Biblio

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F. Licastro, Raschi, E., Carbone, I., and Porcellini, E., “Variants in Antiviral Genes are Risk Factors for Cognitive Decline and Dementia.”, J Alzheimers Dis, 2015.
S. Libard, Walter, J., and Alafuzoff, I., “In vivo Characterization of Biochemical Variants of Amyloid-β in Subjects with Idiopathic Normal Pressure Hydrocephalus and Alzheimer's Disease Neuropathological Change.”, J Alzheimers Dis, vol. 80, no. 3, pp. 1003-1012, 2021.
S. Libard, Laurell, K., Cesarini, K. Giuliana, and Alafuzoff, I., “Progression of Alzheimer's Disease-Related Pathology and Cell Counts in a Patient with Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1451-1462, 2018.
J. - W. Liang, Fang, Z. - Y., Huang, Y., Liuyang, Z. - Y., Zhang, X. - L., Wang, J. - L., Wei, H., Wang, J. - Z., Wang, X. - C., Zeng, J., and Liu, R., “Application of Weighted Gene Co-Expression Network Analysis to Explore the Key Genes in Alzheimer's Disease.”, J Alzheimers Dis, vol. 65, no. 4, pp. 1353-1364, 2018.
X. Liang, Wang, Y., Li, S., Fan, J., Zhou, F., Li, X., Li, S., and Li, Y., “Enhancing Rab7 Activity by Inhibiting TBC1D5 Expression Improves Mitophagy in Alzheimer's Disease Models.”, J Alzheimers Dis, vol. 100, no. 1, pp. 279-296, 2024.
T. Liang, Xue, F., Hang, W., Wen, B., Zhang, Q., Chen, J., Liu, X., and Chen, J., “Neuron-Specific Apolipoprotein E4 (1-272) Fragment Induces Tau Hyperphosphorylation and Axonopathy via Triggering Endoplasmic Reticulum Stress.”, J Alzheimers Dis, vol. 71, no. 2, pp. 597-611, 2019.
H. Liang, Yin, X., Chen, T., Zhang, Y., Zhang, Q., Lin, J., Yin, H., Tang, J., He, Y., Xia, P., Zhu, Y., Li, H., Mo, Y., Li, Y., Wang, Y., Yang, X., and Hu, Z., “Excessive Sedentary Time Is Associated with Cognitive Decline in Older Patients with Minor Ischemic Stroke.”, J Alzheimers Dis, vol. 96, no. 1, pp. 173-181, 2023.
J. Liang, LaFleur, B., Hussainy, S., and Perry, G., “Gene Co-Expression Analysis of Multiple Brain Tissues Reveals Correlation of FAM222A Expression with Multiple Alzheimer's Disease-Related Genes.”, J Alzheimers Dis, vol. 99, no. s2, pp. S249-S263, 2024.
Y. Lian, Xiao, L. Dongxia, Zeng, F., Wu, X., Wang, Z., and Ren, H., “The Experiences of People with Dementia and Their Caregivers in Dementia Diagnosis.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1203-1211, 2017.
I. Liampas, Hatzimanolis, A., Siokas, V., Yannakoulia, M., Kosmidis, M. H., Sakka, P., Hadjigeorgiou, G. M., Scarmeas, N., and Dardiotis, E., “Antihypertensive Medication Class and the Risk of Dementia and Cognitive Decline in Older Adults: A Secondary Analysis of the Prospective HELIAD Cohort.”, J Alzheimers Dis, vol. 89, no. 2, pp. 709-719, 2022.
Y. Li, Jing, B., Liu, H., Li, Y., Gao, X., Li, Y., Mu, B., Yu, H., Cheng, J., Barker, P. B., Wang, H., and Han, Y., “Frequency-Dependent Changes in the Amplitude of Low-Frequency Fluctuations in Mild Cognitive Impairment with Mild Depression.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1175-1187, 2017.
W. Li, Huang, E., and Gao, S., “Type 1 Diabetes Mellitus and Cognitive Impairments: A Systematic Review.”, J Alzheimers Dis, vol. 57, no. 1, pp. 29-36, 2017.
