Biblio
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“Reelin in Alzheimer's Disease, Increased Levels but Impaired Signaling: When More is Less.”, J Alzheimers Dis, vol. 52, no. 2, pp. 403-16, 2016.
, “Reduction of Blood Amyloid-β Oligomers in Alzheimer's Disease Transgenic Mice by c-Abl Kinase Inhibition.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1193-1205, 2016.
, “Reduction of Amyloid-β Plasma Levels by Hemodialysis: An Anti-Amyloid Treatment Strategy?”, J Alzheimers Dis, vol. 50, no. 3, pp. 791-6, 2016.
, “Reduced Cardiovascular Functions in Patients with Alzheimer's Disease.”, J Alzheimers Dis, vol. 58, no. 3, pp. 919-925, 2017.
, “Recovery from Proactive Semantic Interference in Mild Cognitive Impairment and Normal Aging: Relationship to Atrophy in Brain Regions Vulnerable to Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1119-1126, 2017.
, “Recovery from Proactive Semantic Interference in Mild Cognitive Impairment and Normal Aging: Relationship to Atrophy in Brain Regions Vulnerable to Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1119-1126, 2017.
, “Recovery from Proactive Semantic Interference in Mild Cognitive Impairment and Normal Aging: Relationship to Atrophy in Brain Regions Vulnerable to Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1119-1126, 2017.
, “Recovery from Proactive Semantic Interference in Mild Cognitive Impairment and Normal Aging: Relationship to Atrophy in Brain Regions Vulnerable to Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1119-1126, 2017.
, “Recovery from Proactive Semantic Interference and MRI Volume: A Replication and Extension Study.”, J Alzheimers Dis, vol. 59, no. 1, pp. 131-139, 2017.
, “Re-Addressing Dementia by Network Medicine and Mechanism-Based Molecular Endotypes.”, J Alzheimers Dis, vol. 96, no. 1, pp. 47-56, 2023.
, “Rbfox3/NeuN Regulates Alternative Splicing of Tau Exon 10.”, J Alzheimers Dis, vol. 66, no. 4, pp. 1695-1704, 2018.
, “Rbfox3/NeuN Regulates Alternative Splicing of Tau Exon 10.”, J Alzheimers Dis, vol. 66, no. 4, pp. 1695-1704, 2018.
, “The Rationale and Design of the Reducing Pathology in Alzheimer's Disease through Angiotensin TaRgeting (RADAR) Trial.”, J Alzheimers Dis, vol. 61, no. 2, pp. 803-814, 2018.
, “The Rationale and Design of the Reducing Pathology in Alzheimer's Disease through Angiotensin TaRgeting (RADAR) Trial.”, J Alzheimers Dis, vol. 61, no. 2, pp. 803-814, 2018.
, “Rationale and Design of the Mechanistic Potential of Antihypertensives in Preclinical Alzheimer's (HEART) Trial.”, J Alzheimers Dis, vol. 61, no. 2, pp. 815-824, 2018.
, “Rare Variants in PLD3 Increase Risk for Alzheimer's Disease in Han Chinese.”, J Alzheimers Dis, vol. 64, no. 1, pp. 55-59, 2018.
, “Rare Genetic Variant in SORL1 May Increase Penetrance of Alzheimer's Disease in a Family with Several Generations of APOE-ɛ4 Homozygosity.”, J Alzheimers Dis, vol. 56, no. 1, pp. 63-74, 2017.
, “Rapidly Progressive Frontotemporal Dementia Associated with MAPT Mutation G389R.”, J Alzheimers Dis, vol. 55, no. 2, pp. 777-785, 2017.
, “Rapidly Progressive Alzheimer's Disease: Contributions to Clinical-Pathological Definition and Diagnosis.”, J Alzheimers Dis, 2018.
, “A Randomized, Double-Blind, Placebo-Controlled, Phase II Study Assessing Safety, Tolerability, and Efficacy of Bryostatin in the Treatment of Moderately Severe to Severe Alzheimer's Disease.”, J Alzheimers Dis, vol. 67, no. 2, pp. 555-570, 2019.
, “Radiological-Pathological Correlation in Alzheimer's Disease: Systematic Review of Antemortem Magnetic Resonance Imaging Findings.”, J Alzheimers Dis, vol. 57, no. 2, pp. 575-601, 2017.
, “Racial/Ethnic Disparities in Misidentification of Dementia in Medicare Claims: Results from the Washington Heights-Inwood Columbia Aging Project.”, J Alzheimers Dis, vol. 96, no. 1, pp. 359-368, 2023.
, “Race/Ethnic Disparities in Mild Cognitive Impairment and Dementia: The Northern Manhattan Study.”, J Alzheimers Dis, vol. 80, no. 3, pp. 1129-1138, 2021.
, “Rab10 Phosphorylation is a Prominent Pathological Feature in Alzheimer's Disease.”, J Alzheimers Dis, vol. 63, no. 1, pp. 157-165, 2018.
, “Quantitative phosphoproteomic analyses of the inferior parietal lobule from three different pathological stages of Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 45-62, 2016.
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