Biblio
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“UCH-L1 Inhibition Decreases the Microtubule-Binding Function of Tau Protein.”, J Alzheimers Dis, vol. 49, no. 2, pp. 353-63, 2016.
, “Unmet Needs of Community-Dwelling Primary Care Patients with Dementia in Germany: Prevalence and Correlates.”, J Alzheimers Dis, vol. 51, no. 3, pp. 847-55, 2016.
, “Upregulation of Connexin 43 Expression Via C-Jun N-Terminal Kinase Signaling in Prion Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1005-19, 2016.
, “The Utilization of Retinal Nerve Fiber Layer Thickness to Predict Cognitive Deterioration.”, J Alzheimers Dis, vol. 49, no. 2, pp. 399-405, 2016.
, “Vascular Risk Factors and Cognition in Parkinson's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 563-70, 2016.
, “Vascular Risk Factors and Cognition in Parkinson's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 563-70, 2016.
, “Ventilatory Response to Hypercapnia Predicts Dementia with Lewy Bodies in Late-Onset Major Depressive Disorder.”, J Alzheimers Dis, vol. 50, no. 3, pp. 751-8, 2016.
, “Ventilatory Response to Hypercapnia Predicts Dementia with Lewy Bodies in Late-Onset Major Depressive Disorder.”, J Alzheimers Dis, vol. 50, no. 3, pp. 751-8, 2016.
, “Vitamin D and Memory Decline: Two Population-Based Prospective Studies.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1099-108, 2016.
, “Volumetric and shape analysis of the thalamus and striatum in amnestic mild cognitive impairment.”, J Alzheimers Dis, vol. 49, no. 1, pp. 237-49, 2016.
, “What are the Most Frequently Impaired Markers of Neurodegeneration in ADNI Subjects?”, J Alzheimers Dis, vol. 51, no. 3, pp. 793-800, 2016.
, “What are the Most Frequently Impaired Markers of Neurodegeneration in ADNI Subjects?”, J Alzheimers Dis, vol. 51, no. 3, pp. 793-800, 2016.
, “What are the Most Frequently Impaired Markers of Neurodegeneration in ADNI Subjects?”, J Alzheimers Dis, vol. 51, no. 3, pp. 793-800, 2016.
, “White Matter Abnormalities Track Disease Progression in PSEN1 Autosomal Dominant Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 3, pp. 827-35, 2016.
, “δ Scores are Exportable Across Cultural and Linguistic Boundaries.”, J Alzheimers Dis, vol. 49, no. 2, pp. 561-70, 2016.
, “18F-AV-1451 PET Imaging in Three Patients with Probable Cerebral Amyloid Angiopathy.”, J Alzheimers Dis, vol. 57, no. 3, pp. 711-716, 2017.
, “Amyloid-β Deposition and Long-Term Progression in Mild Cognitive Impairment due to Alzheimer's Disease Defined with Amyloid PET Imaging.”, J Alzheimers Dis, vol. 57, no. 3, pp. 765-773, 2017.
, “Exercise Training and Functional Connectivity Changes in Mild Cognitive Impairment and Healthy Elders.”, J Alzheimers Dis, vol. 57, no. 3, pp. 845-856, 2017.
, “Family History of Alzheimer's Disease is Associated with Impaired Perceptual Discrimination of Novel Objects.”, J Alzheimers Dis, vol. 57, no. 3, pp. 735-745, 2017.
, “Family History of Alzheimer's Disease is Associated with Impaired Perceptual Discrimination of Novel Objects.”, J Alzheimers Dis, vol. 57, no. 3, pp. 735-745, 2017.
, “Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 857-871, 2017.
, “Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 857-871, 2017.
, “Low Florbetapir PET Uptake and Normal Aβ1-42 Cerebrospinal Fluid in an APP Ala713Thr Mutation Carrier.”, J Alzheimers Dis, vol. 57, no. 3, pp. 697-703, 2017.
, “Low Florbetapir PET Uptake and Normal Aβ1-42 Cerebrospinal Fluid in an APP Ala713Thr Mutation Carrier.”, J Alzheimers Dis, vol. 57, no. 3, pp. 697-703, 2017.
, “Modulating the Balance of Synaptic and Extrasynaptic NMDA Receptors Shows Positive Effects against Amyloid-β-Induced Neurotoxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 885-897, 2017.
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