Biblio
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Author Title Type [ Year] Filters: Keyword is Alzheimer Disease [Clear All Filters]
“Longitudinal Memory Profiles in Behavioral-Variant Frontotemporal Dementia and Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 3, pp. 775-82, 2016.
, “Longitudinal Protein Changes in Blood Plasma Associated with the Rate of Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1105-14, 2016.
, “Low Plasma Cholinesterase Activities are Associated with Deficits in Spatial Orientation, Reduced Ability to Perform Basic Activities of Daily Living, and Low Body Mass Index in Patients with Progressed Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 3, pp. 801-13, 2016.
, “Lower Prevalence of Alzheimer's Disease among Tibetans: Association with Religious and Genetic Factors.”, J Alzheimers Dis, vol. 50, no. 3, pp. 659-67, 2016.
, “Maintenance of Cognitive Performance and Mood for Individuals with Alzheimer's Disease Following Consumption of a Nutraceutical Formulation: A One-Year, Open-Label Study.”, J Alzheimers Dis, vol. 51, no. 4, pp. 991-5, 2016.
, “MAPT H1 Haplotype is Associated with Late-Onset Alzheimer's Disease Risk in APOEɛ4 Noncarriers: Results from the Dementia Genetics Spanish Consortium.”, J Alzheimers Dis, vol. 49, no. 2, pp. 343-52, 2016.
, “Memory for Public Events in Mild Cognitive Impairment and Alzheimer's Disease: The Importance of Rehearsal.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1023-33, 2016.
, “A Meta-Analysis of Alzheimer's Disease Incidence and Prevalence Comparing African-Americans and Caucasians.”, J Alzheimers Dis, vol. 50, no. 1, pp. 71-6, 2016.
, “Meta-Analysis of Transcriptome Data Related to Hippocampus Biopsies and iPSC-Derived Neuronal Cells from Alzheimer's Disease Patients Reveals an Association with FOXA1 and FOXA2 Gene Regulatory Networks.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1065-82, 2016.
, “Metformin Facilitates Amyloid-β Generation by β- and γ-Secretases via Autophagy Activation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1197-208, 2016.
, “Mindfulness in the Maintenance of Cognitive Capacities in Alzheimer's Disease: A Randomized Clinical Trial.”, J Alzheimers Dis, vol. 50, no. 1, pp. 217-32, 2016.
, “A Mitochondrial Role of SV2a Protein in Aging and Alzheimer's Disease: Studies with Levetiracetam.”, J Alzheimers Dis, vol. 50, no. 1, pp. 201-15, 2016.
, “Moderate-to-High Intensity Physical Exercise in Patients with Alzheimer's Disease: A Randomized Controlled Trial.”, J Alzheimers Dis, vol. 50, no. 2, pp. 443-53, 2016.
, “Molecular markers of amnestic mild cognitive impairment among Mexican Americans.”, J Alzheimers Dis, vol. 49, no. 1, pp. 221-8, 2016.
, “Molecular Signaling Mechanisms of Natural and Synthetic Retinoids for Inhibition of Pathogenesis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 335-52, 2016.
, “Multifunctional Effect of Human Serum Albumin Reduces Alzheimer's Disease Related Pathologies in the 3xTg Mouse Model.”, J Alzheimers Dis, vol. 50, no. 1, pp. 175-88, 2016.
, “Multimodal Magnetic Resonance Imaging in Alzheimer's Disease Patients at Prodromal Stage.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1035-50, 2016.
, “Music and Memory in Alzheimer's Disease and The Potential Underlying Mechanisms.”, J Alzheimers Dis, vol. 51, no. 4, pp. 949-59, 2016.
, “Neuronal activity and amyloid plaque pathology: an update.”, J Alzheimers Dis, vol. 49, no. 1, pp. 13-9, 2016.
, “Neurophysiological assessment of Alzheimer's disease individuals by a single electroencephalographic marker.”, J Alzheimers Dis, vol. 49, no. 1, pp. 159-77, 2016.
, “Non-Verbal Episodic Memory Deficits in Primary Progressive Aphasias are Highly Predictive of Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 51, no. 2, pp. 367-76, 2016.
, “Normal Amplitude of Electroretinography and Visual Evoked Potential Responses in AβPP/PS1 Mice.”, J Alzheimers Dis, vol. 51, no. 1, pp. 21-6, 2016.
, “Novel Selective Calpain 1 Inhibitors as Potential Therapeutics in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 707-21, 2016.
, “N-truncated Aβ2-X starting with position two in sporadic Alzheimer's disease cases and two Alzheimer mouse models.”, J Alzheimers Dis, vol. 49, no. 1, pp. 101-10, 2016.
, “Nutritional Status is Associated with Faster Cognitive Decline and Worse Functional Impairment in the Progression of Dementia: The Cache County Dementia Progression Study1.”, J Alzheimers Dis, vol. 52, no. 1, pp. 33-42, 2016.
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