Biblio
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“Low Prevalence of Mixed Dementia in a Cohort of 2,000 Elderly Patients in a Memory Clinic Setting.”, J Alzheimers Dis, vol. 50, no. 3, pp. 797-806, 2016.
, “Low Serum Insulin-Like Growth Factor-I Predicts Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 2, pp. 641-9, 2016.
, “Lower Late-Life Body-Mass Index is Associated with Higher Cortical Amyloid Burden in Clinically Normal Elderly.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1097-105, 2016.
, “Lower Prevalence of Alzheimer's Disease among Tibetans: Association with Religious and Genetic Factors.”, J Alzheimers Dis, vol. 50, no. 3, pp. 659-67, 2016.
, “LRP/LR Antibody Mediated Rescuing of Amyloid-β-Induced Cytotoxicity is Dependent on PrPc in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 645-57, 2016.
, “Late Onset Alzheimer's Disease Risk Variants in Cognitive Decline: The PATH Through Life Study.”, J Alzheimers Dis, vol. 57, no. 2, pp. 423-436, 2017.
, “Left Frontal Hub Connectivity during Memory Performance Supports Reserve in Aging and Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1381-1392, 2017.
, “Left Ventricular Hypertrophy and Cognitive Decline in Old Age.”, J Alzheimers Dis, vol. 58, no. 1, pp. 275-283, 2017.
, “The level of 24-Hydroxycholesteryl Esters is an Early Marker of Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 2, pp. 825-833, 2017.
, “Levetiracetam Alters Oscillatory Connectivity in Alzheimer's Disease.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1065-1076, 2017.
, “The Link between Type 2 Diabetes and Neurodegeneration: Roles for Amyloid-β, Amylin, and Tau Proteins.”, J Alzheimers Dis, vol. 59, no. 2, pp. 421-432, 2017.
, “Lipid Peroxidation Markers in Coronary Artery Disease Patients with Possible Vascular Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 58, no. 3, pp. 885-896, 2017.
, “Lipidomic Profiles in Diabetes and Dementia.”, J Alzheimers Dis, vol. 59, no. 2, pp. 433-444, 2017.
, “A Lipidomics Approach to Assess the Association Between Plasma Sphingolipids and Verbal Memory Performance in Coronary Artery Disease Patients Undertaking Cardiac Rehabilitation: A C18:0 Signature for Cognitive Response to Exercise.”, J Alzheimers Dis, vol. 60, no. 3, pp. 829-841, 2017.
, “Living Alone with Alzheimer's Disease: Data from SveDem, the Swedish Dementia Registry.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1265-1272, 2017.
, “Localization and Processing of the Amyloid-β Protein Precursor in Mitochondria-Associated Membranes.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1549-1570, 2017.
, “Logopenic Aphasia due to a Strategic Stroke: New Evidence from a Single Case.”, J Alzheimers Dis, vol. 57, no. 3, pp. 717-721, 2017.
, “Longer-Term Investigation of the Value of 18F-FDG-PET and Magnetic Resonance Imaging for Predicting the Conversion of Mild Cognitive Impairment to Alzheimer's Disease: A Multicenter Study.”, J Alzheimers Dis, vol. 60, no. 3, pp. 877-887, 2017.
, “Longitudinal Characterization of [18F]-FDG and [18F]-AV45 Uptake in the Double Transgenic TASTPM Mouse Model.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1537-1548, 2017.
, “A Longitudinal Study on Resting State Functional Connectivity in Behavioral Variant Frontotemporal Dementia and Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 2, pp. 521-537, 2017.
, “Long-Term Cognitive Decline in Dementia with Lewy Bodies in a Large Multicenter, International Cohort.”, J Alzheimers Dis, vol. 57, no. 3, pp. 787-795, 2017.
, “Long-Term Maintenance of Executive-Related Oculomotor Improvements in Older Adults with Self-Reported Cognitive Complaints Following a 24-Week Multiple Modality Exercise Program.”, J Alzheimers Dis, vol. 58, no. 1, pp. 17-22, 2017.
, “Low Arousal Positive Emotional Stimuli Attenuate Aberrant Working Memory Processing in Persons with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1333-1349, 2017.
, “Low Cognitive Awareness, but Not Complaint, is a Good Marker of Preclinical Alzheimer's Disease.”, J Alzheimers Dis, vol. 59, no. 2, pp. 753-762, 2017.
, “Low Erythrocyte Levels of Proteasome and Acyl-Peptide Hydrolase (APEH) Activities in Alzheimer's Disease: A Sign of Defective Proteostasis?”, J Alzheimers Dis, vol. 60, no. 3, pp. 1097-1106, 2017.
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