Biblio
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“Neuronal Expression of Truncated Tau Efficiently Promotes Neurodegeneration in Animal Models: Pitfalls of Toxic Oligomer Analysis.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1017-1025, 2017.
, “Neuroprotective Effect of TREM-2 in Aging and Alzheimer's Disease Model.”, J Alzheimers Dis, vol. 55, no. 1, pp. 199-217, 2017.
, “Neuroprotective Effect of TREM-2 in Aging and Alzheimer's Disease Model.”, J Alzheimers Dis, vol. 55, no. 1, pp. 199-217, 2017.
, “Neuropsychiatric Symptoms and Alzheimer's Disease Biomarkers Predict Driving Decline: Brief Report.”, J Alzheimers Dis, vol. 58, no. 3, pp. 675-680, 2017.
, “New Gold in Them Thar Hills: Testing a Novel Supply Route for Plant-Derived Galanthamine.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1321-1325, 2017.
, “No Genetic Overlap Between Circulating Iron Levels and Alzheimer's Disease.”, J Alzheimers Dis, vol. 59, no. 1, pp. 85-99, 2017.
, “Non-Phosphorylated Tau as a Potential Biomarker of Alzheimer's Disease: Analytical and Diagnostic Characterization.”, J Alzheimers Dis, vol. 55, no. 1, pp. 159-170, 2017.
, “Novel Blood-Based Biomarkers of Cognition, Stress, and Physical or Cognitive Training in Older Adults at Risk of Dementia: Preliminary Evidence for a Role of BDNF, Irisin, and the Kynurenine Pathway.”, J Alzheimers Dis, vol. 59, no. 3, pp. 1097-1111, 2017.
, “One-Year Evolution of Behavioral and Psychological Symptoms of Dementia in Patients Initially Hospitalized in Cognitive Behavioral Units: The EVITAL Prospective Cohort.”, J Alzheimers Dis, vol. 57, no. 1, pp. 147-155, 2017.
, “One-Year Evolution of Behavioral and Psychological Symptoms of Dementia in Patients Initially Hospitalized in Cognitive Behavioral Units: The EVITAL Prospective Cohort.”, J Alzheimers Dis, vol. 57, no. 1, pp. 147-155, 2017.
, “One-Year Evolution of Behavioral and Psychological Symptoms of Dementia in Patients Initially Hospitalized in Cognitive Behavioral Units: The EVITAL Prospective Cohort.”, J Alzheimers Dis, vol. 57, no. 1, pp. 147-155, 2017.
, “Optical Coherence Tomography Reveals Retinal Neuroaxonal Thinning in Frontotemporal Dementia as in Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1101-1107, 2017.
, “Overexpression of Ubiquilin-1 Alleviates Alzheimer's Disease-Caused Cognitive and Motor Deficits and Reduces Amyloid-β Accumulation in Mice.”, J Alzheimers Dis, vol. 59, no. 2, pp. 575-590, 2017.
, “PE859, A Novel Curcumin Derivative, Inhibits Amyloid-β and Tau Aggregation, and Ameliorates Cognitive Dysfunction in Senescence-Accelerated Mouse Prone 8.”, J Alzheimers Dis, vol. 59, no. 1, pp. 313-328, 2017.
, “PET Tau and Amyloid-β Burden in Mild Alzheimer's Disease: Divergent Relationship with Age, Cognition, and Cerebrospinal Fluid Biomarkers.”, J Alzheimers Dis, vol. 60, no. 1, pp. 283-293, 2017.
, “PET Tau and Amyloid-β Burden in Mild Alzheimer's Disease: Divergent Relationship with Age, Cognition, and Cerebrospinal Fluid Biomarkers.”, J Alzheimers Dis, vol. 60, no. 1, pp. 283-293, 2017.
, “Physical Exercise Moderates the Relationship of Apolipoprotein E (APOE) Genotype and Dementia Risk: A Population-Based Study.”, J Alzheimers Dis, vol. 56, no. 1, pp. 297-303, 2017.
, “Physical Exercise Moderates the Relationship of Apolipoprotein E (APOE) Genotype and Dementia Risk: A Population-Based Study.”, J Alzheimers Dis, vol. 56, no. 1, pp. 297-303, 2017.
, “Polymorphonuclear Neutrophil Functions are Differentially Altered in Amnestic Mild Cognitive Impairment and Mild Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 60, no. 1, pp. 23-42, 2017.
, “Polymorphonuclear Neutrophil Functions are Differentially Altered in Amnestic Mild Cognitive Impairment and Mild Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 60, no. 1, pp. 23-42, 2017.
, “Preclinical Amyloid-β and Axonal Degeneration Pathology in Delirium.”, J Alzheimers Dis, vol. 55, no. 1, pp. 371-379, 2017.
, “Predicting Development of Amyotrophic Lateral Sclerosis in Frontotemporal Dementia.”, J Alzheimers Dis, vol. 58, no. 1, pp. 163-170, 2017.
, “Predictors of Discharge Destinations and Three-Month Evolution of Patients Initially Hospitalized in a Cognitive Behavioral Unit.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1259-1266, 2017.
, “Predictors of Discharge Destinations and Three-Month Evolution of Patients Initially Hospitalized in a Cognitive Behavioral Unit.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1259-1266, 2017.
, “Prion Protein Interactome: Identifying Novel Targets in Slowly and Rapidly Progressive Forms of Alzheimer's Disease.”, J Alzheimers Dis, vol. 59, no. 1, pp. 265-275, 2017.
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