Biblio

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A. Tramutola, Pupo, G., Di Domenico, F., Barone, E., Arena, A., Lanzillotta, C., Broekaart, D., Blarzino, C., Head, E., D Butterfield, A., and Perluigi, M., Activation of p53 in Down Syndrome and in the Ts65Dn Mouse Brain is Associated with a Pro-Apoptotic Phenotype., J Alzheimers Dis, vol. 52, no. 1, pp. 359-71, 2016.
N. Ortí-Casañ, Wajant, H., H Kuiperij, B., Hooijsma, A., Tromp, L., Poortman, I. L., Tadema, N., de Lange, J. H. E., Verbeek, M. M., De Deyn, P. P., Naudé, P. J. W., and Eisel, U. L. M., Activation of TNF Receptor 2 Improves Synaptic Plasticity and Enhances Amyloid-β Clearance in an Alzheimer's Disease Mouse Model with Humanized TNF Receptor 2., J Alzheimers Dis, vol. 94, no. 3, pp. 977-991, 2023.
B. Polis and Samson, A. O., Addressing the Discrepancies Between Animal Models and Human Alzheimer's Disease Pathology: Implications for Translational Research., J Alzheimers Dis, vol. 98, no. 4, pp. 1199-1218, 2024.
D. L. Alkon, Sun, M. - K., Tuchman, A. J., and Thompson, R. E., Advanced Alzheimer's Disease Patients Show Safe, Significant, and Persistent Benefit in 6-Month Bryostatin Trial., J Alzheimers Dis, vol. 96, no. 2, pp. 759-766, 2023.
T. Minh Do, Dodacki, A., Alata, W., Calon, F., Nicolic, S., Scherrmann, J. - M., Farinotti, R., and Bourasset, F., Age-Dependent Regulation of the Blood-Brain Barrier Influx/Efflux Equilibrium of Amyloid-β Peptide in a Mouse Model of Alzheimer's Disease (3xTg-AD)., J Alzheimers Dis, vol. 49, no. 2, pp. 287-300, 2016.
T. Minh Do, Dodacki, A., Alata, W., Calon, F., Nicolic, S., Scherrmann, J. - M., Farinotti, R., and Bourasset, F., Age-Dependent Regulation of the Blood-Brain Barrier Influx/Efflux Equilibrium of Amyloid-β Peptide in a Mouse Model of Alzheimer's Disease (3xTg-AD)., J Alzheimers Dis, vol. 49, no. 2, pp. 287-300, 2016.
J. L. King, Wong, A. A., and Brown, R. E., Age-Related Changes in the Spatial Frequency Threshold of Male and Female 3xTg-AD Mice Using OptoMotry., J Alzheimers Dis, vol. 62, no. 2, pp. 591-596, 2018.
Y. Shan, Wang, D. - D., Xu, Y. - X., Wang, C., Cao, L., Liu, Y. - S., and Zhu, C. - Q., Aging as a Precipitating Factor in Chronic Restraint Stress-Induced Tau Aggregation Pathology, and the Protective Effects of Rosmarinic Acid., J Alzheimers Dis, vol. 49, no. 3, pp. 829-44, 2016.
Y. Shan, Wang, D. - D., Xu, Y. - X., Wang, C., Cao, L., Liu, Y. - S., and Zhu, C. - Q., Aging as a Precipitating Factor in Chronic Restraint Stress-Induced Tau Aggregation Pathology, and the Protective Effects of Rosmarinic Acid., J Alzheimers Dis, vol. 49, no. 3, pp. 829-44, 2016.
R. Ali Syed, Hayat, M., Qaiser, H., Uzair, M., Al-Regaiey, K., Khallaf, R., Kaleem, I., and Bashir, S., Aging-Related Protein Alterations in the Brain., J Alzheimers Dis, vol. 99, no. s1, pp. S5-S22, 2024.
