Biblio
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“Cerebral Oxidative Stress and Microvasculature Defects in TNF-α Expressing Transgenic and Porphyromonas gingivalis-Infected ApoE-/- Mice.”, J Alzheimers Dis, vol. 60, no. 2, pp. 359-369, 2017.
, “Cerebral Oxidative Stress and Microvasculature Defects in TNF-α Expressing Transgenic and Porphyromonas gingivalis-Infected ApoE-/- Mice.”, J Alzheimers Dis, vol. 60, no. 2, pp. 359-369, 2017.
, “Cerebrospinal Fluid Amyloid-β 42, Total Tau and Phosphorylated Tau are Low in Patients with Normal Pressure Hydrocephalus: Analogies and Differences with Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 1, pp. 183-200, 2017.
, “Cerebrospinal Fluid Amyloid-β 42, Total Tau and Phosphorylated Tau are Low in Patients with Normal Pressure Hydrocephalus: Analogies and Differences with Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 1, pp. 183-200, 2017.
, “Cerebrospinal Fluid Amyloid-β 42, Total Tau and Phosphorylated Tau are Low in Patients with Normal Pressure Hydrocephalus: Analogies and Differences with Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 1, pp. 183-200, 2017.
, “The Cerebrospinal Fluid Aβ1-42/Aβ1-40 Ratio Improves Concordance with Amyloid-PET for Diagnosing Alzheimer's Disease in a Clinical Setting.”, J Alzheimers Dis, vol. 60, no. 2, pp. 561-576, 2017.
, “Cerebrospinal Fluid Biomarkers as a Diagnostic Tool of the Underlying Pathology of Primary Progressive Aphasia.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1453-1461, 2017.
, “Cerebrospinal Fluid Biomarkers for Alzheimer's Disease: A View of the Regulatory Science Qualification Landscape from the Coalition Against Major Diseases CSF Biomarker Team.”, J Alzheimers Dis, vol. 55, no. 1, pp. 19-35, 2017.
, “Cerebrospinal Fluid Neurogranin as a Biomarker of Neurodegenerative Diseases: A Cross-Sectional Study.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1327-1334, 2017.
, “Challenges and Considerations Related to Studying Dementia in Blacks/African Americans.”, J Alzheimers Dis, vol. 60, no. 1, pp. 1-10, 2017.
, “Change in Odor Identification Impairment is Associated with Improvement with Cholinesterase Inhibitor Treatment in Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1525-1531, 2017.
, “Change in Odor Identification Impairment is Associated with Improvement with Cholinesterase Inhibitor Treatment in Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1525-1531, 2017.
, “Changes in Plasma β-NGF and Its Receptors Expression on Peripheral Blood Monocytes During Alzheimer's Disease Progression.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1005-1017, 2017.
, “Changes in Plasma β-NGF and Its Receptors Expression on Peripheral Blood Monocytes During Alzheimer's Disease Progression.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1005-1017, 2017.
, “Chinese Calligraphy Writing for Augmenting Attentional Control and Working Memory of Older Adults at Risk of Mild Cognitive Impairment: A Randomized Controlled Trial.”, J Alzheimers Dis, vol. 58, no. 3, pp. 735-746, 2017.
, “Chinese Calligraphy Writing for Augmenting Attentional Control and Working Memory of Older Adults at Risk of Mild Cognitive Impairment: A Randomized Controlled Trial.”, J Alzheimers Dis, vol. 58, no. 3, pp. 735-746, 2017.
, “Cholinergic Basal Forebrain Lesion Decreases Neurotrophin Signaling without Affecting Tau Hyperphosphorylation in Genetically Susceptible Mice.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1141-1154, 2017.
, “Chronic Lithium Treatment in a Rat Model of Basal Forebrain Cholinergic Depletion: Effects on Memory Impairment and Neurodegeneration.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1505-1518, 2017.
, “Chronic Lithium Treatment in a Rat Model of Basal Forebrain Cholinergic Depletion: Effects on Memory Impairment and Neurodegeneration.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1505-1518, 2017.
, “Chronic Verubecestat Treatment Suppresses Amyloid Accumulation in Advanced Aged Tg2576-AβPPswe Mice Without Inducing Microhemorrhage.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1393-1413, 2017.
, “Chronotropic Response and Cognitive Function in a Cohort at Risk for Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 1, pp. 351-359, 2017.
, “Chronotropic Response and Cognitive Function in a Cohort at Risk for Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 1, pp. 351-359, 2017.
, “The Citicholinage Study: Citicoline Plus Cholinesterase Inhibitors in Aged Patients Affected with Alzheimer's Disease Study.”, J Alzheimers Dis, vol. 56, no. 2, pp. 557-565, 2017.
, “The Citicholinage Study: Citicoline Plus Cholinesterase Inhibitors in Aged Patients Affected with Alzheimer's Disease Study.”, J Alzheimers Dis, vol. 56, no. 2, pp. 557-565, 2017.
, “The Citicholinage Study: Citicoline Plus Cholinesterase Inhibitors in Aged Patients Affected with Alzheimer's Disease Study.”, J Alzheimers Dis, vol. 56, no. 2, pp. 557-565, 2017.
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