Biblio
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Author [ Title] Type Year Filters: First Letter Of Keyword is G [Clear All Filters]
“The Heritability of Frontotemporal Lobar Degeneration: Validation of Pedigree Classification Criteria in a Northern Italy Cohort.”, J Alzheimers Dis, vol. 61, no. 2, pp. 753-760, 2018.
, “The Heritability of Frontotemporal Lobar Degeneration: Validation of Pedigree Classification Criteria in a Northern Italy Cohort.”, J Alzheimers Dis, vol. 61, no. 2, pp. 753-760, 2018.
, “Hippocampal Lipid Homeostasis in APP/PS1 Mice is Modulated by a Complex Interplay Between Dietary DHA and Estrogens: Relevance for Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 2, pp. 459-81, 2016.
, “Human apoE isoforms differentially regulate brain amyloid-β peptide clearance.”, Sci Transl Med, vol. 3, no. 89, p. 89ra57, 2011.
, “Human Gut Microbiota in Health and Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 2, pp. 549-560, 2018.
, “Human Gut Microbiota in Health and Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 2, pp. 549-560, 2018.
, “Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 857-871, 2017.
, “Increased Serum Acylated Ghrelin Levels in Patients with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 61, no. 2, pp. 545-552, 2018.
, “Increased Vulnerability of the Hippocampus in Transgenic Mice Overexpressing APP and Triple Repeat Tau.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1201-1219, 2018.
, “Influence of Butyrylcholinesterase in Progression of Mild Cognitive Impairment to Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1097-1105, 2018.
, “Integrated Analysis of Alzheimer's Disease and Schizophrenia Dataset Revealed Different Expression Pattern in Learning and Memory.”, J Alzheimers Dis, vol. 51, no. 2, pp. 417-25, 2016.
, “Integrated systems approach identifies genetic nodes and networks in late-onset Alzheimer's disease.”, Cell, vol. 153, no. 3, pp. 707-20, 2013.
, “Integrating the Synergy of the Gut Microbiome into Regenerative Medicine: Relevance to Neurological Disorders.”, J Alzheimers Dis, vol. 87, no. 4, pp. 1451-1460, 2022.
, “Intranasal TAT-haFGF Improves Cognition and Amyloid-β Pathology in an AβPP/PS1 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 985-90, 2016.
, “Investigating Casual Associations Among Gut Microbiota, Metabolites, and Neurodegenerative Diseases: A Mendelian Randomization Study.”, J Alzheimers Dis, vol. 87, no. 1, pp. 211-222, 2022.
, “Investigating Casual Associations Among Gut Microbiota, Metabolites, and Neurodegenerative Diseases: A Mendelian Randomization Study.”, J Alzheimers Dis, vol. 87, no. 1, pp. 211-222, 2022.
, “Investigating Causal Relations Between Circulating Metabolites and Alzheimer's Disease: A Mendelian Randomization Study.”, J Alzheimers Dis, vol. 87, no. 1, pp. 463-477, 2022.
, “Investigating Causal Relations Between Circulating Metabolites and Alzheimer's Disease: A Mendelian Randomization Study.”, J Alzheimers Dis, vol. 87, no. 1, pp. 463-477, 2022.
, “Longitudinal Changes in Functional Brain Connectivity Predicts Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 377-89, 2016.
, “Longitudinal White and Gray Matter Response to Precision Medicine-Guided Intervention for Alzheimer's Disease.”, J Alzheimers Dis, vol. 96, no. 3, pp. 1051-1058, 2023.
, “Lower Prevalence of Alzheimer's Disease among Tibetans: Association with Religious and Genetic Factors.”, J Alzheimers Dis, vol. 50, no. 3, pp. 659-67, 2016.
, “Lower Prevalence of Alzheimer's Disease among Tibetans: Association with Religious and Genetic Factors.”, J Alzheimers Dis, vol. 50, no. 3, pp. 659-67, 2016.
, “LRP/LR Antibody Mediated Rescuing of Amyloid-β-Induced Cytotoxicity is Dependent on PrPc in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 645-57, 2016.
, “MAPT H1 Haplotype is Associated with Late-Onset Alzheimer's Disease Risk in APOEɛ4 Noncarriers: Results from the Dementia Genetics Spanish Consortium.”, J Alzheimers Dis, vol. 49, no. 2, pp. 343-52, 2016.
, “A Mercaptoacetamide-Based Class II Histone Deacetylase Inhibitor Increases Dendritic Spine Density via RasGRF1/ERK Pathway.”, J Alzheimers Dis, vol. 51, no. 2, pp. 591-604, 2016.
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