Biblio
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“Quantitative PET and Histology of Brain Biopsy Reveal Lack of Selective Pittsburgh Compound-B Binding to Intracerebral Amyloidoma.”, J Alzheimers Dis, 2018.
, “Rationale and Design of the Mechanistic Potential of Antihypertensives in Preclinical Alzheimer's (HEART) Trial.”, J Alzheimers Dis, vol. 61, no. 2, pp. 815-824, 2018.
, “The Rationale and Design of the Reducing Pathology in Alzheimer's Disease through Angiotensin TaRgeting (RADAR) Trial.”, J Alzheimers Dis, vol. 61, no. 2, pp. 803-814, 2018.
, “Reaction of Amyloid-β Peptide Antibody with Different Infectious Agents Involved in Alzheimer's Disease.”, J Alzheimers Dis, vol. 63, no. 2, pp. 847-860, 2018.
, “Real-World, Multinational, Retrospective Observational Survey of the ADAS-Cog and Associations with Healthcare Resource Utilization in Patients with Alzheimer's Disease.”, J Alzheimers Dis, vol. 64, no. 3, pp. 899-910, 2018.
, “Regional White Matter Hyperintensity Influences Grey Matter Atrophy in Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 66, no. 2, pp. 533-549, 2018.
, “Relationship Between Body Mass Index, ApoE4 Status, and PET-Based Amyloid and Neurodegeneration Markers in Amyloid-Positive Subjects with Normal Cognition or Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 65, no. 3, pp. 781-791, 2018.
, “Relationship of Area of Soft Drusen in Retina with Cerebral Amyloid-β Accumulation and Blood Amyloid-β Level in the Elderly.”, J Alzheimers Dis, vol. 62, no. 1, pp. 239-245, 2018.
, “Relationships between Cognition and Activities of Daily Living in Alzheimer's Disease During a 5-Year Follow-Up: ALSOVA Study.”, J Alzheimers Dis, vol. 64, no. 1, pp. 269-279, 2018.
, “Relevance of Coded Prodromal Mild Cognitive Impairment in the Routine Treatment of Patients with Dementia in Germany.”, J Alzheimers Dis, vol. 65, no. 2, pp. 393-399, 2018.
, “The Relevance of Iron in the Pathogenesis of Multiple System Atrophy: A Viewpoint.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1253-1273, 2018.
, “Reproducibility of Alzheimer's Disease Cerebrospinal Fluid-Biomarker Measurements under Clinical Routine Conditions.”, J Alzheimers Dis, vol. 62, no. 1, pp. 203-212, 2018.
, “Revolution of Alzheimer Precision Neurology. Passageway of Systems Biology and Neurophysiology.”, J Alzheimers Dis, vol. 64, no. s1, pp. S47-S105, 2018.
, “Revolution of Alzheimer Precision Neurology. Passageway of Systems Biology and Neurophysiology.”, J Alzheimers Dis, vol. 64, no. s1, pp. S47-S105, 2018.
, “Risk Factors Associated with Cortical Thickness and White Matter Hyperintensities in Dementia Free Okinawan Elderly.”, J Alzheimers Dis, vol. 63, no. 1, pp. 365-372, 2018.
, “Role of ASK1/p38 Cascade in a Mouse Model of Alzheimer's Disease and Brain Aging.”, J Alzheimers Dis, vol. 61, no. 1, pp. 259-263, 2018.
, “S-[18F]THK-5117-PET and [11C]PIB-PET Imaging in Idiopathic Normal Pressure Hydrocephalus in Relation to Confirmed Amyloid-β Plaques and Tau in Brain Biopsies.”, J Alzheimers Dis, vol. 64, no. 1, pp. 171-179, 2018.
, “S-[18F]THK-5117-PET and [11C]PIB-PET Imaging in Idiopathic Normal Pressure Hydrocephalus in Relation to Confirmed Amyloid-β Plaques and Tau in Brain Biopsies.”, J Alzheimers Dis, vol. 64, no. 1, pp. 171-179, 2018.
, “Sally-Anne Test in Patients with Alzheimer's Disease Dementia.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1029-1036, 2018.
, “Separating Symptomatic Alzheimer's Disease from Depression based on Structural MRI.”, J Alzheimers Dis, vol. 63, no. 1, pp. 353-363, 2018.
, “Separating Symptomatic Alzheimer's Disease from Depression based on Structural MRI.”, J Alzheimers Dis, vol. 63, no. 1, pp. 353-363, 2018.
, “Serotonin Selective Reuptake Inhibitor Treatment Improves Cognition and Grey Matter Atrophy but not Amyloid Burden During Two-Year Follow-Up in Mild Cognitive Impairment and Alzheimer's Disease Patients with Depressive Symptoms.”, J Alzheimers Dis, vol. 65, no. 3, pp. 793-806, 2018.
, “Sex-Dependent Differences in Spontaneous Autoimmunity in Adult 3xTg-AD Mice.”, J Alzheimers Dis, vol. 63, no. 3, pp. 1191-1205, 2018.
, “Sex-Related Reserve Hypothesis in Alzheimer's Disease: Changes in Cortical Thickness with a Five-Year Longitudinal Follow-Up.”, J Alzheimers Dis, vol. 65, no. 2, pp. 641-649, 2018.
, “Sex-Related Reserve Hypothesis in Alzheimer's Disease: Changes in Cortical Thickness with a Five-Year Longitudinal Follow-Up.”, J Alzheimers Dis, vol. 65, no. 2, pp. 641-649, 2018.
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