Biblio

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2022
D. Asaoka, Xiao, J., Takeda, T., Yanagisawa, N., Yamazaki, T., Matsubara, Y., Sugiyama, H., Endo, N., Higa, M., Kasanuki, K., Ichimiya, Y., Koido, S., Ohno, K., Bernier, F., Katsumata, N., Nagahara, A., Arai, H., Ohkusa, T., and Sato, N., “Effect of Probiotic Bifidobacterium breve in Improving Cognitive Function and Preventing Brain Atrophy in Older Patients with Suspected Mild Cognitive Impairment: Results of a 24-Week Randomized, Double-Blind, Placebo-Controlled Trial.”, J Alzheimers Dis, vol. 88, no. 1, pp. 75-95, 2022.
F. Fan, Liu, H., Shi, X., Ai, Y., Liu, Q., and Cheng, Y., “The Efficacy and Safety of Alzheimer's Disease Therapies: An Updated Umbrella Review.”, J Alzheimers Dis, vol. 85, no. 3, pp. 1195-1204, 2022.
F. J. Castora, Kerns, K. A., Pflanzer, H. K., Hitefield, N. L., Gershon, B., Shugoll, J., Shelton, M., and Coleman, R. A., “Expression Changes in Mitochondrial Genes Affecting Mitochondrial Morphology, Transmembrane Potential, Fragmentation, Amyloidosis, and Neuronal Cell Death Found in Brains of Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 90, no. 1, pp. 119-137, 2022.
F. J. Castora, Kerns, K. A., Pflanzer, H. K., Hitefield, N. L., Gershon, B., Shugoll, J., Shelton, M., and Coleman, R. A., “Expression Changes in Mitochondrial Genes Affecting Mitochondrial Morphology, Transmembrane Potential, Fragmentation, Amyloidosis, and Neuronal Cell Death Found in Brains of Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 90, no. 1, pp. 119-137, 2022.
M. Gentreau, Reynes, C., Sabatier, R., Maller, J. J., Meslin, C., Deverdun, J., Le Bars, E., Raymond, M., Berticat, C., and Artero, S., “Glucometabolic Changes Are Associated with Structural Gray Matter Alterations in Prodromal Dementia.”, J Alzheimers Dis, vol. 89, no. 4, pp. 1293-1302, 2022.
A. Sturchio, Dwivedi, A. K., Malm, T., Wood, M. J. A., Cilia, R., Sharma, J. S., Hill, E. J., Schneider, L. S., Graff-Radford, N. R., Mori, H., Nübling, G., Andaloussi, S. El, Svenningsson, P., Ezzat, K., and Espay, A. J., “High Soluble Amyloid-β42 Predicts Normal Cognition in Amyloid-Positive Individuals with Alzheimer's Disease-Causing Mutations.”, J Alzheimers Dis, vol. 90, no. 1, pp. 333-348, 2022.
A. Sturchio, Dwivedi, A. K., Malm, T., Wood, M. J. A., Cilia, R., Sharma, J. S., Hill, E. J., Schneider, L. S., Graff-Radford, N. R., Mori, H., Nübling, G., Andaloussi, S. El, Svenningsson, P., Ezzat, K., and Espay, A. J., “High Soluble Amyloid-β42 Predicts Normal Cognition in Amyloid-Positive Individuals with Alzheimer's Disease-Causing Mutations.”, J Alzheimers Dis, vol. 90, no. 1, pp. 333-348, 2022.
A. Sturchio, Dwivedi, A. K., Malm, T., Wood, M. J. A., Cilia, R., Sharma, J. S., Hill, E. J., Schneider, L. S., Graff-Radford, N. R., Mori, H., Nübling, G., Andaloussi, S. El, Svenningsson, P., Ezzat, K., and Espay, A. J., “High Soluble Amyloid-β42 Predicts Normal Cognition in Amyloid-Positive Individuals with Alzheimer's Disease-Causing Mutations.”, J Alzheimers Dis, vol. 90, no. 1, pp. 333-348, 2022.
A. Sturchio, Dwivedi, A. K., Malm, T., Wood, M. J. A., Cilia, R., Sharma, J. S., Hill, E. J., Schneider, L. S., Graff-Radford, N. R., Mori, H., Nübling, G., Andaloussi, S. El, Svenningsson, P., Ezzat, K., and Espay, A. J., “High Soluble Amyloid-β42 Predicts Normal Cognition in Amyloid-Positive Individuals with Alzheimer's Disease-Causing Mutations.”, J Alzheimers Dis, vol. 90, no. 1, pp. 333-348, 2022.
L. - T. Hu, Xie, X. - Y., Zhou, G. - F., Wen, Q. - X., Song, L., Luo, B., Deng, X. - J., Pan, Q. - L., and Chen, G. - J., “HMGCS2-Induced Autophagic Degradation of Tau Involves Ketone Body and ANKRD24.”, J Alzheimers Dis, vol. 91, no. 1, pp. 407-426, 2022.
J. Laton, Van Schependom, J., Goossens, J., Wiels, W., Sieben, A., De Deyn, P. Paul, Goeman, J., Streffer, J., van der Zee, J., Martin, J. - J., Van Broeckhoven, C., De Vos, M., Bjerke, M., Nagels, G., and Engelborghs, S., “Improved Alzheimer's Disease versus Frontotemporal Lobar Degeneration Differential Diagnosis Combining EEG and Neurochemical Biomarkers: A Pilot Study.”, J Alzheimers Dis, vol. 90, no. 4, pp. 1739-1747, 2022.
