
, “Increased Total Homocysteine Levels Predict the Risk of Incident Dementia Independent of Cerebral Small-Vessel Diseases and Vascular Risk Factors.”, J Alzheimers Dis, vol. 49, no. 2, pp. 503-13, 2016.
, “Influence of Low-Dose Aspirin on Cerebral Amyloid Angiopathy in Mice.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1037-45, 2016.
, “The Korean Size/Weight Attribute Test: A Semantic Knowledge Test for Korean Older Adults and Brain-Imaging Evidence.”, J Alzheimers Dis, vol. 49, no. 2, pp. 377-86, 2016.
, “The Korean Size/Weight Attribute Test: A Semantic Knowledge Test for Korean Older Adults and Brain-Imaging Evidence.”, J Alzheimers Dis, vol. 49, no. 2, pp. 377-86, 2016.
, “M2 Macrophage Transplantation Ameliorates Cognitive Dysfunction in Amyloid-β-Treated Rats Through Regulation of Microglial Polarization.”, J Alzheimers Dis, vol. 52, no. 2, pp. 483-95, 2016.
, “A Mercaptoacetamide-Based Class II Histone Deacetylase Inhibitor Increases Dendritic Spine Density via RasGRF1/ERK Pathway.”, J Alzheimers Dis, vol. 51, no. 2, pp. 591-604, 2016.
, “Metabolic Syndrome and Mild Cognitive Impairment: A Case-Control Study among Elderly in a Shanghai Suburb.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1175-82, 2016.
, “Mild Cognitive Impairment and Susceptibility to Scams in Old Age.”, J Alzheimers Dis, vol. 49, no. 3, pp. 845-51, 2016.
, “Mini-Mental State Examination in Elderly Chinese: A Population-Based Normative Study.”, J Alzheimers Dis, vol. 53, no. 2, pp. 487-96, 2016.
, “Modulation of miR-19 in Aluminum-Induced Neural Cell Apoptosis.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1149-62, 2016.
, “Morroniside-Induced PP2A Activation Antagonizes Tau Hyperphosphorylation in a Cellular Model of Neurodegeneration.”, J Alzheimers Dis, vol. 51, no. 1, pp. 33-44, 2016.
, “Morroniside-Induced PP2A Activation Antagonizes Tau Hyperphosphorylation in a Cellular Model of Neurodegeneration.”, J Alzheimers Dis, vol. 51, no. 1, pp. 33-44, 2016.
, “Multimodal Classification of Mild Cognitive Impairment Based on Partial Least Squares.”, J Alzheimers Dis, vol. 54, no. 1, pp. 359-71, 2016.
, “Multimodal Classification of Mild Cognitive Impairment Based on Partial Least Squares.”, J Alzheimers Dis, vol. 54, no. 1, pp. 359-71, 2016.
, “Multi-Vitamin B Supplementation Reverses Hypoxia-Induced Tau Hyperphosphorylation and Improves Memory Function in Adult Mice.”, J Alzheimers Dis, vol. 54, no. 1, pp. 297-306, 2016.
, “Neurophysiological assessment of Alzheimer's disease individuals by a single electroencephalographic marker.”, J Alzheimers Dis, vol. 49, no. 1, pp. 159-77, 2016.
, “Older Adults Taking AT1-Receptor Blockers Exhibit Reduced Cerebral Amyloid Retention.”, J Alzheimers Dis, vol. 50, no. 3, pp. 779-89, 2016.
, “Partial Amelioration of Synaptic and Cognitive Deficits by Inhibiting Cofilin Dephosphorylation in an Animal Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1419-32, 2016.
, “Patients that have Undergone Hemodialysis Exhibit Lower Amyloid Deposition in the Brain: Evidence Supporting a Therapeutic Strategy for Alzheimer's Disease by Removal of Blood Amyloid.”, J Alzheimers Dis, vol. 51, no. 4, pp. 997-1002, 2016.
, “Pharmacological Modulations of the Serotonergic System in a Cell-Model of Familial Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 1, pp. 349-61, 2016.
, “PiB-PET Imaging-Based Serum Proteome Profiles Predict Mild Cognitive Impairment and Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1563-76, 2016.
, “Posterior Ventricular Enlargement to Differentiate Dementia with Lewy Bodies from Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1237-43, 2016.
, “A Potential VEP Biomarker for Mild Cognitive Impairment: Evidence from Selective Visual Deficit of Higher-Level Dorsal Pathway.”, J Alzheimers Dis, vol. 53, no. 2, pp. 661-76, 2016.
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