Biblio
“Intranasal TAT-haFGF Improves Cognition and Amyloid-β Pathology in an AβPP/PS1 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 985-90, 2016.
, “Aniracetam: An Evidence-Based Model for Preventing the Accumulation of Amyloid-β Plaques in Alzheimer's Disease.”, J Alzheimers Dis, vol. 98, no. 4, pp. 1235-1241, 2024.
, “REST and stress resistance in ageing and Alzheimer's disease.”, Nature, vol. 507, no. 7493, pp. 448-54, 2014.
, “Dysregulation and Dislocation of SFPQ Disturbed DNA Organization in Alzheimer's Disease and Frontotemporal Dementia.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1311-1321, 2018.
, “A Tale of Two Diseases: Exploring Mechanisms Linking Diabetes Mellitus with Alzheimer's Disease.”, J Alzheimers Dis, vol. 85, no. 2, pp. 485-501, 2022.
, “Hemisphere Asymmetry of Response to Pharmacologic Treatment in an Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 51, no. 2, pp. 333-8, 2016.
, “Overexpression of Metallothionein-1 Modulates the Phenotype of the Tg2576 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 81-95, 2016.
, “Palmitate Increases β-site AβPP-Cleavage Enzyme 1 Activity and Amyloid-β Genesis by Evoking Endoplasmic Reticulum Stress and Subsequent C/EBP Homologous Protein Activation.”, J Alzheimers Dis, vol. 57, no. 3, pp. 907-925, 2017.
, “Early-Life Exposure to Lead (Pb) Alters the Expression of microRNA that Target Proteins Associated with Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1257-64, 2016.
, “Neuroinflammation in Alzheimer's disease and mild cognitive impairment: a field in its infancy.”, J Alzheimers Dis, vol. 19, no. 1, pp. 355-61, 2010.
, “Olfactory Dysfunction and Alzheimer's Disease: A Review.”, J Alzheimers Dis, vol. 99, no. 3, pp. 811-827, 2024.
, “A Long Journey into Aging, Brain Aging, and Alzheimer's Disease Following the Oxidative Stress Tracks.”, J Alzheimers Dis, vol. 62, no. 3, pp. 1319-1335, 2018.
, “Gradual Cerebral Hypoperfusion Impairs Fear Conditioning and Object Recognition Learning and Memory in Mice: Potential Roles of Neurodegeneration and Cholinergic Dysfunction.”, J Alzheimers Dis, vol. 61, no. 1, pp. 283-293, 2018.
, “Hypoxia, Oxidative Stress, and Inflammation: Three Faces of Neurodegenerative Diseases.”, J Alzheimers Dis, vol. 82, no. s1, pp. S109-S126, 2021.
, “α-Lipoic Acid Has the Potential to Normalize Copper Metabolism, Which Is Dysregulated in Alzheimer's Disease.”, J Alzheimers Dis, vol. 85, no. 2, pp. 715-728, 2022.
, “Divergence of human and mouse brain transcriptome highlights Alzheimer disease pathways.”, Proc Natl Acad Sci U S A, vol. 107, no. 28, pp. 12698-703, 2010.
, “Pharmacodynamic and Clinical Effects of Ginkgo Biloba Extract EGb 761 and Its Phytochemical Components in Alzheimer's Disease.”, J Alzheimers Dis, vol. 101, no. s1, pp. S285-S298, 2024.
, “Spatial Memory Impairment is Associated with Intraneural Amyloid-β Immunoreactivity and Dysfunctional Arc Expression in the Hippocampal-CA3 Region of a Transgenic Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 69-79, 2016.
, “Cholinesterase Inhibitor Therapy in Alzheimer's Disease: The Limits and Tolerability of Irreversible CNS-Selective Acetylcholinesterase Inhibition in Primates.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1285-1294, 2017.
, “Ubisol-Q10 (a Nanomicellar Water-Soluble Formulation of CoQ10) Treatment Inhibits Alzheimer-Type Behavioral and Pathological Symptoms in a Double Transgenic Mouse (TgAPEswe, PSEN1dE9) Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 221-236, 2018.
, “Astrocytes release HspB1 in response to amyloid-β exposure in vitro.”, J Alzheimers Dis, vol. 49, no. 1, pp. 251-63, 2016.
, “Senescence Targeting Methods Impact Alzheimer's Disease Features in 3xTg Mice.”, J Alzheimers Dis, vol. 97, no. 4, pp. 1751-1763, 2024.
, “Cigarette Smoke-Induced Alterations in Frontal White Matter Lipid Profiles Demonstrated by MALDI-Imaging Mass Spectrometry: Relevance to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 151-63, 2016.
, “Enalapril Alone or Co-Administered with Losartan Rescues Cerebrovascular Dysfunction, but not Mnemonic Deficits or Amyloidosis in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1183-95, 2016.
, “Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse.”, J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
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