J. Laton, Van Schependom, J., Goossens, J., Wiels, W., Sieben, A., De Deyn, P. Paul, Goeman, J., Streffer, J., van der Zee, J., Martin, J. - J., Van Broeckhoven, C., De Vos, M., Bjerke, M., Nagels, G., and Engelborghs, S., “Improved Alzheimer's Disease versus Frontotemporal Lobar Degeneration Differential Diagnosis Combining EEG and Neurochemical Biomarkers: A Pilot Study.”, J Alzheimers Dis, vol. 90, no. 4, pp. 1739-1747, 2022.
J. Wu, Su, Y., Zhu, W., Mallak, N. Jalili, Lepore, N., Reiman, E. M., Caselli, R. J., Thompson, P. M., Chen, K., and Wang, Y., “Improved Prediction of Amyloid-β and Tau Burden Using Hippocampal Surface Multivariate Morphometry Statistics and Sparse Coding.”, J Alzheimers Dis, vol. 91, no. 2, pp. 637-651, 2022.
Z. D. Green, Vidoni, E. D., Swerdlow, R. H., Burns, J. M., Morris, J. K., and Honea, R. A., “Increased Functional Connectivity of the Precuneus in Individuals with a Family History of Alzheimer's Disease.”, J Alzheimers Dis, vol. 91, no. 2, pp. 559-571, 2022.
K. Wittfeld, Raman, M. R., Conner, S. C., Aslam, A., Teumer, A., Nauck, M., Hosten, N., Habes, M., DeCarli, C., Vasan, R. S., Beiser, A. S., Himali, J. J., Seshadri, S., Grabe, H. J., and Satizabal, C. L., “Insulin-Like Growth Factor, Inflammation, and MRI Markers of Alzheimer's Disease in Predominantly Middle-Aged Adults.”, J Alzheimers Dis, vol. 88, no. 1, pp. 311-32, 2022.
K. Wittfeld, Raman, M. R., Conner, S. C., Aslam, A., Teumer, A., Nauck, M., Hosten, N., Habes, M., DeCarli, C., Vasan, R. S., Beiser, A. S., Himali, J. J., Seshadri, S., Grabe, H. J., and Satizabal, C. L., “Insulin-Like Growth Factor, Inflammation, and MRI Markers of Alzheimer's Disease in Predominantly Middle-Aged Adults.”, J Alzheimers Dis, vol. 88, no. 1, pp. 311-32, 2022.
A. Ticinesi, Mancabelli, L., Carnevali, L., Nouvenne, A., Meschi, T., Del Rio, D., Ventura, M., Sgoifo, A., and Angelino, D., “Interaction Between Diet and Microbiota in the Pathophysiology of Alzheimer's Disease: Focus on Polyphenols and Dietary Fibers.”, J Alzheimers Dis, vol. 86, no. 3, pp. 961-982, 2022.
S. - Y. Huang, Yang, Y. - X., Zhang, Y. - R., Kuo, K., Li, H. - Q., Shen, X. - N., Chen, S. - D., Chen, K. - L., Dong, Q., Tan, L., and Yu, J. - T., “Investigating Causal Relations Between Circulating Metabolites and Alzheimer's Disease: A Mendelian Randomization Study.”, J Alzheimers Dis, vol. 87, no. 1, pp. 463-477, 2022.
Ede Paula F. Resende, Ketelle, R., Karydas, A., Allen, I., Grinberg, L. T., Spina, S., Seeley, W. W., Perry, D. C., Miller, B., and Naasan, G., “Late-Onset Alcohol Abuse as a Presenting Symptom of Neurodegenerative Diseases.”, J Alzheimers Dis, vol. 86, no. 3, pp. 1073-1080, 2022.
Ede Paula F. Resende, Ketelle, R., Karydas, A., Allen, I., Grinberg, L. T., Spina, S., Seeley, W. W., Perry, D. C., Miller, B., and Naasan, G., “Late-Onset Alcohol Abuse as a Presenting Symptom of Neurodegenerative Diseases.”, J Alzheimers Dis, vol. 86, no. 3, pp. 1073-1080, 2022.
K. K. Bawa, Ba, J., Kiss, A., Wang, R. D., Feng, V., Swardfager, W., Andreazza, A., Gallagher, D., Marotta, G., Herrmann, N., and Lanctôt, K. L., “Lipid Peroxidation as a Marker of Apathy and Executive Dysfunction in Patients at Risk for Vascular Cognitive Impairment.”, J Alzheimers Dis, vol. 89, no. 2, pp. 733-743, 2022.
T. Kawarabayashi, Nakamura, T., Sato, K., Seino, Y., Ichii, S., Nakahata, N., Takatama, M., Westaway, D., St George-Hyslop, P., and Shoji, M., “Lipid Rafts Act as a Common Platform for Amyloid-β Oligomer-Induced Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 87, no. 3, pp. 1189-1203, 2022.
T. Kawarabayashi, Nakamura, T., Sato, K., Seino, Y., Ichii, S., Nakahata, N., Takatama, M., Westaway, D., St George-Hyslop, P., and Shoji, M., “Lipid Rafts Act as a Common Platform for Amyloid-β Oligomer-Induced Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 87, no. 3, pp. 1189-1203, 2022.
T. Kawarabayashi, Nakamura, T., Sato, K., Seino, Y., Ichii, S., Nakahata, N., Takatama, M., Westaway, D., St George-Hyslop, P., and Shoji, M., “Lipid Rafts Act as a Common Platform for Amyloid-β Oligomer-Induced Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 87, no. 3, pp. 1189-1203, 2022.
T. Kawarabayashi, Nakamura, T., Sato, K., Seino, Y., Ichii, S., Nakahata, N., Takatama, M., Westaway, D., St George-Hyslop, P., and Shoji, M., “Lipid Rafts Act as a Common Platform for Amyloid-β Oligomer-Induced Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 87, no. 3, pp. 1189-1203, 2022.

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