Biblio
“Genetic Counseling and Testing for Alzheimer's Disease and Frontotemporal Lobar Degeneration: An Italian Consensus Protocol.”, J Alzheimers Dis, vol. 51, no. 1, pp. 277-91, 2016.
, “PRNP P39L Variant is a Rare Cause of Frontotemporal Dementia in Italian Population.”, J Alzheimers Dis, vol. 50, no. 2, pp. 353-7, 2016.
, “Effects of Multiple Genetic Loci on Age at Onset in Frontotemporal Dementia.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1271-1278, 2017.
, “The level of 24-Hydroxycholesteryl Esters is an Early Marker of Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 2, pp. 825-833, 2017.
, “Tau Rather than TDP-43 Proteins are Potential Cerebrospinal Fluid Biomarkers for Frontotemporal Lobar Degeneration Subtypes: A Pilot Study.”, J Alzheimers Dis, vol. 55, no. 2, pp. 585-595, 2017.
, “Altered Expression of Circulating Cdc42 in Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1477-1483, 2018.
, “The Heritability of Frontotemporal Lobar Degeneration: Validation of Pedigree Classification Criteria in a Northern Italy Cohort.”, J Alzheimers Dis, vol. 61, no. 2, pp. 753-760, 2018.
, “Innovative Biomarkers for Alzheimer's Disease: Focus on the Hidden Disease Biomarkers.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1507-1518, 2018.
, “Quantitative Genetics Validates Previous Genetic Variants and Identifies Novel Genetic Players Influencing Alzheimer's Disease Cerebrospinal Fluid Biomarkers.”, J Alzheimers Dis, vol. 66, no. 2, pp. 639-652, 2018.
, “Serum Copper is not Altered in Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 63, no. 4, pp. 1427-1432, 2018.
, “Serum C-Peptide, Visfatin, Resistin, and Ghrelin are Altered in Sporadic and GRN-Associated Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1053-1060, 2018.
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