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Case-Control Studies
X. - X. Liu, Wu, P. - F., Liu, Y. - Z., Jiang, Y. - L., Wan, M. - D., Xiao, X. - W., Yang, Q. - J., Jiao, B., Liao, X. - X., Wang, J. - L., Liu, S. - H., Zhang, X., and Shen, L., “Association Between Serum Vitamins and the Risk of Alzheimer's Disease in Chinese Population.”, J Alzheimers Dis, vol. 85, no. 2, pp. 829-836, 2022.
V. Panzarella, Mauceri, R., Baschi, R., Maniscalco, L., Campisi, G., and Monastero, R., “Oral Health Status in Subjects with Amnestic Mild Cognitive Impairment and Alzheimer's Disease: Data from the Zabút Aging Project.”, J Alzheimers Dis, vol. 87, no. 1, pp. 173-183, 2022.
S. Ge, Kitamura, T., Zha, L., Komatsu, M., Komukai, S., Murata, F., Maeda, M., Gon, Y., Kimura, Y., Kiyohara, K., Sobue, T., and Fukuda, H., “Association of Statin Use with Dementia Risk Among Older Adults in Japan: A Nested Case-Control Study Using the LIFE Study.”, J Alzheimers Dis, vol. 100, no. 3, pp. 987-998, 2024.
Caspase 3
M. A. Burguillos, Deierborg, T., Kavanagh, E., Persson, A., Hajji, N., Garcia-Quintanilla, A., Cano, J., Brundin, P., Englund, E., Venero, J. L., and Joseph, B., “Caspase signalling controls microglia activation and neurotoxicity.”, Nature, vol. 472, no. 7343, pp. 319-24, 2011.
J. Geng, Xia, L., Li, W., Zhao, C., and Dou, F., “Cycloheximide Treatment Causes a ZVAD-Sensitive Protease-Dependent Cleavage of Human Tau in Drosophila Cells.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1161-8, 2016.
M. Rafi Khezri, Ghasemnejad-Berenji, M., and Moloodsouri, D., “The PI3K/AKT Signaling Pathway and Caspase-3 in Alzheimer's Disease: Which One Is the Beginner?”, J Alzheimers Dis, vol. 92, no. 2, pp. 391-393, 2023.
R. Pluta, Bogucka-Kocka, A., Bogucki, J., Kocki, J., and Czuczwar, S. J., “Apoptosis, Autophagy, and Mitophagy Genes in the CA3 Area in an Ischemic Model of Alzheimer's Disease with 2-Year Survival.”, J Alzheimers Dis, vol. 99, no. 4, pp. 1375-1383, 2024.
A. Rustamzadeh, Tafakhori, A., Ariaei, A., Heydari, M., Shah-Abadi, M. Ebrahimi, and Seif, F., “Targeting Caspases 3/6 and Cathepsins L/B May Decrease Laminopathy-Induced Apoptosis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 101, no. 1, pp. 211-221, 2024.
Caspase 6
A. Rustamzadeh, Tafakhori, A., Ariaei, A., Heydari, M., Shah-Abadi, M. Ebrahimi, and Seif, F., “Targeting Caspases 3/6 and Cathepsins L/B May Decrease Laminopathy-Induced Apoptosis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 101, no. 1, pp. 211-221, 2024.
Caspase 7
M. A. Burguillos, Deierborg, T., Kavanagh, E., Persson, A., Hajji, N., Garcia-Quintanilla, A., Cano, J., Brundin, P., Englund, E., Venero, J. L., and Joseph, B., “Caspase signalling controls microglia activation and neurotoxicity.”, Nature, vol. 472, no. 7343, pp. 319-24, 2011.
Caspase 8
M. A. Burguillos, Deierborg, T., Kavanagh, E., Persson, A., Hajji, N., Garcia-Quintanilla, A., Cano, J., Brundin, P., Englund, E., Venero, J. L., and Joseph, B., “Caspase signalling controls microglia activation and neurotoxicity.”, Nature, vol. 472, no. 7343, pp. 319-24, 2011.
Caspase Inhibitors
M. A. Burguillos, Deierborg, T., Kavanagh, E., Persson, A., Hajji, N., Garcia-Quintanilla, A., Cano, J., Brundin, P., Englund, E., Venero, J. L., and Joseph, B., “Caspase signalling controls microglia activation and neurotoxicity.”, Nature, vol. 472, no. 7343, pp. 319-24, 2011.
Caspases
M. A. Burguillos, Deierborg, T., Kavanagh, E., Persson, A., Hajji, N., Garcia-Quintanilla, A., Cano, J., Brundin, P., Englund, E., Venero, J. L., and Joseph, B., “Caspase signalling controls microglia activation and neurotoxicity.”, Nature, vol. 472, no. 7343, pp. 319-24, 2011.
