Biblio

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2018
X. Yin, Jiang, X., Wang, J., Qian, S., Liu, F., and Qian, W., “SIRT1 Deacetylates SC35 and Suppresses Its Function in Tau Exon 10 Inclusion.”, J Alzheimers Dis, vol. 61, no. 2, pp. 561-570, 2018.
X. Yin, Jiang, X., Wang, J., Qian, S., Liu, F., and Qian, W., “SIRT1 Deacetylates SC35 and Suppresses Its Function in Tau Exon 10 Inclusion.”, J Alzheimers Dis, vol. 61, no. 2, pp. 561-570, 2018.
T. M. Scott, Bhadelia, R. A., Qiu, W. Qiao, Folstein, M. F., and Rosenberg, I. H., “Small Vessel Cerebrovascular Pathology Identified by Magnetic Resonance Imaging Is Prevalent in Alzheimer's Disease and Mild Cognitive Impairment: A Potential Target for Intervention.”, J Alzheimers Dis, vol. 65, no. 1, pp. 293-302, 2018.
M. Lozupone, Panza, F., Piccininni, M., Copetti, M., Sardone, R., Imbimbo, B. P., Stella, E., D'Urso, F., Barulli, M. Rosaria, Battista, P., Grasso, A., Tortelli, R., Capozzo, R., Coppola, F., Abbrescia, D. Isabel, Bellomo, A., Giannelli, G., Quaranta, N., Seripa, D., and Logroscino, G., “Social Dysfunction in Older Age and Relationships with Cognition, Depression, and Apathy: The GreatAGE Study.”, J Alzheimers Dis, vol. 65, no. 3, pp. 989-1000, 2018.
F. Chao, Jiang, L., Zhang, Y., Zhou, C., Zhang, L., Tang, J., Liang, X., Qi, Y., Zhu, Y., Ma, J., and Tang, Y., “Stereological Investigation of the Effects of Treadmill Running Exercise on the Hippocampal Neurons in Middle-Aged APP/PS1 Transgenic Mice.”, J Alzheimers Dis, vol. 63, no. 2, pp. 689-703, 2018.
Y. - T. Dong, Cao, K., Tan, L. - C., Wang, X. - L., Qi, X. - L., Xiao, Y., and Guan, Z. - Z., “Stimulation of SIRT1 Attenuates the Level of Oxidative Stress in the Brains of APP/PS1 Double Transgenic Mice and in Primary Neurons Exposed to Oligomers of the Amyloid-β Peptide.”, J Alzheimers Dis, vol. 63, no. 1, pp. 283-301, 2018.
J. P. Quinn, Corbett, N. J., Kellett, K. A. B., and Hooper, N. M., “Tau Proteolysis in the Pathogenesis of Tauopathies: Neurotoxic Fragments and Novel Biomarkers.”, J Alzheimers Dis, vol. 63, no. 1, pp. 13-33, 2018.
E. Guzmán-Vélez, Jaimes, S., Aguirre-Acevedo, D. C., Norton, D. J., Papp, K. V., Amariglio, R., Rentz, D., Baena, A., Henao, E., Tirado, V., Munoz, C., Giraldo, M., Sperling, R. A., Lopera, F., and Quiroz, Y. T., “A Three-Factor Structure of Cognitive Functioning Among Unimpaired Carriers and Non-Carriers of Autosomal-Dominant Alzheimer's Disease.”, J Alzheimers Dis, vol. 65, no. 1, pp. 107-115, 2018.
C. Lage, Suarez, A. Gonzalez, Pozueta, A., Riancho, J., Kazimierczak, M., Bravo, M., Bonilla, J. Jimenez, Torres, Mde Arcocha, Quirce, R., Banzo, I., Vázquez-Higuera, J. Luis, Rabinovici, G. D., Rodriguez-Rodriguez, E., and Sánchez-Juan, P., “Utility of Amyloid and FDG-PET in Clinical Practice: Differences Between Secondary and Tertiary Care Memory Units.”, J Alzheimers Dis, vol. 63, no. 3, pp. 1025-1033, 2018.
2017
L. Zhang, Wang, Y., Xiayu, X., Shi, C., Chen, W., Song, N., Fu, X., Zhou, R., Xu, Y. - F., Huang, L., Zhu, H., Han, Y., and Qin, C., “Altered Gut Microbiota in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1241-1257, 2017.
L. A. Mohamed, Zhu, H., Mousa, Y. M., Wang, E., Qiu, W. Qiao, and Kaddoumi, A., “Amylin Enhances Amyloid-β Peptide Brain to Blood Efflux Across the Blood-Brain Barrier.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1087-1099, 2017.
E. Wang, Zhu, H., Wang, X., Gower, A., Wallack, M., Blusztajn, J. Krzysztof, Kowall, N., and Qiu, W. Qiao, “Amylin Treatment Reduces Neuroinflammation and Ameliorates Abnormal Patterns of Gene Expression in the Cerebral Cortex of an Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 56, no. 1, pp. 47-61, 2017.
