Biblio

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2024
J. Liang, LaFleur, B., Hussainy, S., and Perry, G., “Gene Co-Expression Analysis of Multiple Brain Tissues Reveals Correlation of FAM222A Expression with Multiple Alzheimer's Disease-Related Genes.”, J Alzheimers Dis, vol. 99, no. s2, pp. S249-S263, 2024.
2023
K. P. Kepp, Sensi, S. L., Johnsen, K. B., Barrio, J. R., Høilund-Carlsen, P. F., Neve, R. L., Alavi, A., Herrup, K., Perry, G., Robakis, N. K., Vissel, B., and Espay, A. J., “The Anti-Amyloid Monoclonal Antibody Lecanemab: 16 Cautionary Notes.”, J Alzheimers Dis, vol. 94, no. 2, pp. 497-507, 2023.
C. S. Atwood and Perry, G., “Playing Russian Roulette with Alzheimer's Disease Patients: Do the Cognitive Benefits of Lecanemab Outweigh the Risk of Edema, Stroke and Encephalitis?”, J Alzheimers Dis, vol. 92, no. 3, pp. 799-801, 2023.
2019
M. Liu, Wang, L., Gao, J., Dong, Q., Perry, G., Ma, X., and Wang, X., “Inhibition of Calpain Protects Against Tauopathy in Transgenic P301S Tau Mice.”, J Alzheimers Dis, vol. 69, no. 4, pp. 1077-1087, 2019.
R. J. Castellani and Perry, G., “Tau Biology, Tauopathy, Traumatic Brain Injury, and Diagnostic Challenges.”, J Alzheimers Dis, vol. 67, no. 2, pp. 447-467, 2019.
2018
M. Reale, D'Angelo, C., Costantini, E., Di Nicola, M., Yarla, N. Sastry, Kamal, M. Amjad, Salvador, N., and Perry, G., “Expression Profiling of Cytokine, Cholinergic Markers, and Amyloid-β Deposition in the APPSWE/PS1dE9 Mouse Model of Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 62, no. 1, pp. 467-476, 2018.
V. Vasquez, Mitra, J., Perry, G., Rao, K. S., and Hegde, M. L., “An Inducible Alpha-Synuclein Expressing Neuronal Cell Line Model for Parkinson's Disease1.”, J Alzheimers Dis, vol. 66, no. 2, pp. 453-460, 2018.
G. L. Iverson, C Keene, D., Perry, G., and Castellani, R. J., “The Need to Separate Chronic Traumatic Encephalopathy Neuropathology from Clinical Features.”, J Alzheimers Dis, vol. 61, no. 1, pp. 17-28, 2018.
M. A. Smith, Bowen, R. L., Nguyen, R. Q., Perry, G., Atwood, C. S., and Rimm, A. A., “Putative Gonadotropin-Releasing Hormone Agonist Therapy and Dementia: An Application of Medicare Hospitalization Claims Data.”, J Alzheimers Dis, vol. 63, no. 4, pp. 1269-1277, 2018.
T. Yan, Wang, L., Gao, J., Siedlak, S. L., Huntley, M. L., Termsarasab, P., Perry, G., Chen, S. G., and Wang, X., “Rab10 Phosphorylation is a Prominent Pathological Feature in Alzheimer's Disease.”, J Alzheimers Dis, vol. 63, no. 1, pp. 157-165, 2018.
2017
R. J. Castellani and Perry, G., “Dementia Pugilistica Revisited.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1209-1221, 2017.
R. H. L. H. Rueli, Torres, D. J., Dewing, A. S. T., Kiyohara, A. C., Barayuga, S. M., Bellinger, M. T., Uyehara-Lock, J. H., White, L. R., Moreira, P. I., Berry, M. J., Perry, G., and Bellinger, F. P., “Selenoprotein S Reduces Endoplasmic Reticulum Stress-Induced Phosphorylation of Tau: Potential Role in Selenate Mitigation of Tau Pathology.”, J Alzheimers Dis, vol. 55, no. 2, pp. 749-762, 2017.
S. L. Siedlak, Jiang, Y., Huntley, M. L., Wang, L., Gao, J., Xie, F., Liu, J., Su, B., Perry, G., and Wang, X., “TMEM230 Accumulation in Granulovacuolar Degeneration Bodies and Dystrophic Neurites of Alzheimer's Disease.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1027-1033, 2017.
2016
M. Ángel Ontiveros-Torres, Labra-Barrios, M. Luisa, Díaz-Cintra, S., Aguilar-Vázquez, A. Ruth, Moreno-Campuzano, S., Flores-Rodríguez, P., Luna-Herrera, C., Mena, R., Perry, G., Florán-Garduño, B., Luna-Muñoz, J., and Luna-Arias, J. Pedro, “Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse.”, J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
2012
L. Calderón-Garcidueñas, Kavanaugh, M., Block, M., D'Angiulli, A., Delgado-Chávez, R., Torres-Jardón, R., González-Maciel, A., Reynoso-Robles, R., Osnaya, N., Villarreal-Calderon, R., Guo, R., Hua, Z., Zhu, H., Perry, G., and Diaz, P., “Neuroinflammation, hyperphosphorylated tau, diffuse amyloid plaques, and down-regulation of the cellular prion protein in air pollution exposed children and young adults.”, J Alzheimers Dis, vol. 28, no. 1, pp. 93-107, 2012.