Biblio
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“The Cognitive Change Index as a Measure of Self and Informant Perception of Cognitive Decline: Relation to Neuropsychological Tests.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1145-55, 2016.
, “Cognitive Profile and its Association with Neuroimaging Markers of Non-Demented Cerebral Amyloid Angiopathy Patients in a Stroke Unit.”, J Alzheimers Dis, vol. 52, no. 1, pp. 171-8, 2016.
, “Cognitive Stimulation Modulates Platelet Total Phospholipases A2 Activity in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 50, no. 4, pp. 957-62, 2016.
, “Collapsin Response Mediator Protein-2 (CRMP2) is a Plausible Etiological Factor and Potential Therapeutic Target in Alzheimer's Disease: Comparison and Contrast with Microtubule-Associated Protein Tau.”, J Alzheimers Dis, vol. 53, no. 1, pp. 1-14, 2016.
, “Comparison of Prefrontal Atrophy and Episodic Memory Performance in Dysexecutive Alzheimer's Disease and Behavioral-Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 51, no. 3, pp. 889-903, 2016.
, “Course and Determinants of Anosognosia in Alzheimer's Disease: A 12-Month Follow-up.”, J Alzheimers Dis, vol. 51, no. 2, pp. 357-66, 2016.
, “Decreased Inter-Hemispheric Functional Connectivity in Cognitively Intact Elderly APOE ɛ4 Carriers: A Preliminary Study.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1137-48, 2016.
, “Decreased Levels of VAMP2 and Monomeric Alpha-Synuclein Correlate with Duration of Dementia.”, J Alzheimers Dis, vol. 50, no. 1, pp. 101-10, 2016.
, “Delusions in Patients with Alzheimer's Disease: A Multidimensional Approach.”, J Alzheimers Dis, vol. 51, no. 2, pp. 427-37, 2016.
, “Dementia Apraxia Test (DATE): A Brief Tool to Differentiate Behavioral Variant Frontotemporal Dementia from Alzheimer's Dementia Based on Apraxia Profiles.”, J Alzheimers Dis, vol. 49, no. 3, pp. 593-605, 2016.
, “Diet and Inflammation in Alzheimer's Disease and Related Chronic Diseases: A Review.”, J Alzheimers Dis, vol. 50, no. 2, pp. 301-34, 2016.
, “Dietary high cholesterol and trace metals in the drinking water increase levels of ABCA1 in the rabbit hippocampus and temporal cortex.”, J Alzheimers Dis, vol. 49, no. 1, pp. 201-9, 2016.
, “Different Expression Patterns of Amyloid-β Protein Precursor Secretases in Human and Mouse Hippocampal Neurons: A Potential Contribution to Species Differences in Neuronal Susceptibility to Amyloid-β Pathogenesis.”, J Alzheimers Dis, vol. 51, no. 1, pp. 179-95, 2016.
, “Different Patterns of Correlation between Grey and White Matter Integrity Account for Behavioral and Psychological Symptoms in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 591-604, 2016.
, “Differential Mass Spectrometry Profiles of Tau Protein in the Cerebrospinal Fluid of Patients with Alzheimer's Disease, Progressive Supranuclear Palsy, and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1033-43, 2016.
, “Differential Mass Spectrometry Profiles of Tau Protein in the Cerebrospinal Fluid of Patients with Alzheimer's Disease, Progressive Supranuclear Palsy, and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1033-43, 2016.
, “Differential Membrane Toxicity of Amyloid-β Fragments by Pore Forming Mechanisms.”, J Alzheimers Dis, vol. 51, no. 3, pp. 689-99, 2016.
, “Differential Regulation of N-Methyl-D-Aspartate Receptor Subunits is an Early Event in the Actions of Soluble Amyloid-β(1-40) Oligomers on Hippocampal Neurons.”, J Alzheimers Dis, vol. 51, no. 1, pp. 197-212, 2016.
, “Diminished CRE-Induced Plasticity is Linked to Memory Deficits in Familial Alzheimer's Disease Mice.”, J Alzheimers Dis, vol. 50, no. 2, pp. 477-89, 2016.
, “Diminished CRE-Induced Plasticity is Linked to Memory Deficits in Familial Alzheimer's Disease Mice.”, J Alzheimers Dis, vol. 50, no. 2, pp. 477-89, 2016.
, “Discovering New Genes in the Pathways of Common Sporadic Neurodegenerative Diseases: A Bioinformatics Approach.”, J Alzheimers Dis, vol. 51, no. 1, pp. 293-312, 2016.
, “Distinctive Resting State Network Disruptions Among Alzheimer's Disease, Subcortical Vascular Dementia, and Mixed Dementia Patients.”, J Alzheimers Dis, vol. 50, no. 3, pp. 709-18, 2016.
, “Distinctive Resting State Network Disruptions Among Alzheimer's Disease, Subcortical Vascular Dementia, and Mixed Dementia Patients.”, J Alzheimers Dis, vol. 50, no. 3, pp. 709-18, 2016.
, “Divergent Longitudinal Propagation of White Matter Degradation in Logopenic and Semantic Variants of Primary Progressive Aphasia.”, J Alzheimers Dis, vol. 49, no. 3, pp. 853-61, 2016.
, “Divergent Longitudinal Propagation of White Matter Degradation in Logopenic and Semantic Variants of Primary Progressive Aphasia.”, J Alzheimers Dis, vol. 49, no. 3, pp. 853-61, 2016.
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