Biblio

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2016
L. Yu, Chen, Y., Wang, W., Xiao, Z., and Hong, Y., “Multi-Vitamin B Supplementation Reverses Hypoxia-Induced Tau Hyperphosphorylation and Improves Memory Function in Adult Mice.”, J Alzheimers Dis, vol. 54, no. 1, pp. 297-306, 2016.
X. Zhou, Huang, J., Pan, S., Xu, M., He, R., Ji, Z., and Hu, Y., “Neurodegeneration-Like Pathological and Behavioral Changes in an AAV9-Mediated p25 Overexpression Mouse Model.”, J Alzheimers Dis, vol. 53, no. 3, pp. 843-55, 2016.
X. Zhou, Huang, J., Pan, S., Xu, M., He, R., Ji, Z., and Hu, Y., “Neurodegeneration-Like Pathological and Behavioral Changes in an AAV9-Mediated p25 Overexpression Mouse Model.”, J Alzheimers Dis, vol. 53, no. 3, pp. 843-55, 2016.
X. Zhou, Huang, J., Pan, S., Xu, M., He, R., Ji, Z., and Hu, Y., “Neurodegeneration-Like Pathological and Behavioral Changes in an AAV9-Mediated p25 Overexpression Mouse Model.”, J Alzheimers Dis, vol. 53, no. 3, pp. 843-55, 2016.
E. Luis, Ortiz, A., Eudave, L., Ortega-Cubero, S., Borroni, B., van der Zee, J., Gazzina, S., Caroppo, P., Rubino, E., D'Agata, F., Le Ber, I., Santana, I., Cunha, G., Almeida, M. R., Boutoleau-Bretonnière, C., Hannequin, D., Wallon, D., Rainero, I., Galimberti, D., Van Broeckhoven, C., Pastor, M. A., and Pastor, P., “Neuroimaging Correlates of Frontotemporal Dementia Associated with SQSTM1 Mutations.”, J Alzheimers Dis, vol. 53, no. 1, pp. 303-13, 2016.
D. I. Hirni, Kivisaari, S. L., Krumm, S., Monsch, A. U., Berres, M., Oeksuez, F., Reinhardt, J., Ulmer, S., Kressig, R. W., Stippich, C., and Taylor, K. I., “Neuropsychological Markers of Medial Perirhinal and Entorhinal Cortex Functioning are Impaired Twelve Years Preceding Diagnosis of Alzheimer's Dementia.”, J Alzheimers Dis, vol. 52, no. 2, pp. 573-80, 2016.
E. K. Pickett, Koffie, R. M., Wegmann, S., Henstridge, C. M., Herrmann, A. G., Colom-Cadena, M., Lleo, A., Kay, K. R., Vaught, M., Soberman, R., Walsh, D. M., Hyman, B. T., and Spires-Jones, T. L., “Non-Fibrillar Oligomeric Amyloid-β within Synapses.”, J Alzheimers Dis, vol. 53, no. 3, pp. 787-800, 2016.
E. K. Pickett, Koffie, R. M., Wegmann, S., Henstridge, C. M., Herrmann, A. G., Colom-Cadena, M., Lleo, A., Kay, K. R., Vaught, M., Soberman, R., Walsh, D. M., Hyman, B. T., and Spires-Jones, T. L., “Non-Fibrillar Oligomeric Amyloid-β within Synapses.”, J Alzheimers Dis, vol. 53, no. 3, pp. 787-800, 2016.
E. K. Pickett, Koffie, R. M., Wegmann, S., Henstridge, C. M., Herrmann, A. G., Colom-Cadena, M., Lleo, A., Kay, K. R., Vaught, M., Soberman, R., Walsh, D. M., Hyman, B. T., and Spires-Jones, T. L., “Non-Fibrillar Oligomeric Amyloid-β within Synapses.”, J Alzheimers Dis, vol. 53, no. 3, pp. 787-800, 2016.
S. Ramanan, Flanagan, E., Leyton, C. E., Villemagne, V. L., Rowe, C. C., Hodges, J. R., and Hornberger, M., “Non-Verbal Episodic Memory Deficits in Primary Progressive Aphasias are Highly Predictive of Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 51, no. 2, pp. 367-76, 2016.
S. Ramanan, Flanagan, E., Leyton, C. E., Villemagne, V. L., Rowe, C. C., Hodges, J. R., and Hornberger, M., “Non-Verbal Episodic Memory Deficits in Primary Progressive Aphasias are Highly Predictive of Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 51, no. 2, pp. 367-76, 2016.
E. A. Tanifum, Ghaghada, K., Vollert, C., Head, E., Eriksen, J. L., and Annapragada, A., “A Novel Liposomal Nanoparticle for the Imaging of Amyloid Plaque by Magnetic Resonance Imaging.”, J Alzheimers Dis, vol. 52, no. 2, pp. 731-45, 2016.
M. Cushman, Callas, P. W., McClure, L. A., Unverzagt, F. W., Howard, V. J., Gillett, S. R., Thacker, E. L., and Wadley, V. G., “N-Terminal Pro-B-Type Natriuretic Peptide and Risk of Future Cognitive Impairment in the REGARDS Cohort.”, J Alzheimers Dis, vol. 54, no. 2, pp. 497-503, 2016.