Y. Li, Chang, L., Song, Y., Gao, X., Roselli, F., Liu, J., Zhou, W., Fang, Y., Ling, W., Li, H., Almeida, O. F. X., and Wu, Y., “Astrocytic GluN2A and GluN2B Oppose the Synaptotoxic Effects of Amyloid-β1-40 in Hippocampal Cells.”, J Alzheimers Dis, vol. 54, no. 1, pp. 135-48, 2016.
K. Li, Chan, W., Doody, R. S., Quinn, J., and Luo, S., “Prediction of Conversion to Alzheimer's Disease with Longitudinal Measures and Time-To-Event Data.”, J Alzheimers Dis, vol. 58, no. 2, pp. 361-371, 2017.
H. - H. Li, Lin, S. - L., Huang, C. - N., Lu, F. - J., Chiu, P. - Y., Huang, W. - N., Lai, T. - J., and Lin, C. - L., “miR-302 Attenuates Amyloid-β-Induced Neurotoxicity through Activation of Akt Signaling.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1083-98, 2016.
C. Li, Loewenstein, D. A., Duara, R., Cabrerizo, M., Barker, W., and Adjouadi, M., “The Relationship of Brain Amyloid Load and APOE Status to Regional Cortical Thinning and Cognition in the ADNI Cohort.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1269-1282, 2017.
C. Li and Götz, J., “Pyk2 is a Novel Tau Tyrosine Kinase that is Regulated by the Tyrosine Kinase Fyn.”, J Alzheimers Dis, vol. 64, no. 1, pp. 205-221, 2018.
F. - D. Li, He, F., Chen, T. - R., Xiao, Y. - Y., Lin, S. - T., Shen, W., Wang, X. - Y., Zhai, Y. - J., Shang, X. - P., and Lin, J. - F., “Reproductive history and risk of cognitive impairment in elderly women: a cross-sectional study in eastern China.”, J Alzheimers Dis, vol. 49, no. 1, pp. 139-47, 2016.
S. Li, Yin, J., Nielsen, M., Beach, T. G., Guo, L., and Shi, J., “Sirtuin 3 Mediates Tau Deacetylation.”, J Alzheimers Dis, vol. 69, no. 2, pp. 355-362, 2019.
S. Li, Shao, Y., Li, K., HuangFu, C., Wang, W., Liu, Z., Cai, Z., and Zhao, B., “Vascular Cognitive Impairment and the Gut Microbiota.”, J Alzheimers Dis, vol. 63, no. 4, pp. 1209-1222, 2018.
L. Li, Xu, S., Liu, L., Feng, R., Gong, Y., Zhao, X., Li, J., Cai, J., Feng, N., Wang, L., Wang, X., and Peng, Y., “Multifunctional Compound AD-35 Improves Cognitive Impairment and Attenuates the Production of TNF-α and IL-1β in an Aβ25-35-induced Rat Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1403-1417, 2017.
Q. Li, Cui, J., Fang, C., Liu, M., Min, G., and Li, L., “S-Adenosylmethionine Attenuates Oxidative Stress and Neuroinflammation Induced by Amyloid-β Through Modulation of Glutathione Metabolism.”, J Alzheimers Dis, vol. 58, no. 2, pp. 549-558, 2017.
D. Li, Xie, Q., Xie, J., Ni, M., Wang, J., Gao, Y., Wang, Y., and Tang, Q., “Cerebrospinal Fluid Proteomics Identifies Potential Biomarkers for Early-Onset Alzheimer's Disease.”, J Alzheimers Dis, vol. 100, no. 1, pp. 261-277, 2024.
M. Li, Liu, E., Zhou, Q., Li, S., Wang, X., Liu, Y., Wang, L., Sun, D., Ye, J., Gao, Y., Yang, X., Liu, J., Yang, Y., and Wang, J. - Z., “TRPC1 Null Exacerbates Memory Deficit and Apoptosis Induced by Amyloid-β.”, J Alzheimers Dis, vol. 63, no. 2, pp. 761-772, 2018.
H. Li, Luo, X. - B., Xu, Y., and Hou, X. - Y., “A Brief Ischemic Postconditioning Protects Against Amyloid-β Peptide Neurotoxicity by Downregulating MLK3-MKK3/6-P38MAPK Signal in Rat Hippocampus.”, J Alzheimers Dis, vol. 71, no. 2, pp. 671-684, 2019.

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