Biblio

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Journal Article
E. A. J. Willemse, Durieux-Lu, S., van der Flier, W. M., Pijnenburg, Y. A. L., de Jonge, R., and Teunissen, C. E., “Stability of Progranulin Under Pre-Analytical Conditions in Serum and Cerebrospinal Fluid.”, J Alzheimers Dis, vol. 53, no. 1, pp. 107-16, 2016.
A. Tiiman, Luo, J., Wallin, C., Olsson, L., Lindgren, J., Jarvet, J., Per, R., Sholts, S. B., Rahimipour, S., Abrahams, J. Pieter, Karlström, A. Eriksson, Gräslund, A., and Wärmländer, S. K. T. S., “Specific Binding of Cu(II) Ions to Amyloid-Beta Peptides Bound to Aggregation-Inhibiting Molecules or SDS Micelles Creates Complexes that Generate Radical Oxygen Species.”, J Alzheimers Dis, vol. 54, no. 3, pp. 971-982, 2016.
S. Seddighi, Varma, V. R., An, Y., Varma, S., Beason-Held, L. L., Tanaka, T., Kitner-Triolo, M. H., Kraut, M. A., Davatzikos, C., and Thambisetty, M., “SPARCL1 Accelerates Symptom Onset in Alzheimer's Disease and Influences Brain Structure and Function During Aging.”, J Alzheimers Dis, vol. 61, no. 1, pp. 401-414, 2018.
S. Seddighi, Varma, V. R., An, Y., Varma, S., Beason-Held, L. L., Tanaka, T., Kitner-Triolo, M. H., Kraut, M. A., Davatzikos, C., and Thambisetty, M., “SPARCL1 Accelerates Symptom Onset in Alzheimer's Disease and Influences Brain Structure and Function During Aging.”, J Alzheimers Dis, vol. 61, no. 1, pp. 401-414, 2018.
W. Wang, Zhao, F., Torres, S., Harris, P. L. R., Wang, X., Peng, L., Siedlak, S. L., and Zhu, X., “Space-Like Irradiation Exacerbated Cognitive Deficits and Amyloid Pathology in CRND8 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, 2024.
C. Wu, Ma, Y. - H., Hu, H., Zhao, B., and Tan, L., “Soluble TREM2, Alzheimer's Disease Pathology, and Risk for Progression of Cerebral Small Vessel Disease: A Longitudinal Study.”, J Alzheimers Dis, vol. 92, no. 1, pp. 311-322, 2023.
P. Cañabate, Martínez, G., Rosende-Roca, M., Moreno, M., Preckler, S., Valero, S., Sotolongo, O., Hernandez, I., Alegret, M., Ortega, G., Espinosa, A., Mauleón, A., Vargas, L., Rodríguez, O., Abdelnour, C., Sánchez, D., Martín, E., Ruiz, A., Tárraga, L., and Boada, M., “Social Representation of Dementia: An Analysis of 5,792 Consecutive Cases Evaluated in a Memory Clinic.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1099-1108, 2017.
M. Lozupone, Panza, F., Piccininni, M., Copetti, M., Sardone, R., Imbimbo, B. P., Stella, E., D'Urso, F., Barulli, M. Rosaria, Battista, P., Grasso, A., Tortelli, R., Capozzo, R., Coppola, F., Abbrescia, D. Isabel, Bellomo, A., Giannelli, G., Quaranta, N., Seripa, D., and Logroscino, G., “Social Dysfunction in Older Age and Relationships with Cognition, Depression, and Apathy: The GreatAGE Study.”, J Alzheimers Dis, vol. 65, no. 3, pp. 989-1000, 2018.
M. J. Angevaare, Pieters, J. A., Twisk, J. W. R., and Van Hout, H. P. J., “Social Activity and Cognitive Decline in Older Residents of Long-Term Care Facilities: A Cohort Study.”, J Alzheimers Dis, vol. 98, no. 2, pp. 433-443, 2024.
K. Kitagawa, Toi, S., Hosoya, M., Seki, M., Yamagishi, S., Hoshino, T., and Yoshizawa, H., “Small Vessel Disease Burden Predicts Incident Dementia and Poor Functional Outcome in Independent Outpatients.”, J Alzheimers Dis, 2024.
C. - Di Chen, Zeldich, E., Khodr, C., Camara, K., Tung, T. Yu, Lauder, E. C., Mullen, P., Polanco, T. J., Liu, Y. - Y., Zeldich, D., Xia, W., Van Nostrand, W. E., Brown, L. E., Porco, J. A., and Abraham, C. R., “Small Molecule Amyloid-β Protein Precursor Processing Modulators Lower Amyloid-β Peptide Levels via cKit Signaling.”, J Alzheimers Dis, vol. 67, no. 3, pp. 1089-1106, 2019.
