Biblio
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“Implication of the APP Gene in Intellectual Abilities.”, J Alzheimers Dis, vol. 59, no. 2, pp. 723-735, 2017.
, “Incident Cerebral Microbleeds Detected by Susceptibility Weight-Imaging Help to Identify Patients with Mild Cognitive Impairment Progressing to Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 1, pp. 253-262, 2017.
, “Increased PCSK9 Cerebrospinal Fluid Concentrations in Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 1, pp. 315-320, 2017.
, “Increased PCSK9 Cerebrospinal Fluid Concentrations in Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 1, pp. 315-320, 2017.
, “Increased Serum miR-206 Level Predicts Conversion from Amnestic Mild Cognitive Impairment to Alzheimer's Disease: A 5-Year Follow-up Study.”, J Alzheimers Dis, vol. 55, no. 2, pp. 509-520, 2017.
, “Increased Serum miR-206 Level Predicts Conversion from Amnestic Mild Cognitive Impairment to Alzheimer's Disease: A 5-Year Follow-up Study.”, J Alzheimers Dis, vol. 55, no. 2, pp. 509-520, 2017.
, “Inflammation, Amyloid, and Atrophy in The Aging Brain: Relationships with Longitudinal Changes in Cognition.”, J Alzheimers Dis, vol. 58, no. 3, pp. 829-840, 2017.
, “Inflection Point in Course of Mild Cognitive Impairment: Increased Functional Connectivity of Default Mode Network.”, J Alzheimers Dis, vol. 60, no. 2, pp. 679-690, 2017.
, “Inflection Point in Course of Mild Cognitive Impairment: Increased Functional Connectivity of Default Mode Network.”, J Alzheimers Dis, vol. 60, no. 2, pp. 679-690, 2017.
, “Integration of Multilocus Genetic Risk into the Default Mode Network Longitudinal Trajectory during the Alzheimer's Disease Process.”, J Alzheimers Dis, vol. 56, no. 2, pp. 491-507, 2017.
, “Interactive Effects of Dementia Severity and Comorbidities on Medicare Expenditures.”, J Alzheimers Dis, vol. 57, no. 1, pp. 305-315, 2017.
, “Intracranial Arterial 4D Flow in Individuals with Mild Cognitive Impairment is Associated with Cognitive Performance and Amyloid Positivity.”, J Alzheimers Dis, vol. 60, no. 1, pp. 243-252, 2017.
, “Investigation on the Role of BDNF in the Benefits of Blueberry Extracts for the Improvement of Learning and Memory in Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 56, no. 2, pp. 629-640, 2017.
, “Meta-Analysis of Parkinson's Disease and Alzheimer's Disease Revealed Commonly Impaired Pathways and Dysregulation of NRF2-Dependent Genes.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1525-1539, 2017.
, “Microglial and Neuronal TDP-43 Pathology in Anti-IgLON5-Related Tauopathy.”, J Alzheimers Dis, vol. 59, no. 1, pp. 13-20, 2017.
, “Modulating the Balance of Synaptic and Extrasynaptic NMDA Receptors Shows Positive Effects against Amyloid-β-Induced Neurotoxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 885-897, 2017.
, “Modulating the Balance of Synaptic and Extrasynaptic NMDA Receptors Shows Positive Effects against Amyloid-β-Induced Neurotoxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 885-897, 2017.
, “Modulation of Immune Responses to Herpes Simplex Virus Type 1 by IFNL3 and IRF7 Polymorphisms: A Study in Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 3, pp. 1055-1063, 2017.
, “Motor Phenotype in Neurodegenerative Disorders: Gait and Balance Platform Study Design Protocol for the Ontario Neurodegenerative Research Initiative (ONDRI).”, J Alzheimers Dis, vol. 59, no. 2, pp. 707-721, 2017.
, “Multifunctional Compound AD-35 Improves Cognitive Impairment and Attenuates the Production of TNF-α and IL-1β in an Aβ25-35-induced Rat Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1403-1417, 2017.
, “Neuronal Expression of Truncated Tau Efficiently Promotes Neurodegeneration in Animal Models: Pitfalls of Toxic Oligomer Analysis.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1017-1025, 2017.
, “Neuroprotective Effect of TREM-2 in Aging and Alzheimer's Disease Model.”, J Alzheimers Dis, vol. 55, no. 1, pp. 199-217, 2017.
, “Neuropsychological Symptoms in Sporadic Creutzfeldt-Jakob Disease Patients in Germany.”, J Alzheimers Dis, vol. 59, no. 1, pp. 329-337, 2017.
, “Nitrosylation of Vesicular Transporters in Brain of Amyloid Precursor Protein/Presenilin 1 Double Transgenic Mice.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1683-1692, 2017.
, “Nitrosylation of Vesicular Transporters in Brain of Amyloid Precursor Protein/Presenilin 1 Double Transgenic Mice.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1683-1692, 2017.
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