Biblio
Export 497 results:
Author [ Title] Type Year Filters: First Letter Of Keyword is N [Clear All Filters]
“Different Expression Patterns of Amyloid-β Protein Precursor Secretases in Human and Mouse Hippocampal Neurons: A Potential Contribution to Species Differences in Neuronal Susceptibility to Amyloid-β Pathogenesis.”, J Alzheimers Dis, vol. 51, no. 1, pp. 179-95, 2016.
, “Different Patterns of Correlation between Grey and White Matter Integrity Account for Behavioral and Psychological Symptoms in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 591-604, 2016.
, “Differential Mass Spectrometry Profiles of Tau Protein in the Cerebrospinal Fluid of Patients with Alzheimer's Disease, Progressive Supranuclear Palsy, and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1033-43, 2016.
, “Differential Mass Spectrometry Profiles of Tau Protein in the Cerebrospinal Fluid of Patients with Alzheimer's Disease, Progressive Supranuclear Palsy, and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1033-43, 2016.
, “Differential Membrane Toxicity of Amyloid-β Fragments by Pore Forming Mechanisms.”, J Alzheimers Dis, vol. 51, no. 3, pp. 689-99, 2016.
, “Differential Regulation of N-Methyl-D-Aspartate Receptor Subunits is an Early Event in the Actions of Soluble Amyloid-β(1-40) Oligomers on Hippocampal Neurons.”, J Alzheimers Dis, vol. 51, no. 1, pp. 197-212, 2016.
, “Dimethyl Fumarate is a Potential Therapeutic Option for Alzheimer's Disease.”, J Alzheimers Dis, vol. 85, no. 1, pp. 443-456, 2022.
, “Dimethyl Fumarate is a Potential Therapeutic Option for Alzheimer's Disease.”, J Alzheimers Dis, vol. 85, no. 1, pp. 443-456, 2022.
, “Dimethyl Fumarate is a Potential Therapeutic Option for Alzheimer's Disease.”, J Alzheimers Dis, vol. 85, no. 1, pp. 443-456, 2022.
, “Diminished CRE-Induced Plasticity is Linked to Memory Deficits in Familial Alzheimer's Disease Mice.”, J Alzheimers Dis, vol. 50, no. 2, pp. 477-89, 2016.
, “Diminished CRE-Induced Plasticity is Linked to Memory Deficits in Familial Alzheimer's Disease Mice.”, J Alzheimers Dis, vol. 50, no. 2, pp. 477-89, 2016.
, “Directed conversion of Alzheimer's disease patient skin fibroblasts into functional neurons.”, Cell, vol. 146, no. 3, pp. 359-71, 2011.
, “Discovering New Genes in the Pathways of Common Sporadic Neurodegenerative Diseases: A Bioinformatics Approach.”, J Alzheimers Dis, vol. 51, no. 1, pp. 293-312, 2016.
, “Disrupted energy metabolism and neuronal circuit dysfunction in cognitive impairment and Alzheimer's disease.”, Lancet Neurol, vol. 10, no. 2, pp. 187-98, 2011.
, “Distinctive Resting State Network Disruptions Among Alzheimer's Disease, Subcortical Vascular Dementia, and Mixed Dementia Patients.”, J Alzheimers Dis, vol. 50, no. 3, pp. 709-18, 2016.
, “Distinctive Resting State Network Disruptions Among Alzheimer's Disease, Subcortical Vascular Dementia, and Mixed Dementia Patients.”, J Alzheimers Dis, vol. 50, no. 3, pp. 709-18, 2016.
, “Divergent Longitudinal Propagation of White Matter Degradation in Logopenic and Semantic Variants of Primary Progressive Aphasia.”, J Alzheimers Dis, vol. 49, no. 3, pp. 853-61, 2016.
, “Divergent Longitudinal Propagation of White Matter Degradation in Logopenic and Semantic Variants of Primary Progressive Aphasia.”, J Alzheimers Dis, vol. 49, no. 3, pp. 853-61, 2016.
, “Does Microglial Activation Influence Hippocampal Volume and Neuronal Function in Alzheimer's Disease and Parkinson's Disease Dementia?”, J Alzheimers Dis, vol. 51, no. 4, pp. 1275-89, 2016.
, “Does Microglial Activation Influence Hippocampal Volume and Neuronal Function in Alzheimer's Disease and Parkinson's Disease Dementia?”, J Alzheimers Dis, vol. 51, no. 4, pp. 1275-89, 2016.
, “Dose-Dependent Neuroprotection and Neurotoxicity of Simvastatin through Reduction of Farnesyl Pyrophosphate in Mice Treated with Intracerebroventricular Injection of Aβ 1-42.”, J Alzheimers Dis, vol. 50, no. 2, pp. 501-16, 2016.
, “Drivers: A Biologically Contextualized, Cross-Inferential View of the Epidemiology of Neurodegenerative Disorders.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1003-22, 2016.
, “Early and Persistent O-GlcNAc Protein Modification in the Streptozotocin Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 237-249, 2018.
, “Early in vivo Effects of the Human Mutant Amyloid-β Protein Precursor (hAβPPSwInd) on the Mouse Olfactory Bulb.”, J Alzheimers Dis, vol. 49, no. 2, pp. 443-57, 2016.
, “Early in vivo Effects of the Human Mutant Amyloid-β Protein Precursor (hAβPPSwInd) on the Mouse Olfactory Bulb.”, J Alzheimers Dis, vol. 49, no. 2, pp. 443-57, 2016.
,