Biblio
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“Increased Electron Paramagnetic Resonance Signal Correlates with Mitochondrial Dysfunction and Oxidative Stress in an Alzheimer's disease Mouse Brain.”, J Alzheimers Dis, vol. 51, no. 2, pp. 571-80, 2016.
, “Increased Foxo3a Nuclear Translocation and Activity is an Early Neuronal Response to βγ-Secretase-Mediated Processing of the Amyloid-β Protein Precursor: Utility of an AβPP-GAL4 Reporter Assay.”, J Alzheimers Dis, vol. 61, no. 2, pp. 673-688, 2018.
, “Increased Vulnerability of the Hippocampus in Transgenic Mice Overexpressing APP and Triple Repeat Tau.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1201-1219, 2018.
, “Inhibition of Cholesterol Biosynthesis Reduces γ-Secretase Activity and Amyloid-β Generation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1057-68, 2016.
, “Inhibition of Histone Deacetylase 3 Restores Amyloid-β Oligomer-Induced Plasticity Deficit in Hippocampal CA1 Pyramidal Neurons.”, J Alzheimers Dis, vol. 51, no. 3, pp. 783-91, 2016.
, “Integrated systems approach identifies genetic nodes and networks in late-onset Alzheimer's disease.”, Cell, vol. 153, no. 3, pp. 707-20, 2013.
, “Intranasal TAT-haFGF Improves Cognition and Amyloid-β Pathology in an AβPP/PS1 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 985-90, 2016.
, “Iron Regulates Apolipoprotein E Expression and Secretion in Neurons and Astrocytes.”, J Alzheimers Dis, vol. 51, no. 2, pp. 471-87, 2016.
, “JNK: A Putative Link Between Insulin Signaling and VGLUT1 in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 4, pp. 963-7, 2016.
, “Lentivirus-Mediated HDAC3 Inhibition Attenuates Oxidative Stress in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1411-1424, 2018.
, “The Locus Coeruleus in Aging and Alzheimer's Disease: A Postmortem and Brain Imaging Review.”, J Alzheimers Dis, vol. 83, no. 1, pp. 5-22, 2021.
, “A Long Journey into Aging, Brain Aging, and Alzheimer's Disease Following the Oxidative Stress Tracks.”, J Alzheimers Dis, vol. 62, no. 3, pp. 1319-1335, 2018.
, “LRP/LR Antibody Mediated Rescuing of Amyloid-β-Induced Cytotoxicity is Dependent on PrPc in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 645-57, 2016.
, “Matrix Metalloproteinase in Blood-Brain Barrier Breakdown in Dementia.”, J Alzheimers Dis, vol. 49, no. 4, pp. 893-903, 2016.
, “Memory impairment in transgenic Alzheimer mice requires cellular prion protein.”, J Neurosci, vol. 30, no. 18, pp. 6367-74, 2010.
, “A Mercaptoacetamide-Based Class II Histone Deacetylase Inhibitor Increases Dendritic Spine Density via RasGRF1/ERK Pathway.”, J Alzheimers Dis, vol. 51, no. 2, pp. 591-604, 2016.
, “Mesenchymal Stem Cell-Derived Extracellular Vesicles Suppresses iNOS Expression and Ameliorates Neural Impairment in Alzheimer's Disease Mice.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1005-1013, 2018.
, “Metallothioneins in Prion- and Amyloid-Related Diseases.”, J Alzheimers Dis, vol. 51, no. 3, pp. 637-56, 2016.
, “Metformin Facilitates Amyloid-β Generation by β- and γ-Secretases via Autophagy Activation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1197-208, 2016.
, “A Mitochondrial Role of SV2a Protein in Aging and Alzheimer's Disease: Studies with Levetiracetam.”, J Alzheimers Dis, vol. 50, no. 1, pp. 201-15, 2016.
, “Modulating the Balance of Synaptic and Extrasynaptic NMDA Receptors Shows Positive Effects against Amyloid-β-Induced Neurotoxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 885-897, 2017.
, “Modulation of miR-19 in Aluminum-Induced Neural Cell Apoptosis.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1149-62, 2016.
, “Molecular Signaling Mechanisms of Natural and Synthetic Retinoids for Inhibition of Pathogenesis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 335-52, 2016.
, “Multifunctional Effect of Human Serum Albumin Reduces Alzheimer's Disease Related Pathologies in the 3xTg Mouse Model.”, J Alzheimers Dis, vol. 50, no. 1, pp. 175-88, 2016.
, “Naphthoquinone-Tryptophan Hybrid Inhibits Aggregation of the Tau-Derived Peptide PHF6 and Reduces Neurotoxicity.”, J Alzheimers Dis, vol. 51, no. 1, pp. 165-78, 2016.
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