Biblio
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“Recent Advances in Drug Delivery Systems Targeting Insulin Signalling for the Treatment of Alzheimer's Disease.”, J Alzheimers Dis, vol. 98, no. 4, pp. 1169-1179, 2024.
, “Research Participants' Perspectives on Precision Diagnostics for Alzheimer's Disease.”, J Alzheimers Dis, vol. 97, no. 3, pp. 1261-1274, 2024.
, “Safety Profile of Pimavanserin Therapy in Elderly Patients with Neurodegenerative Disease-Related Neuropsychiatric Symptoms: A Phase 3B Study.”, J Alzheimers Dis, vol. 98, no. 1, pp. 265-274, 2024.
, “A Scoping Review of Alzheimers Disease Hypotheses: An Array of Uni- and Multi-Factorial Theories.”, J Alzheimers Dis, vol. 99, no. 3, pp. 843-856, 2024.
, “Senescence Targeting Methods Impact Alzheimer's Disease Features in 3xTg Mice.”, J Alzheimers Dis, vol. 97, no. 4, pp. 1751-1763, 2024.
, “Serial Cerebrospinal Fluid Sampling Reveals Trajectories of Potential Synaptic Biomarkers in Early Stages of Alzheimer's Disease.”, J Alzheimers Dis, vol. 100, no. s1, pp. S103-S114, 2024.
, “Serial Cerebrospinal Fluid Sampling Reveals Trajectories of Potential Synaptic Biomarkers in Early Stages of Alzheimer's Disease.”, J Alzheimers Dis, vol. 100, no. s1, pp. S103-S114, 2024.
, “Social support may buffer the effect of ε4 allele on cognition in older adults.”, J Alzheimers Dis, vol. 102, no. 4, pp. 967-974, 2024.
, “Social support may buffer the effect of ε4 allele on cognition in older adults.”, J Alzheimers Dis, vol. 102, no. 4, pp. 967-974, 2024.
, “Symptomatic Clusters Related to Amyloid Positivity in Cognitively Unimpaired Individuals.”, J Alzheimers Dis, vol. 100, no. 1, pp. 193-205, 2024.
, “Symptomatic Clusters Related to Amyloid Positivity in Cognitively Unimpaired Individuals.”, J Alzheimers Dis, vol. 100, no. 1, pp. 193-205, 2024.
, “Symptomatic Clusters Related to Amyloid Positivity in Cognitively Unimpaired Individuals.”, J Alzheimers Dis, vol. 100, no. 1, pp. 193-205, 2024.
, “A systematic review of lifestyle-based interventions for managing Alzheimer's disease: Insights from randomized controlled trials.”, J Alzheimers Dis, vol. 102, no. 4, pp. 943-966, 2024.
, “A systematic review of lifestyle-based interventions for managing Alzheimer's disease: Insights from randomized controlled trials.”, J Alzheimers Dis, vol. 102, no. 4, pp. 943-966, 2024.
, “Alzheimer's Disease and Neurosyphilis: Meaningful Commonalities and Differences of Clinical Phenotype and Pathophysiological Biomarkers.”, J Alzheimers Dis, vol. 94, no. 2, pp. 611-625, 2023.
, “Alzheimer's Disease Pathology Outside of the Cerebrum Is Related to a Higher Odds of Dementia.”, J Alzheimers Dis, vol. 96, no. 2, pp. 563-578, 2023.
, “Alzheimer's Disease Pathology Outside of the Cerebrum Is Related to a Higher Odds of Dementia.”, J Alzheimers Dis, vol. 96, no. 2, pp. 563-578, 2023.
, “The Anti-Amyloid Monoclonal Antibody Lecanemab: 16 Cautionary Notes.”, J Alzheimers Dis, vol. 94, no. 2, pp. 497-507, 2023.
, “Associations Between Ambient Air Pollution and Cognitive Abilities from Midlife to Early Old Age: Modification by APOE Genotype.”, J Alzheimers Dis, vol. 93, no. 1, pp. 193-209, 2023.
, “Associations Between Ambient Air Pollution and Cognitive Abilities from Midlife to Early Old Age: Modification by APOE Genotype.”, J Alzheimers Dis, vol. 93, no. 1, pp. 193-209, 2023.
, “Associations of the Harvard Automated Phone Task and Alzheimer's Disease Pathology in Cognitively Normal Older Adults: Preliminary Findings.”, J Alzheimers Dis, vol. 94, no. 1, pp. 217-226, 2023.
, “Cardiorespiratory Fitness Attenuates the Deleterious Effects of Sleep Apnea on Cerebral Structure and Perfusion in the Wisconsin Sleep Cohort Study.”, J Alzheimers Dis, vol. 95, no. 2, pp. 427-435, 2023.
, “Cardiorespiratory Fitness Attenuates the Deleterious Effects of Sleep Apnea on Cerebral Structure and Perfusion in the Wisconsin Sleep Cohort Study.”, J Alzheimers Dis, vol. 95, no. 2, pp. 427-435, 2023.
, “Cerebral Gray and White Matter Monogalactosyl Diglyceride Levels Rise with the Progression of Alzheimer's Disease.”, J Alzheimers Dis, vol. 95, no. 4, pp. 1623-1634, 2023.
, “Cerebral Gray and White Matter Monogalactosyl Diglyceride Levels Rise with the Progression of Alzheimer's Disease.”, J Alzheimers Dis, vol. 95, no. 4, pp. 1623-1634, 2023.
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