Biblio

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Journal Article
Y. Kim, Kim, C., Jang, H. Young, and Mook-Jung, I., “Inhibition of Cholesterol Biosynthesis Reduces γ-Secretase Activity and Amyloid-β Generation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1057-68, 2016.
B. S. Reive, Lau, V., Sánchez-Lafuente, C. L., Henri-Bhargava, A., Kalynchuk, L. E., Tremblay, M. - È., and Caruncho, H. J., “The Inflammation-Induced Dysregulation of Reelin Homeostasis Hypothesis of Alzheimer's Disease.”, J Alzheimers Dis, vol. 100, no. 4, pp. 1099-1119, 2024.
A. Arner, Rockenstein, E., Mante, M., Florio, J., Masliah, D., Salehi, B., Adame, A., Overk, C., Masliah, E., and Rissman, R. A., “Increased Vulnerability of the Hippocampus in Transgenic Mice Overexpressing APP and Triple Repeat Tau.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1201-1219, 2018.
B. M. Law, Guest, A. L., Pullen, M. W. J., Perkinton, M. S., and Williams, R. J., “Increased Foxo3a Nuclear Translocation and Activity is an Early Neuronal Response to βγ-Secretase-Mediated Processing of the Amyloid-β Protein Precursor: Utility of an AβPP-GAL4 Reporter Assay.”, J Alzheimers Dis, vol. 61, no. 2, pp. 673-688, 2018.
D. Fang, Zhang, Z., Li, H., Yu, Q., Douglas, J. T., Bratasz, A., Kuppusamy, P., and Yan, S. ShiDu, “Increased Electron Paramagnetic Resonance Signal Correlates with Mitochondrial Dysfunction and Oxidative Stress in an Alzheimer's disease Mouse Brain.”, J Alzheimers Dis, vol. 51, no. 2, pp. 571-80, 2016.
A. Ishikawa, Tokunaga, M., Maeda, J., Minamihisamatsu, T., Shimojo, M., Takuwa, H., Ono, M., Ni, R., Hirano, S., Kuwabara, S., Ji, B., Zhang, M. - R., Aoki, I., Suhara, T., Higuchi, M., and Sahara, N., “In Vivo Visualization of Tau Accumulation, Microglial Activation, and Brain Atrophy in a Mouse Model of Tauopathy rTg4510.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1037-1052, 2018.
T. Saksida, Koprivica, I., Vujičić, M., Stošić-Grujičić, S., Perović, M., Kanazir, S., and Stojanović, I., “Impaired IL-17 Production in Gut-Residing Immune Cells of 5xFAD Mice with Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 61, no. 2, pp. 619-630, 2018.
S. Simonovitch, Schmukler, E., Bespalko, A., Iram, T., Frenkel, D., Holtzman, D. M., Masliah, E., Michaelson, D. M., and Pinkas-Kramarski, R., “Impaired Autophagy in APOE4 Astrocytes.”, J Alzheimers Dis, vol. 51, no. 3, pp. 915-27, 2016.
A. Merelli, Repetto, M., Lazarowski, A., and Auzmendi, J., “Hypoxia, Oxidative Stress, and Inflammation: Three Faces of Neurodegenerative Diseases.”, J Alzheimers Dis, vol. 82, no. s1, pp. S109-S126, 2021.
P. Jul, Volbracht, C., de Jong, I. E. M., Helboe, L., Elvang, A. Brandt, and Pedersen, J. Torleif, “Hyperactivity with Agitative-Like Behavior in a Mouse Tauopathy Model.”, J Alzheimers Dis, vol. 49, no. 3, pp. 783-95, 2016.
M. Romeo, Stravalaci, M., Beeg, M., Rossi, A., Fiordaliso, F., Corbelli, A., Salmona, M., Gobbi, M., Cagnotto, A., and Diomede, L., “Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 857-871, 2017.
S. Kumfu, Charununtakorn, S. T., Jaiwongkam, T., Chattipakorn, N., and Chattipakorn, S. C., “Humanin Exerts Neuroprotection During Cardiac Ischemia-Reperfusion Injury.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1343-1353, 2018.
