Biblio

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D
E. Niemantsverdriet, Goossens, J., Struyfs, H., Martin, J. - J., Goeman, J., De Deyn, P. Paul, Vanderstichele, H., and Engelborghs, S., “Diagnostic Impact of Cerebrospinal Fluid Biomarker (Pre-)Analytical Variability in Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 97-106, 2016.
O. Bousiges, Cretin, B., Lavaux, T., Philippi, N., Jung, B., Hezard, S., Heitz, C., Demuynck, C., Gabel, A., Martin-Hunyadi, C., and Blanc, F., “Diagnostic Value of Cerebrospinal Fluid Biomarkers (Phospho-Tau181, total-Tau, Aβ42, and Aβ40) in Prodromal Stage of Alzheimer's Disease and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1069-83, 2016.
S. Gelman, Palma, J., Tombaugh, G., and Ghavami, A., “Differences in Synaptic Dysfunction Between rTg4510 and APP/PS1 Mouse Models of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 195-208, 2018.
S. Gelman, Palma, J., Tombaugh, G., and Ghavami, A., “Differences in Synaptic Dysfunction Between rTg4510 and APP/PS1 Mouse Models of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 195-208, 2018.
Z. - Q. Xu, Huang, H., Chen, Y. - L., Gao, Y. - Y., Xu, J., Marshall, C., Cai, Z. - Y., and Xiao, M., “Different Expression Patterns of Amyloid-β Protein Precursor Secretases in Human and Mouse Hippocampal Neurons: A Potential Contribution to Species Differences in Neuronal Susceptibility to Amyloid-β Pathogenesis.”, J Alzheimers Dis, vol. 51, no. 1, pp. 179-95, 2016.
L. Grangeon, Paquet, C., Bombois, S., Quillard-Muraine, M., Martinaud, O., Bourre, B., Lefaucheur, R., Nicolas, G., Dumurgier, J., Gerardin, E., Jan, M., Laplanche, J. - L., Peoc'h, K., Hugon, J., Pasquier, F., Maltête, D., Hannequin, D., and Wallon, D., “Differential Diagnosis of Dementia with High Levels of Cerebrospinal Fluid Tau Protein.”, J Alzheimers Dis, vol. 51, no. 3, pp. 905-13, 2016.
N. R. Barthélemy, Gabelle, A., Hirtz, C., Fenaille, F., Sergeant, N., Schraen-Maschke, S., Vialaret, J., Buée, L., Junot, C., Becher, F., and Lehmann, S., “Differential Mass Spectrometry Profiles of Tau Protein in the Cerebrospinal Fluid of Patients with Alzheimer's Disease, Progressive Supranuclear Palsy, and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1033-43, 2016.
N. R. Barthélemy, Gabelle, A., Hirtz, C., Fenaille, F., Sergeant, N., Schraen-Maschke, S., Vialaret, J., Buée, L., Junot, C., Becher, F., and Lehmann, S., “Differential Mass Spectrometry Profiles of Tau Protein in the Cerebrospinal Fluid of Patients with Alzheimer's Disease, Progressive Supranuclear Palsy, and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1033-43, 2016.
N. R. Barthélemy, Gabelle, A., Hirtz, C., Fenaille, F., Sergeant, N., Schraen-Maschke, S., Vialaret, J., Buée, L., Junot, C., Becher, F., and Lehmann, S., “Differential Mass Spectrometry Profiles of Tau Protein in the Cerebrospinal Fluid of Patients with Alzheimer's Disease, Progressive Supranuclear Palsy, and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1033-43, 2016.
H. Kaur Rai, Kernaghan, D., Schoonmade, L., Egan, K. J., and Pot, A. Margriet, “Digital Technologies to Prevent Social Isolation and Loneliness in Dementia: A Systematic Review.”, J Alzheimers Dis, vol. 90, no. 2, pp. 513-528, 2022.
L. Qiang, Fujita, R., Yamashita, T., Angulo, S., Rhinn, H., Rhee, D., Doege, C., Chau, L., Aubry, L., Vanti, W. B., Moreno, H., and Abeliovich, A., “Directed conversion of Alzheimer's disease patient skin fibroblasts into functional neurons.”, Cell, vol. 146, no. 3, pp. 359-71, 2011.
H. Xie, Zhang, C., Wang, Y., Huang, S., Cui, W., Yang, W., Koski, L., Xu, X., Li, Y., Zheng, M., He, M., Fu, J., Shi, X., Wang, K., Tang, G., Wang, B., and Huo, Y., “Distinct Patterns of Cognitive Aging Modified by Education Level and Gender among Adults with Limited or No Formal Education: A Normative Study of the Mini-Mental State Examination.”, J Alzheimers Dis, vol. 49, no. 4, pp. 961-9, 2016.
