Biblio
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“Human ApoE ɛ2 Promotes Regulatory Mechanisms of Bioenergetic and Synaptic Function in Female Brain: A Focus on V-type H+-ATPase.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1015-31, 2016.
, “Human Truncated Tau Induces Mature Neurofibrillary Pathology in a Mouse Model of Human Tauopathy.”, J Alzheimers Dis, vol. 54, no. 2, pp. 831-43, 2016.
, “"I Don't Think Of It As An Illness": Illness Representations in Mild to Moderate Dementia.”, J Alzheimers Dis, vol. 51, no. 1, pp. 139-50, 2016.
, “Identification of Physician-Diagnosed Alzheimer's Disease and Related Dementias in Population-Based Administrative Data: A Validation Study Using Family Physicians' Electronic Medical Records.”, J Alzheimers Dis, vol. 54, no. 1, pp. 337-49, 2016.
, “Immunization with Small Amyloid-β-derived Cyclopeptide Conjugates Diminishes Amyloid-β-Induced Neurodegeneration in Mice.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1111-23, 2016.
, “Impact of Omega-3 Fatty Acid Supplementation on Memory Functions in Healthy Older Adults.”, J Alzheimers Dis, vol. 51, no. 3, pp. 713-25, 2016.
, “Impacts of High Serum Total Cholesterol Level on Brain Functional Connectivity in Non-Demented Elderly.”, J Alzheimers Dis, vol. 50, no. 2, pp. 455-63, 2016.
, “Increase of α-Secretase ADAM10 in Platelets Along Cognitively Healthy Aging.”, J Alzheimers Dis, vol. 50, no. 3, pp. 817-26, 2016.
, “Increased Intrinsic Activity of Medial-Temporal Lobe Subregions is Associated with Decreased Cortical Thickness of Medial-Parietal Areas in Patients with Alzheimer's Disease Dementia.”, J Alzheimers Dis, vol. 51, no. 1, pp. 313-26, 2016.
, “Increased Levels of Extracellular Microvesicle Markers and Decreased Levels of Endocytic/Exocytic Proteins in the Alzheimer's Disease Brain.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1671-1686, 2016.
, “Inequalities in Access to Treatment and Care for Patients with Dementia and Immigrant Background: A Danish Nationwide Study.”, J Alzheimers Dis, vol. 54, no. 2, pp. 505-14, 2016.
, “Insight into the Dissociation of Behavior from Histology in Synucleinopathies and in Related Neurodegenerative Diseases.”, J Alzheimers Dis, vol. 52, no. 3, pp. 831-41, 2016.
, “Integrated Analysis of Alzheimer's Disease and Schizophrenia Dataset Revealed Different Expression Pattern in Learning and Memory.”, J Alzheimers Dis, vol. 51, no. 2, pp. 417-25, 2016.
, “Interaction Between Midlife Blood Glucose and APOE Genotype Predicts Later Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1553-62, 2016.
, “The Language Profile of Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 50, no. 2, pp. 359-71, 2016.
, “The Language Profile of Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 50, no. 2, pp. 359-71, 2016.
, “A Late Life Risk Index for Severe Cognitive Impairment in Mexico.”, J Alzheimers Dis, vol. 52, no. 1, pp. 191-203, 2016.
, “Late-Life Depressive Symptoms and Lifetime History of Major Depression: Cognitive Deficits are Largely Due to Incipient Dementia rather than Depression.”, J Alzheimers Dis, vol. 54, no. 1, pp. 185-99, 2016.
, “Late-Life Depressive Symptoms and Lifetime History of Major Depression: Cognitive Deficits are Largely Due to Incipient Dementia rather than Depression.”, J Alzheimers Dis, vol. 54, no. 1, pp. 185-99, 2016.
, “Late-Life Depressive Symptoms and Lifetime History of Major Depression: Cognitive Deficits are Largely Due to Incipient Dementia rather than Depression.”, J Alzheimers Dis, vol. 54, no. 1, pp. 185-99, 2016.
, “Late-Life Depressive Symptoms and Lifetime History of Major Depression: Cognitive Deficits are Largely Due to Incipient Dementia rather than Depression.”, J Alzheimers Dis, vol. 54, no. 1, pp. 185-99, 2016.
, “Late-Life Depressive Symptoms and Lifetime History of Major Depression: Cognitive Deficits are Largely Due to Incipient Dementia rather than Depression.”, J Alzheimers Dis, vol. 54, no. 1, pp. 185-99, 2016.
, “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.
, “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.
, “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.
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