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“Cost Related to Dementia in the Young and the Impact of Etiological Subtype on Cost.”, J Alzheimers Dis, vol. 49, no. 2, pp. 277-85, 2016.
, “Cost Related to Dementia in the Young and the Impact of Etiological Subtype on Cost.”, J Alzheimers Dis, vol. 49, no. 2, pp. 277-85, 2016.
, “Cycloheximide Treatment Causes a ZVAD-Sensitive Protease-Dependent Cleavage of Human Tau in Drosophila Cells.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1161-8, 2016.
, “Daily Function as Predictor of Dementia in Cognitive Impairment, No Dementia (CIND) and Mild Cognitive Impairment (MCI): An 8-Year Follow-Up in the ILSA Study.”, J Alzheimers Dis, vol. 53, no. 2, pp. 505-15, 2016.
, “A Decade of Cerebrospinal Fluid Biomarkers for Alzheimer's Disease in Belgium.”, J Alzheimers Dis, vol. 54, no. 1, pp. 383-95, 2016.
, “Decreased Inter-Hemispheric Functional Connectivity in Cognitively Intact Elderly APOE ɛ4 Carriers: A Preliminary Study.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1137-48, 2016.
, “Delusions in Patients with Alzheimer's Disease: A Multidimensional Approach.”, J Alzheimers Dis, vol. 51, no. 2, pp. 427-37, 2016.
, “Dementia and Rapid Mortality: Who is at Risk?”, J Alzheimers Dis, vol. 53, no. 1, pp. 135-42, 2016.
, “Dementia Apraxia Test (DATE): A Brief Tool to Differentiate Behavioral Variant Frontotemporal Dementia from Alzheimer's Dementia Based on Apraxia Profiles.”, J Alzheimers Dis, vol. 49, no. 3, pp. 593-605, 2016.
, “Describing the Sequence of Cognitive Decline in Alzheimer's Disease Patients: Results from an Observational Study.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1065-80, 2016.
, “Detection of Alzheimer's Disease by Three-Dimensional Displacement Field Estimation in Structural Magnetic Resonance Imaging.”, J Alzheimers Dis, vol. 50, no. 1, pp. 233-48, 2016.
, “Developing Cognitive Markers of Alzheimer's Disease for Primary Care: Implications for Behavioral and Global Prevention.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1259-1272, 2016.
, “Development of a Standardized Approach to Disclosing Amyloid Imaging Research Results in Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 52, no. 1, pp. 17-24, 2016.
, “Diagnosing Dementia in the Clinical Setting: Can Amyloid PET Provide Additional Value Over Cerebrospinal Fluid?”, J Alzheimers Dis, vol. 54, no. 4, pp. 1297-1302, 2016.
, “Diagnosis of Dementia in the Specialist Setting: A Comparison Between the Swedish Dementia Registry (SveDem) and the Registry of Dementias of Girona (ReDeGi).”, J Alzheimers Dis, vol. 53, no. 4, pp. 1341-51, 2016.
, “Diagnostic Value of Cerebrospinal Fluid Biomarkers (Phospho-Tau181, total-Tau, Aβ42, and Aβ40) in Prodromal Stage of Alzheimer's Disease and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1069-83, 2016.
, “Differential Diagnosis of Dementia with High Levels of Cerebrospinal Fluid Tau Protein.”, J Alzheimers Dis, vol. 51, no. 3, pp. 905-13, 2016.
, “Differential Diagnosis of Dementia with High Levels of Cerebrospinal Fluid Tau Protein.”, J Alzheimers Dis, vol. 51, no. 3, pp. 905-13, 2016.
, “Differential Effects of APOE Genotypes on the Anterior and Posterior Subnetworks of Default Mode Network in Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1409-1423, 2016.
, “Differential Effects of APOE Genotypes on the Anterior and Posterior Subnetworks of Default Mode Network in Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1409-1423, 2016.
, “Differential Impairment of Cognitive and Affective Mentalizing Abilities in Neurodegenerative Dementias: Evidence from Behavioral Variant of Frontotemporal Dementia, Alzheimer's Disease, and Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1011-22, 2016.
, “Differential Mass Spectrometry Profiles of Tau Protein in the Cerebrospinal Fluid of Patients with Alzheimer's Disease, Progressive Supranuclear Palsy, and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1033-43, 2016.
, “Differential Regulation of N-Methyl-D-Aspartate Receptor Subunits is an Early Event in the Actions of Soluble Amyloid-β(1-40) Oligomers on Hippocampal Neurons.”, J Alzheimers Dis, vol. 51, no. 1, pp. 197-212, 2016.
, “Differential Regulation of N-Methyl-D-Aspartate Receptor Subunits is an Early Event in the Actions of Soluble Amyloid-β(1-40) Oligomers on Hippocampal Neurons.”, J Alzheimers Dis, vol. 51, no. 1, pp. 197-212, 2016.
, “Differential Regulation of N-Methyl-D-Aspartate Receptor Subunits is an Early Event in the Actions of Soluble Amyloid-β(1-40) Oligomers on Hippocampal Neurons.”, J Alzheimers Dis, vol. 51, no. 1, pp. 197-212, 2016.
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