J. A. Miller, Horvath, S., and Geschwind, D. H., “Divergence of human and mouse brain transcriptome highlights Alzheimer disease pathways.”, Proc Natl Acad Sci U S A, vol. 107, no. 28, pp. 12698-703, 2010.
S. Tu, Leyton, C. E., Hodges, J. R., Piguet, O., and Hornberger, M., “Divergent Longitudinal Propagation of White Matter Degradation in Logopenic and Semantic Variants of Primary Progressive Aphasia.”, J Alzheimers Dis, vol. 49, no. 3, pp. 853-61, 2016.
E. Gómez-Tortosa, Prieto-Jurczynska, C., Serrano, S., Franco-Macías, E., Olivié, L., Gallego, J., Guerrero-López, R., Trujillo-Tiebas, M. José, Ayuso, C., Ruiz, P. García, Pérez-Pérez, J., and Sainz, M. José, “Diversity of Cognitive Phenotypes Associated with C9ORF72 Hexanucleotide Expansion.”, J Alzheimers Dis, vol. 52, no. 1, pp. 25-31, 2016.
K. A. Southam, Vincent, A. J., and Small, D. H., “Do Microglia Default on Network Maintenance in Alzheimer's Disease?”, J Alzheimers Dis, vol. 51, no. 3, pp. 657-69, 2016.
G. Faxen-Irving, Falahati, F., Basun, H., Eriksdotter, M., Vedin, I., Wahlund, L. - O., Schultzberg, M., Hjorth, E., Palmblad, J., Cederholm, T., and Freund-Levi, Y., “Does Fatty Acid Composition in Subcutaneous Adipose Tissue Differ between Patients with Alzheimer's Disease and Cohabiting Proxies?”, J Alzheimers Dis, vol. 61, no. 2, pp. 515-519, 2018.
G. D. Femminella, Ninan, S., Atkinson, R., Fan, Z., Brooks, D. J., and Edison, P., “Does Microglial Activation Influence Hippocampal Volume and Neuronal Function in Alzheimer's Disease and Parkinson's Disease Dementia?”, J Alzheimers Dis, vol. 51, no. 4, pp. 1275-89, 2016.
T. Schütt, Helboe, L., Pedersen, L. Østergaar, Waldemar, G., Berendt, M., and Pedersen, J. Torleif, “Dogs with Cognitive Dysfunction as a Spontaneous Model for Early Alzheimer's Disease: A Translational Study of Neuropathological and Inflammatory Markers.”, J Alzheimers Dis, vol. 52, no. 2, pp. 433-49, 2016.
T. Schütt, Helboe, L., Pedersen, L. Østergaar, Waldemar, G., Berendt, M., and Pedersen, J. Torleif, “Dogs with Cognitive Dysfunction as a Spontaneous Model for Early Alzheimer's Disease: A Translational Study of Neuropathological and Inflammatory Markers.”, J Alzheimers Dis, vol. 52, no. 2, pp. 433-49, 2016.
H. Jin, Chen, T., Li, G., Wang, C., Zhang, B., Cao, X., Sha, S., Wan, Q., and Chen, L., “Dose-Dependent Neuroprotection and Neurotoxicity of Simvastatin through Reduction of Farnesyl Pyrophosphate in Mice Treated with Intracerebroventricular Injection of Aβ 1-42.”, J Alzheimers Dis, vol. 50, no. 2, pp. 501-16, 2016.
H. Jin, Chen, T., Li, G., Wang, C., Zhang, B., Cao, X., Sha, S., Wan, Q., and Chen, L., “Dose-Dependent Neuroprotection and Neurotoxicity of Simvastatin through Reduction of Farnesyl Pyrophosphate in Mice Treated with Intracerebroventricular Injection of Aβ 1-42.”, J Alzheimers Dis, vol. 50, no. 2, pp. 501-16, 2016.
H. Jin, Chen, T., Li, G., Wang, C., Zhang, B., Cao, X., Sha, S., Wan, Q., and Chen, L., “Dose-Dependent Neuroprotection and Neurotoxicity of Simvastatin through Reduction of Farnesyl Pyrophosphate in Mice Treated with Intracerebroventricular Injection of Aβ 1-42.”, J Alzheimers Dis, vol. 50, no. 2, pp. 501-16, 2016.
E
J. Paulo Alme Santos, Vizuete, A., Hansen, F., Biasibetti, R., and Gonçalves, C. - A., “Early and Persistent O-GlcNAc Protein Modification in the Streptozotocin Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 237-249, 2018.
J. Paulo Alme Santos, Vizuete, A., Hansen, F., Biasibetti, R., and Gonçalves, C. - A., “Early and Persistent O-GlcNAc Protein Modification in the Streptozotocin Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 237-249, 2018.

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