Biblio
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“Multi-Vitamin B Supplementation Reverses Hypoxia-Induced Tau Hyperphosphorylation and Improves Memory Function in Adult Mice.”, J Alzheimers Dis, vol. 54, no. 1, pp. 297-306, 2016.
, “Neurodegeneration-Like Pathological and Behavioral Changes in an AAV9-Mediated p25 Overexpression Mouse Model.”, J Alzheimers Dis, vol. 53, no. 3, pp. 843-55, 2016.
, “Neurodegeneration-Like Pathological and Behavioral Changes in an AAV9-Mediated p25 Overexpression Mouse Model.”, J Alzheimers Dis, vol. 53, no. 3, pp. 843-55, 2016.
, “Neurodegeneration-Like Pathological and Behavioral Changes in an AAV9-Mediated p25 Overexpression Mouse Model.”, J Alzheimers Dis, vol. 53, no. 3, pp. 843-55, 2016.
, “Neuroimaging Correlates of Frontotemporal Dementia Associated with SQSTM1 Mutations.”, J Alzheimers Dis, vol. 53, no. 1, pp. 303-13, 2016.
, “Neuropsychological Markers of Medial Perirhinal and Entorhinal Cortex Functioning are Impaired Twelve Years Preceding Diagnosis of Alzheimer's Dementia.”, J Alzheimers Dis, vol. 52, no. 2, pp. 573-80, 2016.
, “Non-Fibrillar Oligomeric Amyloid-β within Synapses.”, J Alzheimers Dis, vol. 53, no. 3, pp. 787-800, 2016.
, “Non-Fibrillar Oligomeric Amyloid-β within Synapses.”, J Alzheimers Dis, vol. 53, no. 3, pp. 787-800, 2016.
, “Non-Fibrillar Oligomeric Amyloid-β within Synapses.”, J Alzheimers Dis, vol. 53, no. 3, pp. 787-800, 2016.
, “Non-Verbal Episodic Memory Deficits in Primary Progressive Aphasias are Highly Predictive of Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 51, no. 2, pp. 367-76, 2016.
, “Non-Verbal Episodic Memory Deficits in Primary Progressive Aphasias are Highly Predictive of Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 51, no. 2, pp. 367-76, 2016.
, “A Novel Liposomal Nanoparticle for the Imaging of Amyloid Plaque by Magnetic Resonance Imaging.”, J Alzheimers Dis, vol. 52, no. 2, pp. 731-45, 2016.
, “N-Terminal Pro-B-Type Natriuretic Peptide and Risk of Future Cognitive Impairment in the REGARDS Cohort.”, J Alzheimers Dis, vol. 54, no. 2, pp. 497-503, 2016.
, “N-truncated Aβ2-X starting with position two in sporadic Alzheimer's disease cases and two Alzheimer mouse models.”, J Alzheimers Dis, vol. 49, no. 1, pp. 101-10, 2016.
, “Older Adults Taking AT1-Receptor Blockers Exhibit Reduced Cerebral Amyloid Retention.”, J Alzheimers Dis, vol. 50, no. 3, pp. 779-89, 2016.
, “Optimized Standard Operating Procedures for the Analysis of Cerebrospinal Fluid Aβ42 and the Ratios of Aβ Isoforms Using Low Protein Binding Tubes.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1121-32, 2016.
, “Optimized Standard Operating Procedures for the Analysis of Cerebrospinal Fluid Aβ42 and the Ratios of Aβ Isoforms Using Low Protein Binding Tubes.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1121-32, 2016.
, “An Oral Aβ Vaccine Using a Recombinant Adeno-Associated Virus Vector in Aged Monkeys: Reduction in Plaque Amyloid and Increase in Aβ Oligomers.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1047-1059, 2016.
, “An Oral Aβ Vaccine Using a Recombinant Adeno-Associated Virus Vector in Aged Monkeys: Reduction in Plaque Amyloid and Increase in Aβ Oligomers.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1047-1059, 2016.
, “Overexpression of Metallothionein-1 Modulates the Phenotype of the Tg2576 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 81-95, 2016.
, “A Pathway Based Classification Method for Analyzing Gene Expression for Alzheimer's Disease Diagnosis.”, J Alzheimers Dis, vol. 49, no. 3, pp. 659-69, 2016.
, “Patients that have Undergone Hemodialysis Exhibit Lower Amyloid Deposition in the Brain: Evidence Supporting a Therapeutic Strategy for Alzheimer's Disease by Removal of Blood Amyloid.”, J Alzheimers Dis, vol. 51, no. 4, pp. 997-1002, 2016.
, “Patients that have Undergone Hemodialysis Exhibit Lower Amyloid Deposition in the Brain: Evidence Supporting a Therapeutic Strategy for Alzheimer's Disease by Removal of Blood Amyloid.”, J Alzheimers Dis, vol. 51, no. 4, pp. 997-1002, 2016.
, “Patients that have Undergone Hemodialysis Exhibit Lower Amyloid Deposition in the Brain: Evidence Supporting a Therapeutic Strategy for Alzheimer's Disease by Removal of Blood Amyloid.”, J Alzheimers Dis, vol. 51, no. 4, pp. 997-1002, 2016.
, “Patients with Amyloid-Negative Mild Cognitive Impairment have Cortical Hypometabolism but the Hippocampus is Preserved.”, J Alzheimers Dis, vol. 53, no. 2, pp. 651-60, 2016.
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