A. Savastano, Klafki, H., Haußmann, U., Oberstein, T. Jan, Muller, P., Wirths, O., Wiltfang, J., and Bayer, T. A., “N-truncated Aβ2-X starting with position two in sporadic Alzheimer's disease cases and two Alzheimer mouse models.”, J Alzheimers Dis, vol. 49, no. 1, pp. 101-10, 2016.
D. A. Nation, Ho, J., and Yew, B., “Older Adults Taking AT1-Receptor Blockers Exhibit Reduced Cerebral Amyloid Retention.”, J Alzheimers Dis, vol. 50, no. 3, pp. 779-89, 2016.
H. Marcel Joh Vanderstichele, Janelidze, S., Demeyer, L., Coart, E., Stoops, E., Herbst, V., Mauroo, K., Brix, B., and Hansson, O., “Optimized Standard Operating Procedures for the Analysis of Cerebrospinal Fluid Aβ42 and the Ratios of Aβ Isoforms Using Low Protein Binding Tubes.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1121-32, 2016.
H. Marcel Joh Vanderstichele, Janelidze, S., Demeyer, L., Coart, E., Stoops, E., Herbst, V., Mauroo, K., Brix, B., and Hansson, O., “Optimized Standard Operating Procedures for the Analysis of Cerebrospinal Fluid Aβ42 and the Ratios of Aβ Isoforms Using Low Protein Binding Tubes.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1121-32, 2016.
H. Hara, Ono, F., Nakamura, S., Matsumoto, S. - E., Jin, H., Hattori, N., and Tabira, T., “An Oral Aβ Vaccine Using a Recombinant Adeno-Associated Virus Vector in Aged Monkeys: Reduction in Plaque Amyloid and Increase in Aβ Oligomers.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1047-1059, 2016.
H. Hara, Ono, F., Nakamura, S., Matsumoto, S. - E., Jin, H., Hattori, N., and Tabira, T., “An Oral Aβ Vaccine Using a Recombinant Adeno-Associated Virus Vector in Aged Monkeys: Reduction in Plaque Amyloid and Increase in Aβ Oligomers.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1047-1059, 2016.
Y. Manso, Comes, G., López-Ramos, J. C., Belfiore, M., Molinero, A., Giralt, M., Carrasco, J., Adlard, P. A., Bush, A. I., Delgado-García, J. María, and Hidalgo, J., “Overexpression of Metallothionein-1 Modulates the Phenotype of the Tg2576 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 81-95, 2016.
N. Voyle, Keohane, A., Newhouse, S., Lunnon, K., Johnston, C., Soininen, H., Kloszewska, I., Mecocci, P., Tsolaki, M., Vellas, B., Lovestone, S., Hodges, A., Kiddle, S., and Dobson, R. Jb, “A Pathway Based Classification Method for Analyzing Gene Expression for Alzheimer's Disease Diagnosis.”, J Alzheimers Dis, vol. 49, no. 3, pp. 659-69, 2016.
K. Sakai, Senda, T., Hata, R., Kuroda, M., Hasegawa, M., Kato, M., Abe, M., Kawaguchi, K., Nakai, S., Hiki, Y., Yuzawa, Y., and Kitaguchi, N., “Patients that have Undergone Hemodialysis Exhibit Lower Amyloid Deposition in the Brain: Evidence Supporting a Therapeutic Strategy for Alzheimer's Disease by Removal of Blood Amyloid.”, J Alzheimers Dis, vol. 51, no. 4, pp. 997-1002, 2016.
K. Sakai, Senda, T., Hata, R., Kuroda, M., Hasegawa, M., Kato, M., Abe, M., Kawaguchi, K., Nakai, S., Hiki, Y., Yuzawa, Y., and Kitaguchi, N., “Patients that have Undergone Hemodialysis Exhibit Lower Amyloid Deposition in the Brain: Evidence Supporting a Therapeutic Strategy for Alzheimer's Disease by Removal of Blood Amyloid.”, J Alzheimers Dis, vol. 51, no. 4, pp. 997-1002, 2016.
K. Sakai, Senda, T., Hata, R., Kuroda, M., Hasegawa, M., Kato, M., Abe, M., Kawaguchi, K., Nakai, S., Hiki, Y., Yuzawa, Y., and Kitaguchi, N., “Patients that have Undergone Hemodialysis Exhibit Lower Amyloid Deposition in the Brain: Evidence Supporting a Therapeutic Strategy for Alzheimer's Disease by Removal of Blood Amyloid.”, J Alzheimers Dis, vol. 51, no. 4, pp. 997-1002, 2016.
B. Hanseeuw, Dricot, L., Lhommel, R., Quenon, L., and Ivanoiu, A., “Patients with Amyloid-Negative Mild Cognitive Impairment have Cortical Hypometabolism but the Hippocampus is Preserved.”, J Alzheimers Dis, vol. 53, no. 2, pp. 651-60, 2016.

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