Biblio
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“Application of Weighted Gene Co-Expression Network Analysis to Explore the Key Genes in Alzheimer's Disease.”, J Alzheimers Dis, vol. 65, no. 4, pp. 1353-1364, 2018.
, “Neuron-Specific Apolipoprotein E4 (1-272) Fragment Induces Tau Hyperphosphorylation and Axonopathy via Triggering Endoplasmic Reticulum Stress.”, J Alzheimers Dis, vol. 71, no. 2, pp. 597-611, 2019.
, “Excessive Sedentary Time Is Associated with Cognitive Decline in Older Patients with Minor Ischemic Stroke.”, J Alzheimers Dis, vol. 96, no. 1, pp. 173-181, 2023.
, “The Experiences of People with Dementia and Their Caregivers in Dementia Diagnosis.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1203-1211, 2017.
, “Antihypertensive Medication Class and the Risk of Dementia and Cognitive Decline in Older Adults: A Secondary Analysis of the Prospective HELIAD Cohort.”, J Alzheimers Dis, vol. 89, no. 2, pp. 709-719, 2022.
, “Frequency-Dependent Changes in the Amplitude of Low-Frequency Fluctuations in Mild Cognitive Impairment with Mild Depression.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1175-1187, 2017.
, “Type 1 Diabetes Mellitus and Cognitive Impairments: A Systematic Review.”, J Alzheimers Dis, vol. 57, no. 1, pp. 29-36, 2017.
, “Astrocytic GluN2A and GluN2B Oppose the Synaptotoxic Effects of Amyloid-β1-40 in Hippocampal Cells.”, J Alzheimers Dis, vol. 54, no. 1, pp. 135-48, 2016.
, “Prediction of Conversion to Alzheimer's Disease with Longitudinal Measures and Time-To-Event Data.”, J Alzheimers Dis, vol. 58, no. 2, pp. 361-371, 2017.
, “miR-302 Attenuates Amyloid-β-Induced Neurotoxicity through Activation of Akt Signaling.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1083-98, 2016.
, “Pyk2 is a Novel Tau Tyrosine Kinase that is Regulated by the Tyrosine Kinase Fyn.”, J Alzheimers Dis, vol. 64, no. 1, pp. 205-221, 2018.
, “The Relationship of Brain Amyloid Load and APOE Status to Regional Cortical Thinning and Cognition in the ADNI Cohort.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1269-1282, 2017.
, “Reproductive history and risk of cognitive impairment in elderly women: a cross-sectional study in eastern China.”, J Alzheimers Dis, vol. 49, no. 1, pp. 139-47, 2016.
, “Vascular Cognitive Impairment and the Gut Microbiota.”, J Alzheimers Dis, vol. 63, no. 4, pp. 1209-1222, 2018.
, “Sirtuin 3 Mediates Tau Deacetylation.”, J Alzheimers Dis, vol. 69, no. 2, pp. 355-362, 2019.
, “Multifunctional Compound AD-35 Improves Cognitive Impairment and Attenuates the Production of TNF-α and IL-1β in an Aβ25-35-induced Rat Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1403-1417, 2017.
, “S-Adenosylmethionine Attenuates Oxidative Stress and Neuroinflammation Induced by Amyloid-β Through Modulation of Glutathione Metabolism.”, J Alzheimers Dis, vol. 58, no. 2, pp. 549-558, 2017.
, “TRPC1 Null Exacerbates Memory Deficit and Apoptosis Induced by Amyloid-β.”, J Alzheimers Dis, vol. 63, no. 2, pp. 761-772, 2018.
, “A Brief Ischemic Postconditioning Protects Against Amyloid-β Peptide Neurotoxicity by Downregulating MLK3-MKK3/6-P38MAPK Signal in Rat Hippocampus.”, J Alzheimers Dis, vol. 71, no. 2, pp. 671-684, 2019.
, “Tau Pathology Promotes the Reorganization of the Extracellular Matrix and Inhibits the Formation of Perineuronal Nets by Regulating the Expression and the Distribution of Hyaluronic Acid Synthases.”, J Alzheimers Dis, vol. 57, no. 2, pp. 395-409, 2017.
, “Age and its association with low insulin and high amyloid-β peptides in blood.”, J Alzheimers Dis, vol. 49, no. 1, pp. 129-37, 2016.
, “Greater Regional Cortical Thickness is Associated with Selective Vulnerability to Atrophy in Alzheimer's Disease, Independent of Amyloid Load and APOE Genotype.”, J Alzheimers Dis, vol. 69, no. 1, pp. 145-156, 2019.
, “The Utility of the Cognitive Function Instrument (CFI) to Detect Cognitive Decline in Non-Demented Older Adults.”, J Alzheimers Dis, vol. 60, no. 2, pp. 427-437, 2017.
, “Transthyretin Suppresses Amyloid-β Secretion by Interfering with Processing of the Amyloid-β Protein Precursor.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1263-75, 2016.
, “Diosgenin Attenuates Lipopolysaccharide-Induced Parkinson's Disease by Inhibiting the TLR/NF-κB Pathway.”, J Alzheimers Dis, vol. 64, no. 3, pp. 943-955, 2018.
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