Biblio

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2018
J. R. Burrell, Ballard, K. J., Halliday, G. M., and Hodges, J. R., “Aphasia in Progressive Supranuclear Palsy: As Severe as Progressive Non-Fluent Aphasia.”, J Alzheimers Dis, vol. 61, no. 2, pp. 705-715, 2018.
J. R. Burrell, Ballard, K. J., Halliday, G. M., and Hodges, J. R., “Aphasia in Progressive Supranuclear Palsy: As Severe as Progressive Non-Fluent Aphasia.”, J Alzheimers Dis, vol. 61, no. 2, pp. 705-715, 2018.
J. R. Burrell, Ballard, K. J., Halliday, G. M., and Hodges, J. R., “Aphasia in Progressive Supranuclear Palsy: As Severe as Progressive Non-Fluent Aphasia.”, J Alzheimers Dis, vol. 61, no. 2, pp. 705-715, 2018.
R. M. Ahmed, Highton-Williamson, E., Caga, J., Thornton, N., Ramsey, E., Zoing, M., Kim, W. Scott, Halliday, G. M., Piguet, O., Hodges, J. R., I Farooqi, S., and Kiernan, M. C., “Lipid Metabolism and Survival Across the Frontotemporal Dementia-Amyotrophic Lateral Sclerosis Spectrum: Relationships to Eating Behavior and Cognition.”, J Alzheimers Dis, vol. 61, no. 2, pp. 773-783, 2018.
2016
S. Tu, Leyton, C. E., Hodges, J. R., Piguet, O., and Hornberger, M., “Divergent Longitudinal Propagation of White Matter Degradation in Logopenic and Semantic Variants of Primary Progressive Aphasia.”, J Alzheimers Dis, vol. 49, no. 3, pp. 853-61, 2016.
S. Hsieh, Leyton, C. E., Caga, J., Flanagan, E., Kaizik, C., O'Connor, C. M., Kiernan, M. C., Hodges, J. R., Piguet, O., and Mioshi, E., “The Evolution of Caregiver Burden in Frontotemporal Dementia with and without Amyotrophic Lateral Sclerosis.”, J Alzheimers Dis, vol. 49, no. 3, pp. 875-85, 2016.
F. Kumfor, Teo, D., Miller, L., Lah, S., Mioshi, E., Hodges, J. R., Piguet, O., and Irish, M., “Examining the Relationship Between Autobiographical Memory Impairment and Carer Burden in Dementia Syndromes.”, J Alzheimers Dis, vol. 51, no. 1, pp. 237-48, 2016.
S. Ramanan, Flanagan, E., Leyton, C. E., Villemagne, V. L., Rowe, C. C., Hodges, J. R., and Hornberger, M., “Non-Verbal Episodic Memory Deficits in Primary Progressive Aphasias are Highly Predictive of Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 51, no. 2, pp. 367-76, 2016.