Biblio
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“Is Computerized Working Memory Training Effective in Healthy Older Adults? Evidence from a Multi-Site, Randomized Controlled Trial.”, J Alzheimers Dis, vol. 65, no. 3, pp. 931-949, 2018.
, “Concordance Between Cerebrospinal Fluid Biomarkers with Alzheimer's Disease Pathology Between Three Independent Assay Platforms.”, J Alzheimers Dis, vol. 61, no. 1, pp. 169-183, 2018.
, “Connected Speech Features from Picture Description in Alzheimer's Disease: A Systematic Review.”, J Alzheimers Dis, vol. 65, no. 2, pp. 519-542, 2018.
, “Consent to Medical Procedures of Patients with Neurodegenerative Diseases: A Comparative Study of Legal Regulations in Selected European Countries and in the United States.”, J Alzheimers Dis, vol. 63, no. 1, pp. 53-67, 2018.
, “Constructing a Local Potential Participant Registry to Improve Alzheimer's Disease Clinical Research Recruitment.”, J Alzheimers Dis, vol. 63, no. 3, pp. 1055-1063, 2018.
, “Contribution of Bilingualism to Cognitive Reserve of an Italian Literature Professor at High Risk for Alzheimer's Disease.”, J Alzheimers Dis, vol. 66, no. 4, pp. 1389-1395, 2018.
, “The Conundrum of GSK3 Inhibitors: Is it the Dawn of a New Beginning?”, J Alzheimers Dis, vol. 64, no. s1, pp. S547-S554, 2018.
, “Correlates of Subjective Cognitive Decline in Lesbian, Gay, Bisexual, and Transgender Older Adults.”, J Alzheimers Dis, vol. 64, no. 1, pp. 91-102, 2018.
, “Crosstalk between Peripheral Small Vessel Properties and Anxious-like Profiles: Sex, Genotype, and Interaction Effects in Mice with Normal Aging and 3×Tg-AD mice at Advanced Stages of Disease.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1531-1538, 2018.
, “Cysteine-Rich Repeat Domains 2 and 4 are Amyloid-β Binding Domains of Neurotrophin Receptor p75NTR and Potential Targets to Block Amyloid-β Neurotoxicity.”, J Alzheimers Dis, vol. 63, no. 1, pp. 139-147, 2018.
, “Can Better Management of Periodontal Disease Delay the Onset and Progression of Alzheimer's Disease?”, J Alzheimers Dis, vol. 58, no. 2, pp. 337-348, 2017.
, “Can Musical or Painting Interventions Improve Chronic Pain, Mood, Quality of Life, and Cognition in Patients with Mild Alzheimer's Disease? Evidence from a Randomized Controlled Trial.”, J Alzheimers Dis, vol. 60, no. 2, pp. 663-677, 2017.
, “Capsaicin Attenuates Amyloid-β-Induced Synapse Loss and Cognitive Impairments in Mice.”, J Alzheimers Dis, vol. 59, no. 2, pp. 683-694, 2017.
, “Care Plan Improvement in Nursing Homes: An Integrative Review.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1621-1638, 2017.
, “CD44 Splice Variants as Potential Players in Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1137-1149, 2017.
, “Central Olfactory Dysfunction in Alzheimer's Disease and Mild Cognitive Impairment: A Functional MRI Study.”, J Alzheimers Dis, vol. 59, no. 1, pp. 359-368, 2017.
, “Central Review of Amyloid-Related Imaging Abnormalities in Two Phase III Clinical Trials of Bapineuzumab in Mild-To-Moderate Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 57, no. 2, pp. 557-573, 2017.
, “Cerebral Amyloid Angiopathy-Related Microbleeds and Cerebrospinal Fluid Biomarkers in Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 3, pp. 905-913, 2017.
, “Cerebral Artery Pulsatility is Associated with Cognitive Impairment and Predicts Dementia in Individuals with Subjective Memory Decline or Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 60, no. 2, pp. 625-632, 2017.
, “Cerebral Hemodynamics in Mild Cognitive Impairment: A Systematic Review.”, J Alzheimers Dis, vol. 59, no. 1, pp. 369-385, 2017.
, “Cerebral Oxidative Stress and Microvasculature Defects in TNF-α Expressing Transgenic and Porphyromonas gingivalis-Infected ApoE-/- Mice.”, J Alzheimers Dis, vol. 60, no. 2, pp. 359-369, 2017.
, “Cerebrospinal Fluid Amyloid-β 42, Total Tau and Phosphorylated Tau are Low in Patients with Normal Pressure Hydrocephalus: Analogies and Differences with Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 1, pp. 183-200, 2017.
, “The Cerebrospinal Fluid Aβ1-42/Aβ1-40 Ratio Improves Concordance with Amyloid-PET for Diagnosing Alzheimer's Disease in a Clinical Setting.”, J Alzheimers Dis, vol. 60, no. 2, pp. 561-576, 2017.
, “Cerebrospinal Fluid Aβ42/40 Corresponds Better than Aβ42 to Amyloid PET in Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 2, pp. 813-822, 2017.
, “Cerebrospinal Fluid Aβ42/Aβ40 as a Means to Limiting Tube- and Storage-Dependent Pre-Analytical Variability in Clinical Setting.”, J Alzheimers Dis, vol. 57, no. 2, pp. 437-445, 2017.
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