M. Wu, Shi, H., He, Y., Yuan, L., Qu, X., Zhang, J., Wang, Z., Cai, H., and Qi, J., “Colivelin Ameliorates Impairments in Cognitive Behaviors and Synaptic Plasticity in APP/PS1 Transgenic Mice.”, J Alzheimers Dis, vol. 59, no. 3, pp. 1067-1078, 2017.
M. Wu, Shi, H., He, Y., Yuan, L., Qu, X., Zhang, J., Wang, Z., Cai, H., and Qi, J., “Colivelin Ameliorates Impairments in Cognitive Behaviors and Synaptic Plasticity in APP/PS1 Transgenic Mice.”, J Alzheimers Dis, vol. 59, no. 3, pp. 1067-1078, 2017.
L. Zhang, Sun, C., Jin, Y., Gao, K., Shi, X., Qiu, W., Ma, C., and Zhang, L., “Dickkopf 3 (Dkk3) Improves Amyloid-β Pathology, Cognitive Dysfunction, and Cerebral Glucose Metabolism in a Transgenic Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 2, pp. 733-746, 2017.
J. Wang, Liu, Y., Cheng, X., Zhang, X., Liu, F., Liu, G., Qiao, S., Ni, M., Zhou, W., Zhang, Y., and Li, F., “The Effects of LW-AFC on the Hippocampal Transcriptome in Senescence-Accelerated Mouse Prone 8 Strain, a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 57, no. 1, pp. 227-240, 2017.
Y. Jayne Tan, Ng, A. S. L., Vipin, A., Lim, J. K. W., Chander, R. J., Ji, F., Qiu, Y., Ting, S. K. S., Hameed, S., Lee, T. - S., Zeng, L., Kandiah, N., and Zhou, J., “Higher Peripheral TREM2 mRNA Levels Relate to Cognitive Deficits and Hippocampal Atrophy in Alzheimer's Disease and Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 58, no. 2, pp. 413-423, 2017.
T. A. Lusardi, Phillips, J. I., Wiedrick, J. T., Harrington, C. A., Lind, B., Lapidus, J. A., Quinn, J. F., and Saugstad, J. A., “MicroRNAs in Human Cerebrospinal Fluid as Biomarkers for Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1223-1233, 2017.
H. - K. Lee, Kwon, B., Lemere, C. A., de la Monte, S., Itamura, K., Ha, A. Y., and Querfurth, H. W., “mTORC2 (Rictor) in Alzheimer's Disease and Reversal of Amyloid-β Expression-Induced Insulin Resistance and Toxicity in Rat Primary Cortical Neurons.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1015-1036, 2017.
J. Dong, Qin, W., Wei, C., Tang, Y., Wang, Q., and Jia, J., “A Novel PSEN1 K311R Mutation Discovered in Chinese Families with Late-Onset Alzheimer's Disease Affects Amyloid-β Production and Tau Phosphorylation.”, J Alzheimers Dis, vol. 57, no. 2, pp. 613-623, 2017.
M. Quarmley, Moberg, P. J., Mechanic-Hamilton, D., Kabadi, S., Arnold, S. E., Wolk, D. A., and Roalf, D. R., “Odor Identification Screening Improves Diagnostic Classification in Incipient Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1497-1507, 2017.
O. O. Adegoke, Qiao, F., Liu, Y., Longley, K., Feng, S., and Wang, H., “Overexpression of Ubiquilin-1 Alleviates Alzheimer's Disease-Caused Cognitive and Motor Deficits and Reduces Amyloid-β Accumulation in Mice.”, J Alzheimers Dis, vol. 59, no. 2, pp. 575-590, 2017.
K. Li, Chan, W., Doody, R. S., Quinn, J., and Luo, S., “Prediction of Conversion to Alzheimer's Disease with Longitudinal Measures and Time-To-Event Data.”, J Alzheimers Dis, vol. 58, no. 2, pp. 361-371, 2017.
M. Wei, Zhao, B., Huo, K., Deng, Y., Shang, S., Liu, J., Li, Y., Ma, L., Jiang, Y., Dang, L., Chen, C., Wei, S., Zhang, J., Yang, H., Gao, F., and Qu, Q., “Sleep Deprivation Induced Plasma Amyloid-β Transport Disturbance in Healthy Young Adults.”, J Alzheimers Dis, vol. 57, no. 3, pp. 899-906, 2017.
B. Garcia, Martin, C., Garcia-Suarez, O., Muniz-Alonso, B., Ordiales, H., Fernandez-Menendez, S., Santos-Juanes, J., Lorente-Gea, L., Castanon, S., Vicente-Etxenausia, I., Batista, K. Manuel Pin, Ruiz-Diaz, I., Caballero-Martinez, M. Cristina, Merayo-Lloves, J., Guerra-Merino, I., Quiros, L. M., and Fernandez-Vega, I., “Upregulated Expression of Heparanase and Heparanase 2 in the Brains of Alzheimer's Disease.”, J Alzheimers Dis, vol. 58, no. 1, pp. 185-192, 2017.

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