J. M. Castellano, Kim, J., Stewart, F. R., Jiang, H., DeMattos, R. B., Patterson, B. W., Fagan, A. M., Morris, J. C., Mawuenyega, K. G., Cruchaga, C., Goate, A. M., Bales, K. R., Paul, S. M., Bateman, R. J., and Holtzman, D. M., “Human apoE isoforms differentially regulate brain amyloid-β peptide clearance.”, Sci Transl Med, vol. 3, no. 89, p. 89ra57, 2011.
M. Díaz, Fabelo, N., Casañas-Sánchez, V., Marin, R., Gómez, T., Quinto-Alemany, D., and Pérez, J. A., “Hippocampal Lipid Homeostasis in APP/PS1 Mice is Modulated by a Complex Interplay Between Dietary DHA and Estrogens: Relevance for Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 2, pp. 459-81, 2016.
J. Valentin-Escalera, Leclerc, M., and Calon, F., “High-Fat Diets in Animal Models of Alzheimer's Disease: How Can Eating Too Much Fat Increase Alzheimer's Disease Risk?”, J Alzheimers Dis, vol. 97, no. 3, pp. 977-1005, 2024.
A. Manousopoulou, Saito, S., Yamamoto, Y., Al-Daghri, N. M., Ihara, M., Carare, R. O., and Garbis, S. D., “Hemisphere Asymmetry of Response to Pharmacologic Treatment in an Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 51, no. 2, pp. 333-8, 2016.
S. Shi, Liang, D., Bao, M., Xie, Y., Xu, W., Wang, L., Wang, Z., and Qiao, Z., “Gx-50 Inhibits Neuroinflammation via α7 nAChR Activation of the JAK2/STAT3 and PI3K/AKT Pathways.”, J Alzheimers Dis, vol. 50, no. 3, pp. 859-71, 2016.
D. Cuervo-Zanatta, Garcia-Mena, J., and Perez-Cruz, C., “Gut Microbiota Alterations and Cognitive Impairment Are Sexually Dissociated in a Transgenic Mice Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 82, no. s1, pp. S195-S214, 2021.
D. Chu, Tan, J., Xie, S., Jin, N., Yin, X., Gong, C. - X., Iqbal, K., and Liu, F., “GSK-3β is Dephosphorylated by PP2A in a Leu309 Methylation-Independent Manner.”, J Alzheimers Dis, vol. 49, no. 2, pp. 365-75, 2016.
J. Mehla, Lacoursiere, S., Stuart, E., McDonald, R. J., and Mohajerani, M. H., “Gradual Cerebral Hypoperfusion Impairs Fear Conditioning and Object Recognition Learning and Memory in Mice: Potential Roles of Neurodegeneration and Cholinergic Dysfunction.”, J Alzheimers Dis, vol. 61, no. 1, pp. 283-293, 2018.
R. Weissmann, Hüttenrauch, M., Kacprowski, T., Bouter, Y., Pradier, L., Bayer, T. A., Kuss, A. W., and Wirths, O., “Gene Expression Profiling in the APP/PS1KI Mouse Model of Familial Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 397-409, 2016.
W. Zhang, Guo, Y., Li, B., Zhang, Q., Liu, J. - H., Gu, G. - J., Wang, J. - H., Bao, R. - K., Chen, Y. - J., and Xu, J. - R., “GDF11 Rejuvenates Cerebrovascular Structure and Function in an Animal Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 2, pp. 807-819, 2018.
G. He, Luo, W., Li, P., Remmers, C., Netzer, W. J., Hendrick, J., Bettayeb, K., Flajolet, M., Gorelick, F., Wennogle, L. P., and Greengard, P., “Gamma-secretase activating protein is a therapeutic target for Alzheimer's disease.”, Nature, vol. 467, no. 7311, pp. 95-8, 2010.
S. Tok, Ahnaou, A., and Drinkenburg, W., “Functional Neurophysiological Biomarkers of Early-Stage Alzheimer's Disease: A Perspective of Network Hyperexcitability in Disease Progression.”, J Alzheimers Dis, vol. 88, no. 3, pp. 809-836, 2022.
O. J. L'Esperance, McGhee, J., Davidson, G., Niraula, S., Smith, A. S., Sosunov, A. A., Yan, S. ShiDu, and Subramanian, J., “Functional Connectivity Favors Aberrant Visual Network c-Fos Expression Accompanied by Cortical Synapse Loss in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 101, no. 1, pp. 111-131, 2024.

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