Biblio
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“NLRP3 Inflammasome: A Starring Role in Amyloid-β- and Tau-Driven Pathological Events in Alzheimer's Disease.”, J Alzheimers Dis, vol. 83, no. 3, pp. 939-961, 2021.
, “A Novel Approach to the Treatment and Prevention of Alzheimer's Disease Based on the Pathology and Microbiology.”, J Alzheimers Dis, vol. 84, no. 1, pp. 61-67, 2021.
, “Plasma Biomarkers of Alzheimer's Disease in African Americans.”, J Alzheimers Dis, vol. 79, no. 1, pp. 323-334, 2021.
, “Plasma Biomarkers of Alzheimer's Disease in African Americans.”, J Alzheimers Dis, vol. 79, no. 1, pp. 323-334, 2021.
, “Porphyromonas gingivalis Outer Membrane Vesicles as the Major Driver of and Explanation for Neuropathogenesis, the Cholinergic Hypothesis, Iron Dyshomeostasis, and Salivary Lactoferrin in Alzheimer's Disease.”, J Alzheimers Dis, vol. 82, no. 4, pp. 1417-1450, 2021.
, “Quality of Life in Advanced Dementia with Late Onset, Young Onset, and Very Young Onset.”, J Alzheimers Dis, vol. 80, no. 1, pp. 283-297, 2021.
, “White Matter Connectivity in Incident Mild Cognitive Impairment: A Diffusion Spectrum Imaging Study of World Trade Center Responders at Midlife.”, J Alzheimers Dis, vol. 80, no. 3, pp. 1209-1219, 2021.
, “COVID-19: Review of a 21st Century Pandemic from Etiology to Neuro-psychiatric Implications.”, J Alzheimers Dis, vol. 77, no. 2, pp. 459-504, 2020.
, “Death Rate Due to COVID-19 in Alzheimer's Disease and Frontotemporal Dementia.”, J Alzheimers Dis, vol. 78, no. 2, pp. 537-541, 2020.
, “Neurobiology of COVID-19.”, J Alzheimers Dis, vol. 76, no. 1, pp. 3-19, 2020.
, “Potential Novel Role of COVID-19 in Alzheimer's Disease and Preventative Mitigation Strategies.”, J Alzheimers Dis, vol. 76, no. 1, pp. 21-25, 2020.
, “Telemedicine for Delivery of Care in Frontotemporal Lobar Degeneration During COVID-19 Pandemic: Results from Southern Italy.”, J Alzheimers Dis, vol. 76, no. 2, pp. 481-489, 2020.
, “Alzheimer's Disease Progression: Factors Influencing Cognitive Decline.”, J Alzheimers Dis, vol. 61, no. 2, pp. 785-791, 2018.
, “Alzheimer's Disease: Recent Concepts on the Relation of Mitochondrial Disturbances, Excitotoxicity, Neuroinflammation, and Kynurenines.”, J Alzheimers Dis, vol. 62, no. 2, pp. 523-547, 2018.
, “Alzheimer's Disease: Recent Concepts on the Relation of Mitochondrial Disturbances, Excitotoxicity, Neuroinflammation, and Kynurenines.”, J Alzheimers Dis, vol. 62, no. 2, pp. 523-547, 2018.
, “Association between α-Klotho and Deep White Matter Lesions in the Brain: A Pilot Case Control Study Using Brain MRI.”, J Alzheimers Dis, vol. 61, no. 1, pp. 145-155, 2018.
, “Association of Cerebrospinal Fluid (CSF) Insulin with Cognitive Performance and CSF Biomarkers of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 309-320, 2018.
, “Association of the New Variant Tyr424Asp at TBK1 Gene with Amyotrophic Lateral Sclerosis and Cognitive Decline.”, J Alzheimers Dis, vol. 61, no. 1, pp. 41-46, 2018.
, “Circulating Progenitor Cells Correlate with Memory, Posterior Cortical Thickness, and Hippocampal Perfusion.”, J Alzheimers Dis, vol. 61, no. 1, pp. 91-101, 2018.
, “Cognitive Variability Predicts Incident Alzheimer's Disease and Mild Cognitive Impairment Comparable to a Cerebrospinal Fluid Biomarker.”, J Alzheimers Dis, vol. 61, no. 1, pp. 79-89, 2018.
, “Designing an Internet-Based Multidomain Intervention for the Prevention of Cardiovascular Disease and Cognitive Impairment in Older Adults: The HATICE Trial.”, J Alzheimers Dis, vol. 62, no. 2, pp. 649-663, 2018.
, “Detection of Dementia Cases in Two Swedish Health Registers: A Validation Study.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1301-1310, 2018.
, “Early and Persistent O-GlcNAc Protein Modification in the Streptozotocin Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 237-249, 2018.
, “Early Stage Alterations in CA1 Extracellular Region Proteins Indicate Dysregulation of IL6 and Iron Homeostasis in the 5XFAD Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1399-1410, 2018.
, “Early Stage Alterations in CA1 Extracellular Region Proteins Indicate Dysregulation of IL6 and Iron Homeostasis in the 5XFAD Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1399-1410, 2018.
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