Biblio
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“Protein Phosphorylation is a Key Mechanism in Alzheimer's Disease.”, J Alzheimers Dis, vol. 58, no. 4, pp. 953-978, 2017.
, “Protein Kinase Activity Decreases with Higher Braak Stages of Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 49, no. 4, pp. 927-43, 2016.
, “Propagation of tau pathology in a model of early Alzheimer's disease.”, Neuron, vol. 73, no. 4, pp. 685-97, 2012.
, “Promising Role of Neuromodulation in Predicting the Progression of Mild Cognitive Impairment to Dementia.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1375-88, 2016.
, “Promising Role of Neuromodulation in Predicting the Progression of Mild Cognitive Impairment to Dementia.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1375-88, 2016.
, “Prominent Non-Memory Deficits in Alzheimer's Disease Are Associated with Faster Disease Progression.”, J Alzheimers Dis, 2018.
, “Prolonged Visual Facial Grasp in Frontotemporal Dementia.”, J Alzheimers Dis, vol. 53, no. 1, pp. 327-35, 2016.
, “Progression of Alzheimer's Disease-Related Pathology and Cell Counts in a Patient with Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1451-1462, 2018.
, “The Progression of Alzheimer's Disease: Are Fast Decliners Really Fast? A Four-Year Follow-Up.”, J Alzheimers Dis, vol. 57, no. 3, pp. 775-786, 2017.
, “The Progression of Alzheimer's Disease: Are Fast Decliners Really Fast? A Four-Year Follow-Up.”, J Alzheimers Dis, vol. 57, no. 3, pp. 775-786, 2017.
, “Progression from Subjective Cognitive Decline to Mild Cognitive Impairment or Dementia: The Role of Baseline Cognitive Performance.”, J Alzheimers Dis, vol. 86, no. 4, pp. 1763-1774, 2022.
, “Progression from Prodromal Alzheimer's Disease to Mild Alzheimer's Disease Dementia in the Verubecestat APECS Study: Adjudicating Diagnostic Transitions.”, J Alzheimers Dis, vol. 92, no. 1, pp. 341-348, 2023.
, “Progress of Brain Amyloid Deposition in Familial Alzheimer's Disease with Taiwan D678H APP Mutation.”, J Alzheimers Dis, vol. 66, no. 2, pp. 775-787, 2018.
, “Progranulin Protein Levels in Cerebrospinal Fluid in Primary Neurodegenerative Dementias.”, J Alzheimers Dis, vol. 50, no. 2, pp. 539-46, 2016.
, “Progranulin Protein Levels in Cerebrospinal Fluid in Primary Neurodegenerative Dementias.”, J Alzheimers Dis, vol. 50, no. 2, pp. 539-46, 2016.
, “Progranulin Protein Levels in Cerebrospinal Fluid in Primary Neurodegenerative Dementias.”, J Alzheimers Dis, vol. 50, no. 2, pp. 539-46, 2016.
, “Prognostic Significance of Mild Cognitive Impairment Subtypes for Dementia and Mortality: Data from the NEDICES Cohort.”, J Alzheimers Dis, vol. 50, no. 3, pp. 719-31, 2016.
, “Profiling of Specific Gene Expression Pathways in Peripheral Cells from Prodromal Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1289-1294, 2018.
, “Prodromal Dementia with Lewy Bodies and Prodromal Alzheimer's Disease: A Comparison of the Cognitive and Clinical Profiles.”, J Alzheimers Dis, vol. 58, no. 2, pp. 463-470, 2017.
, “Prodromal Alzheimer's Disease Demonstrates Increased Errors at a Simple and Automated Anti-Saccade Task.”, J Alzheimers Dis, vol. 65, no. 4, pp. 1209-1223, 2018.
, “Prodromal Alzheimer's Disease Demonstrates Increased Errors at a Simple and Automated Anti-Saccade Task.”, J Alzheimers Dis, vol. 65, no. 4, pp. 1209-1223, 2018.
, “proBDNF Accelerates Brain Amyloid-β Deposition and Learning and Memory Impairment in APPswePS1dE9 Transgenic Mice.”, J Alzheimers Dis, vol. 59, no. 3, pp. 941-949, 2017.
, “PRNP P39L Variant is a Rare Cause of Frontotemporal Dementia in Italian Population.”, J Alzheimers Dis, vol. 50, no. 2, pp. 353-7, 2016.
, “PRNP P39L Variant is a Rare Cause of Frontotemporal Dementia in Italian Population.”, J Alzheimers Dis, vol. 50, no. 2, pp. 353-7, 2016.
, “Prions and Neurodegenerative Diseases: A Focus on Alzheimer's Disease.”, J Alzheimers Dis, vol. 85, no. 2, pp. 503-518, 2022.
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