Biblio
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Author Title Type [ Year] Filters: First Letter Of Last Name is V [Clear All Filters]
“Feasibility of a Memory Clinic-Based Physical Activity Prescription Program.”, J Alzheimers Dis, vol. 53, no. 1, pp. 161-70, 2016.
, “Fine Particulate Matter, Residential Proximity to Major Roads, and Markers of Small Vessel Disease in a Memory Study Population.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1315-23, 2016.
, “Fluoxetine Treatment Induces Seizure Behavior and Premature Death in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 51, no. 3, pp. 677-82, 2016.
, “Fluoxetine Treatment Induces Seizure Behavior and Premature Death in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 51, no. 3, pp. 677-82, 2016.
, “Fluoxetine Treatment Induces Seizure Behavior and Premature Death in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 51, no. 3, pp. 677-82, 2016.
, “Formulaic Language in People with Probable Alzheimer's Disease: A Frequency-Based Approach.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1145-60, 2016.
, “Heterogeneous Language Profiles in Patients with Primary Progressive Aphasia due to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 581-90, 2016.
, “Heterogeneous Language Profiles in Patients with Primary Progressive Aphasia due to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 581-90, 2016.
, “Heterogeneous Language Profiles in Patients with Primary Progressive Aphasia due to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 581-90, 2016.
, “Heterogeneous Language Profiles in Patients with Primary Progressive Aphasia due to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 581-90, 2016.
, “High Risk of Dementia in Ventricular Enlargement with Normal Pressure Hydrocephalus Related Symptoms1.”, J Alzheimers Dis, vol. 52, no. 2, pp. 497-507, 2016.
, “Human Truncated Tau Induces Mature Neurofibrillary Pathology in a Mouse Model of Human Tauopathy.”, J Alzheimers Dis, vol. 54, no. 2, pp. 831-43, 2016.
, “Hyperactivity with Agitative-Like Behavior in a Mouse Tauopathy Model.”, J Alzheimers Dis, vol. 49, no. 3, pp. 783-95, 2016.
, “Immunization Against Specific Fragments of Neurotrophin p75 Receptor Protects Forebrain Cholinergic Neurons in the Olfactory Bulbectomized Mice.”, J Alzheimers Dis, vol. 53, no. 1, pp. 289-301, 2016.
, “Immunization Against Specific Fragments of Neurotrophin p75 Receptor Protects Forebrain Cholinergic Neurons in the Olfactory Bulbectomized Mice.”, J Alzheimers Dis, vol. 53, no. 1, pp. 289-301, 2016.
, “Immunization with Small Amyloid-β-derived Cyclopeptide Conjugates Diminishes Amyloid-β-Induced Neurodegeneration in Mice.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1111-23, 2016.
, “Immunization with Small Amyloid-β-derived Cyclopeptide Conjugates Diminishes Amyloid-β-Induced Neurodegeneration in Mice.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1111-23, 2016.
, “Integrating Biomarkers for Underlying Alzheimer's Disease in Mild Cognitive Impairment in Daily Practice: Comparison of a Clinical Decision Support System with Individual Biomarkers.”, J Alzheimers Dis, vol. 50, no. 1, pp. 261-70, 2016.
, “Intraneuronal Amylin Deposition, Peroxidative Membrane Injury and Increased IL-1β Synthesis in Brains of Alzheimer's Disease Patients with Type-2 Diabetes and in Diabetic HIP Rats.”, J Alzheimers Dis, vol. 53, no. 1, pp. 259-72, 2016.
, “The Language Profile of Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 50, no. 2, pp. 359-71, 2016.
, “A Late Life Risk Index for Severe Cognitive Impairment in Mexico.”, J Alzheimers Dis, vol. 52, no. 1, pp. 191-203, 2016.
, “Longitudinal Changes in Functional Brain Connectivity Predicts Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 377-89, 2016.
, “Longitudinal Protein Changes in Blood Plasma Associated with the Rate of Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1105-14, 2016.
, “Low Prevalence of Mixed Dementia in a Cohort of 2,000 Elderly Patients in a Memory Clinic Setting.”, J Alzheimers Dis, vol. 50, no. 3, pp. 797-806, 2016.
, “Low Serum Insulin-Like Growth Factor-I Predicts Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 2, pp. 641-9, 2016.
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