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“Copy Number Variants in Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 1, pp. 37-52, 2017.
, “The Correlation between Inflammatory Biomarkers and Polygenic Risk Score in Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 1, pp. 25-36, 2017.
, “Cortical Cerebral Microinfarcts on 3 Tesla MRI in Patients with Vascular Cognitive Impairment.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1443-1450, 2017.
, “Cortical Iron Reflects Severity of Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1533-1545, 2017.
, “Cortical Metabolic and Cognitive Correlates of Disorientation in Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 2, pp. 707-719, 2017.
, “Cortical Thickness and Depressive Symptoms in Cognitively Normal Individuals: The Mayo Clinic Study of Aging.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1273-1281, 2017.
, “Cortical Thickness and Microstructural White Matter Changes Detect Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 56, no. 1, pp. 415-428, 2017.
, “Cost-Utility of Using Alzheimer's Disease Biomarkers in Cerebrospinal Fluid to Predict Progression from Mild Cognitive Impairment to Dementia.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1477-1487, 2017.
, “A Critical Assessment of Research on Neurotransmitters in Alzheimer's Disease.”, J Alzheimers Dis, vol. 57, no. 4, pp. 969-974, 2017.
, “Cross-Cultural Applicability of the Montreal Cognitive Assessment (MoCA): A Systematic Review.”, J Alzheimers Dis, vol. 58, no. 3, pp. 789-801, 2017.
, “Cross-Sectional and Longitudinal Cognitive Correlates of FDDNP PET and CSF Amyloid-β and Tau in Parkinson's Disease1.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1261-1272, 2017.
, “A Cross-Validation of FDG- and Amyloid-PET Biomarkers in Mild Cognitive Impairment for the Risk Prediction to Dementia due to Alzheimer's Disease in a Clinical Setting.”, J Alzheimers Dis, vol. 59, no. 2, pp. 603-614, 2017.
, “Curcumin Ameliorates Neuroinflammation, Neurodegeneration, and Memory Deficits in p25 Transgenic Mouse Model that Bears Hallmarks of Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1429-1442, 2017.
, “Curcumin Inhibits Tau Aggregation and Disintegrates Preformed Tau Filaments in vitro.”, J Alzheimers Dis, vol. 60, no. 3, pp. 999-1014, 2017.
, “Current Approaches and Clinician Attitudes to the Use of Cerebrospinal Fluid Biomarkers in Diagnostic Evaluation of Dementia in Europe.”, J Alzheimers Dis, vol. 60, no. 1, pp. 201-210, 2017.
, “Cyclic cis-Locked Phospho-Dipeptides Reduce Entry of AβPP into Amyloidogenic Processing Pathway.”, J Alzheimers Dis, vol. 55, no. 1, pp. 391-410, 2017.
, “Cyclophilin D Promotes Brain Mitochondrial F1FO ATP Synthase Dysfunction in Aging Mice.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1351-1362, 2017.
, “Daily Physical Activity Patterns During the Early Stage of Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 2, pp. 659-667, 2017.
, “Damage to the Frontal Aslant Tract Accounts for Visuo-Constructive Deficits in Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 3, pp. 1015-1024, 2017.
, “Death of Dementia Patients in Psychiatric Hospitals and Regional Supply of Psychiatric Services: Study of the National Data from 1996 to 2014 in Japan.”, J Alzheimers Dis, vol. 56, no. 2, pp. 817-824, 2017.
, “Decrease of Amyloid-β Levels by Curcumin Derivative via Modulation of Amyloid-β Protein Precursor Trafficking.”, J Alzheimers Dis, vol. 56, no. 2, pp. 529-542, 2017.
, “Decreased Expression of hsa-miR-4274 in Cerebrospinal Fluid of Normal Pressure Hydrocephalus Mimics with Parkinsonian Syndromes.”, J Alzheimers Dis, vol. 56, no. 1, pp. 317-325, 2017.
, “Decreased Glucose Metabolism in Medial Prefrontal Areas is Associated with Nutritional Status in Patients with Prodromal and Early Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 1, pp. 225-233, 2017.
, “Decreased Methylation of the Mitochondrial D-Loop Region in Late-Onset Alzheimer's Disease.”, J Alzheimers Dis, vol. 59, no. 2, pp. 559-564, 2017.
, “Decreased N-Acetyl Aspartate/Myo-Inositol Ratio in the Posterior Cingulate Cortex Shown by Magnetic Resonance Spectroscopy May Be One of the Risk Markers of Preclinical Alzheimer's Disease: A 7-Year Follow-Up Study.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1411-1427, 2017.
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