Biblio
“Neuro-Immuno-Gene- and Genome-Editing-Therapy for Alzheimer's Disease: Are We There Yet?”, J Alzheimers Dis, vol. 65, no. 2, pp. 321-344, 2018.
, “Neuropathologic Correlates of Psychiatric Symptoms in Alzheimer's Disease.”, J Alzheimers Dis, vol. 66, no. 1, pp. 115-126, 2018.
, “Neuroprotective Effects of Amylin Analogues on Alzheimer's Disease Pathogenesis and Cognition.”, J Alzheimers Dis, vol. 66, no. 1, pp. 11-23, 2018.
, “Neuroprotective Effects of Ferruginol, Jatrophone, and Junicedric Acid Against Amyloid-β Injury in Hippocampal Neurons.”, J Alzheimers Dis, vol. 63, no. 2, pp. 705-723, 2018.
, “Neuroprotective Effects of Ginsenoside Rf on Amyloid-β-Induced Neurotoxicity in vitro and in vivo.”, J Alzheimers Dis, vol. 64, no. 1, pp. 309-322, 2018.
, “Neuropsychiatric and Cognitive Subtypes among Community-Dwelling Older Persons and the Association with DSM-5 Mild Neurocognitive Disorder: Latent Class Analysis.”, J Alzheimers Dis, vol. 62, no. 2, pp. 675-686, 2018.
, “Neuropsychiatric Symptoms and the Diagnostic Stability of Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1841-1855, 2018.
, “Neuropsychiatric Symptoms Complicating the Diagnosis of Alzheimer's Disease: A Case Report.”, J Alzheimers Dis, vol. 66, no. 4, pp. 1363-1369, 2018.
, “Neuropsychological Predictors of Long-Term (10 Years) Mild Cognitive Impairment Stability.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1703-1711, 2018.
, “Neurotoxic Effects of Aβ6-42 Peptides Mimicking Putative Products Formed by the Angiotensin Converting Enzyme.”, J Alzheimers Dis, vol. 66, no. 1, pp. 263-270, 2018.
, “New Beginnings in Alzheimer's Disease: The Most Prevalent Tauopathy.”, J Alzheimers Dis, vol. 64, no. s1, pp. S529-S534, 2018.
, “New Insights into the Spontaneous Human Alzheimer's Disease-Like Model Octodon degus: Unraveling Amyloid-β Peptide Aggregation and Age-Related Amyloid Pathology.”, J Alzheimers Dis, vol. 66, no. 3, pp. 1145-1163, 2018.
, “A New Perspective in Utilizing MMP-9 as a Therapeutic Target for Alzheimer's Disease and Type 2 Diabetes Mellitus.”, J Alzheimers Dis, vol. 64, no. 1, pp. 1-16, 2018.
, “No Effects of Black Tea on Cognitive Decline Among Older US Men: A Prospective Cohort Study.”, J Alzheimers Dis, vol. 65, no. 1, pp. 99-105, 2018.
, “NO-Dependent Akt Inactivation by S-Nitrosylation as a Possible Mechanism of STZ-Induced Neuronal Insulin Resistance.”, J Alzheimers Dis, vol. 65, no. 4, pp. 1427-1443, 2018.
, “A Nomogram for Predicting Amyloid PET Positivity in Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 66, no. 2, pp. 681-691, 2018.
, “Non-Ashkenazi Jewish Origin is Associated with Early Onset Alzheimer's Disease.”, J Alzheimers Dis, vol. 65, no. 3, pp. 877-884, 2018.
, “Non-Genetic Risk Factors for Degenerative and Vascular Young Onset Dementia: Results from the INSPIRED and KGOW Studies.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1747-1758, 2018.
, “Non-Phosphorylated Tau in Cerebrospinal Fluid is a Marker of Alzheimer's Disease Continuum in Young Urbanites Exposed to Air Pollution.”, J Alzheimers Dis, vol. 66, no. 4, pp. 1437-1451, 2018.
, “A Novel Antibody Targeting Tau Phosphorylated at Serine 235 Detects Neurofibrillary Tangles.”, J Alzheimers Dis, vol. 61, no. 3, pp. 899-905, 2018.
, “The Novel DA-CH3 Dual Incretin Restores Endoplasmic Reticulum Stress and Autophagy Impairments to Attenuate Alzheimer-Like Pathology and Cognitive Decrements in the APPSWE/PS1ΔE9 Mouse Model.”, J Alzheimers Dis, vol. 66, no. 1, pp. 195-218, 2018.
, “Novel GRN Mutations in Alzheimer's Disease and Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1683-1689, 2018.
, “Novel Key Players in the Development of Tau Neuropathology: Focus on the 5-Lipoxygenase.”, J Alzheimers Dis, vol. 64, no. s1, pp. S481-S489, 2018.
, “Novel Lipidized Analog of Prolactin-Releasing Peptide Improves Memory Impairment and Attenuates Hyperphosphorylation of Tau Protein in a Mouse Model of Tauopathy.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1725-1736, 2018.
, “A Novel Therapeutic Approach to Treat Alzheimer's Disease by Neurotrophic Support During the Period of Synaptic Compensation.”, J Alzheimers Dis, vol. 62, no. 3, pp. 1211-1218, 2018.
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