A. Cagnin, Fragiacomo, F., Camporese, G., Turco, M., Bussè, C., Ermani, M., and Montagnese, S., “Sleep-Wake Profile in Dementia with Lewy Bodies, Alzheimer's Disease, and Normal Aging.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1529-1536, 2017.
A. Cassa Macedo, Balouch, S., and Tabet, N., “Is Sleep Disruption a Risk Factor for Alzheimer's Disease?”, J Alzheimers Dis, vol. 58, no. 4, pp. 993-1002, 2017.
M. Igase, Ohara, M., Igase, K., Kato, T., Okada, Y., Ochi, M., Tabara, Y., Kohara, K., and Ohyagi, Y., “Skin Autofluorescence Examination as a Diagnostic Tool for Mild Cognitive Impairment in Healthy People.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1481-1487, 2017.
K. Yordanova, Koldrack, P., Heine, C., Henkel, R., Martin, M., Teipel, S., and Kirste, T., “Situation Model for Situation-Aware Assistance of Dementia Patients in Outdoor Mobility.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1461-1476, 2017.
N. R. Barthélemy, Toth, B., Manser, P. T., Sanabria-Bohórquez, S., Teng, E., Keeley, M., Bateman, R. J., Weimer, R. M., and Wildsmith, K. R., “Site-Specific Cerebrospinal Fluid Tau Hyperphosphorylation in Response to Alzheimer's Disease Brain Pathology: Not All Tau Phospho-Sites are Hyperphosphorylated.”, J Alzheimers Dis, vol. 85, no. 1, pp. 415-429, 2021.
N. R. Barthélemy, Toth, B., Manser, P. T., Sanabria-Bohórquez, S., Teng, E., Keeley, M., Bateman, R. J., Weimer, R. M., and Wildsmith, K. R., “Site-Specific Cerebrospinal Fluid Tau Hyperphosphorylation in Response to Alzheimer's Disease Brain Pathology: Not All Tau Phospho-Sites are Hyperphosphorylated.”, J Alzheimers Dis, vol. 85, no. 1, pp. 415-429, 2021.
C. N. Trumbore, “Shear-Induced Amyloid Formation in the Brain: IV. Effects on Synapses Surrounding Senile Plaque and in Plaque-Free Regions.”, J Alzheimers Dis, vol. 66, no. 1, pp. 57-73, 2018.
C. N. Trumbore, “Shear-Induced Amyloid Formation in the Brain: III. The Roles of Shear Energy and Seeding in a Proposed Shear Model.”, J Alzheimers Dis, vol. 65, no. 1, pp. 47-70, 2018.
C. N. Trumbore, “Shear-Induced Amyloid Formation in the Brain: II. An Experimental System for Monitoring Amyloid Shear Processes and Investigating Potential Spinal Tap Problems.”, J Alzheimers Dis, vol. 59, no. 2, pp. 543-557, 2017.
C. N. Trumbore, “Shear-Induced Amyloid Formation in the Brain: I. Potential Vascular and Parenchymal Processes.”, J Alzheimers Dis, vol. 54, no. 2, pp. 457-70, 2016.
A. M. Kueider, An, Y., Tanaka, T., Kitner-Triolo, M. H., Studenski, S., Ferrucci, L., and Thambisetty, M., “Sex-Dependent Associations of Serum Uric Acid with Brain Function During Aging.”, J Alzheimers Dis, vol. 60, no. 2, pp. 699-706, 2017.
A. M. Kueider, An, Y., Tanaka, T., Kitner-Triolo, M. H., Studenski, S., Ferrucci, L., and Thambisetty, M., “Sex-Dependent Associations of Serum Uric Acid with Brain Function During Aging.”, J Alzheimers Dis, vol. 60, no. 2, pp. 699-706, 2017.
L. - L. Li, Ma, Y. - H., Bi, Y. - L., Sun, F. - R., Hu, H., Hou, X. - H., Xu, W., Shen, X. - N., Dong, Q., Tan, L., Yang, J. - L., and Yu, J. - T., “Serum Uric Acid May Aggravate Alzheimer's Disease Risk by Affecting Amyloidosis in Cognitively Intact Older Adults: The CABLE Study.”, J Alzheimers Dis, vol. 81, no. 1, pp. 389-401, 2021.
P. Chatterjee, Mohammadi, M., Goozee, K., Shah, T. M., Sohrabi, H. R., Dias, C. B., Shen, K., Asih, P. R., Dave, P., Pedrini, S., Ashton, N. J., Hye, A., Taddei, K., Lovejoy, D. B., Zetterberg, H., Blennow, K., and Martins, R. N., “Serum Hepcidin Levels in Cognitively Normal Older Adults with High Neocortical Amyloid-β Load.”, J Alzheimers Dis, vol. 76, no. 1, pp. 291-301, 2020.

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