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Cell Line
G. He, Luo, W., Li, P., Remmers, C., Netzer, W. J., Hendrick, J., Bettayeb, K., Flajolet, M., Gorelick, F., Wennogle, L. P., and Greengard, P., Gamma-secretase activating protein is a therapeutic target for Alzheimer's disease., Nature, vol. 467, no. 7311, pp. 95-8, 2010.
S. A. Villeda, Plambeck, K. E., Middeldorp, J., Castellano, J. M., Mosher, K. I., Luo, J., Smith, L. K., Bieri, G., Lin, K., Berdnik, D., Wabl, R., Udeochu, J., Wheatley, E. G., Zou, B., Simmons, D. A., Xie, X. S., Longo, F. M., and Wyss-Coray, T., Young blood reverses age-related impairments in cognitive function and synaptic plasticity in mice., Nat Med, vol. 20, no. 6, pp. 659-63, 2014.
J. Geng, Xia, L., Li, W., Zhao, C., and Dou, F., Cycloheximide Treatment Causes a ZVAD-Sensitive Protease-Dependent Cleavage of Human Tau in Drosophila Cells., J Alzheimers Dis, vol. 49, no. 4, pp. 1161-8, 2016.
C. Stockburger, Miano, D., Baeumlisberger, M., Pallas, T., Arrey, T. N., Karas, M., Friedland, K., and Müller, W. E., A Mitochondrial Role of SV2a Protein in Aging and Alzheimer's Disease: Studies with Levetiracetam., J Alzheimers Dis, vol. 50, no. 1, pp. 201-15, 2016.
C. -cui Yang, Kuai, X. -xian, bin Gao, W. -, Yu, J. -chun, Wang, Q., Li, L., and Zhang, L., Morroniside-Induced PP2A Activation Antagonizes Tau Hyperphosphorylation in a Cellular Model of Neurodegeneration., J Alzheimers Dis, vol. 51, no. 1, pp. 33-44, 2016.
Y. Kim, Kim, C., Jang, H. Young, and Mook-Jung, I., Inhibition of Cholesterol Biosynthesis Reduces γ-Secretase Activity and Amyloid-β Generation., J Alzheimers Dis, vol. 51, no. 4, pp. 1057-68, 2016.
R. Raha-Chowdhury, Henderson, J. W., Raha, A. Alexander, Stott, S. R. W., Vuono, R., Foscarin, S., Wilson, L., Annus, T., Fincham, R., Allinson, K., Devalia, V., Friedland, R. P., Holland, A., and Zaman, S. H., Erythromyeloid-Derived TREM2: A Major Determinant of Alzheimer's Disease Pathology in Down Syndrome., J Alzheimers Dis, vol. 61, no. 3, pp. 1143-1162, 2018.
K. Metsla, Kirss, S., Laks, K., Sildnik, G., Palgi, M., Palumaa, T., Tõugu, V., and Palumaa, P., α-Lipoic Acid Has the Potential to Normalize Copper Metabolism, Which Is Dysregulated in Alzheimer's Disease., J Alzheimers Dis, vol. 85, no. 2, pp. 715-728, 2022.