Biblio
“Social Robot Interventions for People with Dementia: A Systematic Review on Effects and Quality of Reporting.”, J Alzheimers Dis, vol. 79, no. 2, pp. 773-792, 2021.
, “Social Representation of Dementia: An Analysis of 5,792 Consecutive Cases Evaluated in a Memory Clinic.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1099-1108, 2017.
, “Social Isolation and Cognitive Function in Later Life: A Systematic Review and Meta-Analysis.”, J Alzheimers Dis, vol. 70, no. s1, pp. S119-S144, 2019.
, “Social Dysfunction in Older Age and Relationships with Cognition, Depression, and Apathy: The GreatAGE Study.”, J Alzheimers Dis, vol. 65, no. 3, pp. 989-1000, 2018.
, “Social Cognition in the Frontal Variant of Alzheimer's Disease: A Case Study.”, J Alzheimers Dis, vol. 55, no. 2, pp. 459-463, 2017.
, “Social Cognition Deficits: The Key to Discriminate Behavioral Variant Frontotemporal Dementia from Alzheimer's Disease Regardless of Amnesia?”, J Alzheimers Dis, vol. 49, no. 4, pp. 1065-74, 2016.
, “Social Activity and Cognitive Decline in Older Residents of Long-Term Care Facilities: A Cohort Study.”, J Alzheimers Dis, vol. 98, no. 2, pp. 433-443, 2024.
, “So Close Yet So Far: Executive Contribution to Memory Processing in Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1005-1014, 2016.
, “Smoking, APOE Genotype, and Cognitive Decline: The Rotterdam Study.”, J Alzheimers Dis, vol. 57, no. 4, pp. 1191-1195, 2017.
, “Small Vessel Cerebrovascular Pathology Identified by Magnetic Resonance Imaging Is Prevalent in Alzheimer's Disease and Mild Cognitive Impairment: A Potential Target for Intervention.”, J Alzheimers Dis, vol. 65, no. 1, pp. 293-302, 2018.
, “Small RNA Sequencing in the Tg4-42 Mouse Model Suggests the Involvement of snoRNAs in the Etiology of Alzheimer's Disease.”, J Alzheimers Dis, vol. 87, no. 4, pp. 1671-1681, 2022.
, “A Small Molecule Inhibitor of Plasminogen Activator Inhibitor-1 Reduces Brain Amyloid-β Load and Improves Memory in an Animal Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 64, no. 2, pp. 447-457, 2018.
, “Small Molecule Amyloid-β Protein Precursor Processing Modulators Lower Amyloid-β Peptide Levels via cKit Signaling.”, J Alzheimers Dis, vol. 67, no. 3, pp. 1089-1106, 2019.
, “Sleep-Wake Profile in Dementia with Lewy Bodies, Alzheimer's Disease, and Normal Aging.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1529-1536, 2017.
, “Sleep-Wake Cycle in Alzheimer's Disease Is Associated with Tau Pathology and Orexin Dysregulation.”, J Alzheimers Dis, vol. 74, no. 2, pp. 501-508, 2020.
, “Sleep EEG Detects Epileptiform Activity in Alzheimer's Disease with High Sensitivity.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1175-1183, 2017.
, “Is Sleep Disruption a Risk Factor for Alzheimer's Disease?”, J Alzheimers Dis, vol. 58, no. 4, pp. 993-1002, 2017.
, “Sleep Deprivation Induced Plasma Amyloid-β Transport Disturbance in Healthy Young Adults.”, J Alzheimers Dis, vol. 57, no. 3, pp. 899-906, 2017.
, “Sleep Deprivation, a Link Between Post-Traumatic Stress Disorder and Alzheimer's Disease.”, J Alzheimers Dis, vol. 79, no. 4, pp. 1443-1449, 2021.
, “Sleep Apnea, Cognitive Profile, and Vascular Changes: An Intriguing Relationship.”, J Alzheimers Dis, vol. 60, no. 3, pp. 1195-1203, 2017.
, “Sleep and EEG Power Spectral Analysis in Three Transgenic Mouse Models of Alzheimer's Disease: APP/PS1, 3xTgAD, and Tg2576.”, J Alzheimers Dis, vol. 64, no. 4, pp. 1325-1336, 2018.
, “Skin Autofluorescence Examination as a Diagnostic Tool for Mild Cognitive Impairment in Healthy People.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1481-1487, 2017.
, “A Six-Month Cognitive-Motor and Aerobic Exercise Program Improves Executive Function in Persons with an Objective Cognitive Impairment: A Pilot Investigation Using the Antisaccade Task.”, J Alzheimers Dis, vol. 54, no. 3, pp. 923-931, 2016.
, “Situation Model for Situation-Aware Assistance of Dementia Patients in Outdoor Mobility.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1461-1476, 2017.
, “Site-Specific Cerebrospinal Fluid Tau Hyperphosphorylation in Response to Alzheimer's Disease Brain Pathology: Not All Tau Phospho-Sites are Hyperphosphorylated.”, J Alzheimers Dis, vol. 85, no. 1, pp. 415-429, 2021.
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