Biblio
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“Efficacy and Safety of Sustained Release Donepezil High Dose versus Immediate Release Donepezil Standard Dose in Japanese Patients with Severe Alzheimer's Disease: A Randomized, Double-Blind Trial.”, J Alzheimers Dis, vol. 52, no. 1, pp. 345-57, 2016.
, “Empirically Defining Trajectories of Late-Life Cognitive and Functional Decline.”, J Alzheimers Dis, vol. 50, no. 1, pp. 271-82, 2016.
, “Enalapril Alone or Co-Administered with Losartan Rescues Cerebrovascular Dysfunction, but not Mnemonic Deficits or Amyloidosis in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1183-95, 2016.
, “Enalapril Alone or Co-Administered with Losartan Rescues Cerebrovascular Dysfunction, but not Mnemonic Deficits or Amyloidosis in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1183-95, 2016.
, “The Evolution of Caregiver Burden in Frontotemporal Dementia with and without Amyotrophic Lateral Sclerosis.”, J Alzheimers Dis, vol. 49, no. 3, pp. 875-85, 2016.
, “The Evolution of Caregiver Burden in Frontotemporal Dementia with and without Amyotrophic Lateral Sclerosis.”, J Alzheimers Dis, vol. 49, no. 3, pp. 875-85, 2016.
, “The Evolution of Caregiver Burden in Frontotemporal Dementia with and without Amyotrophic Lateral Sclerosis.”, J Alzheimers Dis, vol. 49, no. 3, pp. 875-85, 2016.
, “Examining the Relationship Between Autobiographical Memory Impairment and Carer Burden in Dementia Syndromes.”, J Alzheimers Dis, vol. 51, no. 1, pp. 237-48, 2016.
, “Exercise Plus Cognitive Performance Over and Above Exercise Alone in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 50, no. 1, pp. 19-25, 2016.
, “Exercise Plus Cognitive Performance Over and Above Exercise Alone in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 50, no. 1, pp. 19-25, 2016.
, “Formal Psychiatric Disorders are not Overrepresented in Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1249-56, 2016.
, “Functional Connectivity of Ventral and Dorsal Visual Streams in Posterior Cortical Atrophy.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1119-30, 2016.
, “Functional Connectivity of Ventral and Dorsal Visual Streams in Posterior Cortical Atrophy.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1119-30, 2016.
, “Future Dementia Severity is Almost Entirely Explained by the Latent Variable δ's Intercept and Slope.”, J Alzheimers Dis, vol. 49, no. 2, pp. 521-9, 2016.
, “Future Dementia Severity is Almost Entirely Explained by the Latent Variable δ's Intercept and Slope.”, J Alzheimers Dis, vol. 49, no. 2, pp. 521-9, 2016.
, “Genetic Counseling and Testing for Alzheimer's Disease and Frontotemporal Lobar Degeneration: An Italian Consensus Protocol.”, J Alzheimers Dis, vol. 51, no. 1, pp. 277-91, 2016.
, “Genetic Influences on Plasma Homocysteine Levels in African Americans and Yoruba Nigerians.”, J Alzheimers Dis, vol. 49, no. 4, pp. 991-1003, 2016.
, “Genetic Influences on Plasma Homocysteine Levels in African Americans and Yoruba Nigerians.”, J Alzheimers Dis, vol. 49, no. 4, pp. 991-1003, 2016.
, “Genetic Influences on Plasma Homocysteine Levels in African Americans and Yoruba Nigerians.”, J Alzheimers Dis, vol. 49, no. 4, pp. 991-1003, 2016.
, “Greater specificity for cerebrospinal fluid P-tau231 over P-tau181 in the differentiation of healthy controls from Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 93-100, 2016.
, “Greater specificity for cerebrospinal fluid P-tau231 over P-tau181 in the differentiation of healthy controls from Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 93-100, 2016.
, “Greater than the Sum of Its Parts: δ can be Constructed from Item Level Data.”, J Alzheimers Dis, vol. 49, no. 2, pp. 571-9, 2016.
, “GSK-3β is Dephosphorylated by PP2A in a Leu309 Methylation-Independent Manner.”, J Alzheimers Dis, vol. 49, no. 2, pp. 365-75, 2016.
, “GSK-3β is Dephosphorylated by PP2A in a Leu309 Methylation-Independent Manner.”, J Alzheimers Dis, vol. 49, no. 2, pp. 365-75, 2016.
, “Gx-50 Inhibits Neuroinflammation via α7 nAChR Activation of the JAK2/STAT3 and PI3K/AKT Pathways.”, J Alzheimers Dis, vol. 50, no. 3, pp. 859-71, 2016.
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