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“Short-Chain Fatty Acids and Lipopolysaccharide as Mediators Between Gut Dysbiosis and Amyloid Pathology in Alzheimer's Disease.”, J Alzheimers Dis, vol. 78, no. 2, pp. 683-697, 2020.
, “Short-Term Effects of Rhythmic Sensory Stimulation in Alzheimer's Disease: An Exploratory Pilot Study.”, J Alzheimers Dis, vol. 52, no. 2, pp. 651-60, 2016.
, “Short-Term Response is not Predictive of Long-Term Response to Acetylcholinesterase Inhibitors in Old Age Subjects with Alzheimer's Disease: A "Real World" Study.”, J Alzheimers Dis, vol. 56, no. 1, pp. 239-248, 2017.
, “Single Nucleus Transcriptome Data from Alzheimer's Disease Mouse Models Yield New Insight into Pathophysiology.”, J Alzheimers Dis, vol. 90, no. 3, pp. 1233-1247, 2022.
, “Sirtuin 2 Inhibition Improves Cognitive Performance and Acts on Amyloid-β Protein Precursor Processing in Two Alzheimer's Disease Mouse Models.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1193-207, 2016.
, “Sleep Deprivation, a Link Between Post-Traumatic Stress Disorder and Alzheimer's Disease.”, J Alzheimers Dis, vol. 79, no. 4, pp. 1443-1449, 2021.
, “Sleep Deprivation Induced Plasma Amyloid-β Transport Disturbance in Healthy Young Adults.”, J Alzheimers Dis, vol. 57, no. 3, pp. 899-906, 2017.
, “Sleep-Wake Profile in Dementia with Lewy Bodies, Alzheimer's Disease, and Normal Aging.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1529-1536, 2017.
, “Sleep-Wake Profile in Dementia with Lewy Bodies, Alzheimer's Disease, and Normal Aging.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1529-1536, 2017.
, “Small Molecule Amyloid-β Protein Precursor Processing Modulators Lower Amyloid-β Peptide Levels via cKit Signaling.”, J Alzheimers Dis, vol. 67, no. 3, pp. 1089-1106, 2019.
, “Social Dysfunction in Older Age and Relationships with Cognition, Depression, and Apathy: The GreatAGE Study.”, J Alzheimers Dis, vol. 65, no. 3, pp. 989-1000, 2018.
, “Social Dysfunction in Older Age and Relationships with Cognition, Depression, and Apathy: The GreatAGE Study.”, J Alzheimers Dis, vol. 65, no. 3, pp. 989-1000, 2018.
, “Social Dysfunction in Older Age and Relationships with Cognition, Depression, and Apathy: The GreatAGE Study.”, J Alzheimers Dis, vol. 65, no. 3, pp. 989-1000, 2018.
, “Social Isolation and Cognitive Function in Later Life: A Systematic Review and Meta-Analysis.”, J Alzheimers Dis, vol. 70, no. s1, pp. S119-S144, 2019.
, “Social Isolation and Cognitive Function in Later Life: A Systematic Review and Meta-Analysis.”, J Alzheimers Dis, vol. 70, no. s1, pp. S119-S144, 2019.
, “Social Representation of Dementia: An Analysis of 5,792 Consecutive Cases Evaluated in a Memory Clinic.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1099-1108, 2017.
, “Social support may buffer the effect of ε4 allele on cognition in older adults.”, J Alzheimers Dis, vol. 102, no. 4, pp. 967-974, 2024.
, “Social support may buffer the effect of ε4 allele on cognition in older adults.”, J Alzheimers Dis, vol. 102, no. 4, pp. 967-974, 2024.
, “The Societal Cost of Dementia in Singapore: Results from the WiSE Study.”, J Alzheimers Dis, vol. 51, no. 2, pp. 439-49, 2016.
, “The Societal Cost of Dementia in Singapore: Results from the WiSE Study.”, J Alzheimers Dis, vol. 51, no. 2, pp. 439-49, 2016.
, “The Societal Cost of Dementia in Singapore: Results from the WiSE Study.”, J Alzheimers Dis, vol. 51, no. 2, pp. 439-49, 2016.
, “Soluble amyloid beta-protein dimers isolated from Alzheimer cortex directly induce Tau hyperphosphorylation and neuritic degeneration.”, Proc Natl Acad Sci U S A, vol. 108, no. 14, pp. 5819-24, 2011.
, “Soluble Oligomers Require a Ganglioside to Trigger Neuronal Calcium Overload.”, J Alzheimers Dis, vol. 60, no. 3, pp. 923-938, 2017.
, “Soluble Oligomers Require a Ganglioside to Trigger Neuronal Calcium Overload.”, J Alzheimers Dis, vol. 60, no. 3, pp. 923-938, 2017.
, “Soluble Oligomers Require a Ganglioside to Trigger Neuronal Calcium Overload.”, J Alzheimers Dis, vol. 60, no. 3, pp. 923-938, 2017.
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