S. J. Czuczwar, Kocki, J., Miziak, B., Bogucki, J., Bogucka-Kocka, A., and Pluta, R., Alpha-, Beta-, and Gamma-Secretase, Amyloid Precursor Protein, and Tau Protein Genes in the Hippocampal CA3 Subfield in an Ischemic Model of Alzheimer's Disease with Survival up to 2 Years., J Alzheimers Dis, vol. 98, no. 1, pp. 151-161, 2024.
C. Grobler, van Tongeren, M., Gettemans, J., Kell, D. B., and Pretorius, E., Alzheimer's Disease: A Systems View Provides a Unifying Explanation of Its Development., J Alzheimers Dis, vol. 91, no. 1, pp. 43-70, 2023.
U. Rüb, Stratmann, K., Heinsen, H., Seidel, K., Bouzrou, M., and Korf, H. - W., Alzheimer's Disease: Characterization of the Brain Sites of the Initial Tau Cytoskeletal Pathology Will Improve the Success of Novel Immunological Anti-Tau Treatment Approaches., J Alzheimers Dis, vol. 57, no. 3, pp. 683-696, 2017.
S. Hatashita and Wakebe, D., Amyloid-β Deposition and Long-Term Progression in Mild Cognitive Impairment due to Alzheimer's Disease Defined with Amyloid PET Imaging., J Alzheimers Dis, vol. 57, no. 3, pp. 765-773, 2017.
I. Ferrer, Amyloid-β Pathology Is the Common Nominator Proteinopathy of the Primate Brain Aging., J Alzheimers Dis, vol. 100, no. s1, pp. S153-S164, 2024.
R. W. B. Love, Aniracetam: An Evidence-Based Model for Preventing the Accumulation of Amyloid-β Plaques in Alzheimer's Disease., J Alzheimers Dis, vol. 98, no. 4, pp. 1235-1241, 2024.
F. Piazza, Greenberg, S. M., Savoiardo, M., Gardinetti, M., Chiapparini, L., Raicher, I., Nitrini, R., Sakaguchi, H., Brioschi, M., Billo, G., Colombo, A., Lanzani, F., Piscosquito, G., Carriero, M. Rita, Giaccone, G., Tagliavini, F., Ferrarese, C., and DiFrancesco, J. C., Anti-amyloid β autoantibodies in cerebral amyloid angiopathy-related inflammation: implications for amyloid-modifying therapies., Ann Neurol, vol. 73, no. 4, pp. 449-58, 2013.
J. Bastrup, Hansen, K. H., Poulsen, T. B. G., Kastaniegaard, K., Asuni, A. A., Christensen, S., Belling, D., Helboe, L., Stensballe, A., and Volbracht, C., Anti-Aβ Antibody Aducanumab Regulates the Proteome of Senile Plaques and Closely Surrounding Tissue in a Transgenic Mouse Model of Alzheimer's Disease., J Alzheimers Dis, vol. 79, no. 1, pp. 249-265, 2021.
T. E. Golde, Schneider, L. S., and Koo, E. H., Anti-aβ therapeutics in Alzheimer's disease: the need for a paradigm shift., Neuron, vol. 69, no. 2, pp. 203-13, 2011.
J. J. Gomar, Conejero-Goldberg, C., Davies, P., and Goldberg, T. E., Anti-Correlated Cerebrospinal Fluid Biomarker Trajectories in Preclinical Alzheimer's Disease., J Alzheimers Dis, vol. 51, no. 4, pp. 1085-97, 2016.
J. C. Morris, Roe, C. M., Xiong, C., Fagan, A. M., Goate, A. M., Holtzman, D. M., and Mintun, M. A., APOE predicts amyloid-beta but not tau Alzheimer pathology in cognitively normal aging., Ann Neurol, vol. 67, no. 1, pp. 122-31, 2010.
J. C. Morris, Roe, C. M., Xiong, C., Fagan, A. M., Goate, A. M., Holtzman, D. M., and Mintun, M. A., APOE predicts amyloid-beta but not tau Alzheimer pathology in cognitively normal aging., Ann Neurol, vol. 67, no. 1, pp. 122-31, 2010.

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