L. - S. Liu, Bai, X. - Q., Gao, Y., Wu, Q., Ren, Z., Li, Q., Pan, L. - H., He, N. - Y., Peng, J., and Tang, Z. - H., “PCSK9 Promotes oxLDL-Induced PC12 Cell Apoptosis Through the Bcl-2/Bax-Caspase 9/3 Signaling Pathway.”, J Alzheimers Dis, vol. 57, no. 3, pp. 723-734, 2017.
Catechol O-Methyltransferase
S. Sapkota and Dixon, R. A., “A Network of Genetic Effects on Non-Demented Cognitive Aging: Alzheimer's Genetic Risk (CLU + CR1 + PICALM) Intensifies Cognitive Aging Genetic Risk (COMT + BDNF) Selectively for APOEɛ4 Carriers.”, J Alzheimers Dis, vol. 62, no. 2, pp. 887-900, 2018.
Cathepsin B
A. Rustamzadeh, Tafakhori, A., Ariaei, A., Heydari, M., Shah-Abadi, M. Ebrahimi, and Seif, F., “Targeting Caspases 3/6 and Cathepsins L/B May Decrease Laminopathy-Induced Apoptosis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 101, no. 1, pp. 211-221, 2024.
Cathepsin D
J. A. Snir, Suchy, M., Bindseil, G. A., Kovacs, M., Chronik, B. A., Hudson, R. H. E., Pasternak, S. H., and Bartha, R., “An Aspartyl Cathepsin Targeted PET Agent: Application in an Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1241-1252, 2018.
Cathepsin L
A. Rustamzadeh, Tafakhori, A., Ariaei, A., Heydari, M., Shah-Abadi, M. Ebrahimi, and Seif, F., “Targeting Caspases 3/6 and Cathepsins L/B May Decrease Laminopathy-Induced Apoptosis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 101, no. 1, pp. 211-221, 2024.
Cattle
E. Vallino Costassa, Fiorini, M., Zanusso, G., Peletto, S., Acutis, P., Baioni, E., Maurella, C., Tagliavini, F., Catania, M., Gallo, M., Faro, M. Lo, Chieppa, M. Novella, Meloni, D., D'Angelo, A., Paciello, O., Ghidoni, R., Tonoli, E., Casalone, C., and Corona, C., “Characterization of Amyloid-β Deposits in Bovine Brains.”, J Alzheimers Dis, vol. 51, no. 3, pp. 875-87, 2016.
N. Lelental, Brandner, S., Kofanova, O., Blennow, K., Zetterberg, H., Andreasson, U., Engelborghs, S., Mroczko, B., Gabryelewicz, T., Teunissen, C., Mollenhauer, B., Parnetti, L., Chiasserini, D., Molinuevo, J. Luis, Perret-Liaudet, A., Verbeek, M. M., Andreasen, N., Brosseron, F., Bahl, J. M. C., Herukka, S. - K., Hausner, L., Frölich, L., Labonte, A., Poirier, J., Miller, A. - M., Zilka, N., Kovacech, B., Urbani, A., Suardi, S., Oliveira, C., Baldeiras, I., Dubois, B., Rot, U., Lehmann, S., Skinningsrud, A., Betsou, F., Wiltfang, J., Gkatzima, O., Winblad, B., Buchfelder, M., Kornhuber, J., and Lewczuk, P., “Comparison of Different Matrices as Potential Quality Control Samples for Neurochemical Dementia Diagnostics.”, J Alzheimers Dis, vol. 52, no. 1, pp. 51-64, 2016.
Causality
N. Samudra, Ranasinghe, K., Kirsch, H., Rankin, K., and Miller, B., “Etiology and Clinical Significance of Network Hyperexcitability in Alzheimer's Disease: Unanswered Questions and Next Steps.”, J Alzheimers Dis, vol. 92, no. 1, pp. 13-27, 2023.
Cause of Death
J. Benito-León, Contador, I., Mitchell, A. J., Domingo-Santos, Á., and Bermejo-Pareja, F., “Performance on Specific Cognitive Domains and Cause of Death: A Prospective Population-Based Study in Non-Demented Older Adults (NEDICES).”, J Alzheimers Dis, vol. 51, no. 2, pp. 533-44, 2016.
D. Rizzuto, Feldman, A. L., Karlsson, I. K., Aslan, A. K. Dahl, Gatz, M., and Pedersen, N. L., “Detection of Dementia Cases in Two Swedish Health Registers: A Validation Study.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1301-1310, 2018.
R. Ono, Sakurai, T., Sugimoto, T., Uchida, K., Nakagawa, T., Noguchi, T., Komatsu, A., Arai, H., and Saito, T., “Mortality Risks and Causes of Death by Dementia Types in a Japanese Cohort with Dementia: NCGG-STORIES.”, J Alzheimers Dis, vol. 92, no. 2, pp. 487-498, 2023.
cdc42 GTP-Binding Protein
C. Saraceno, Catania, M., Paterlini, A., Fostinelli, S., Ciani, M., Zanardini, R., Binetti, G., Di Fede, G., Caroppo, P., Benussi, L., Ghidoni, R., and Bolognin, S., “Altered Expression of Circulating Cdc42 in Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1477-1483, 2018.

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