Biblio
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“Deaths with Dementia in Indigenous and Non-Indigenous Australians: A Nationwide Study.”, J Alzheimers Dis, vol. 81, no. 4, pp. 1589-1599, 2021.
, “Diagnosing Mild Cognitive Impairment Among Racially Diverse Older Adults: Comparison of Consensus, Actuarial, and Statistical Methods.”, J Alzheimers Dis, vol. 85, no. 2, pp. 627-644, 2021.
, “Diet May Moderate the Relationship Between Arterial Stiffness and Cognitive Performance in Older Adults.”, J Alzheimers Dis, vol. 85, no. 2, pp. 815-828, 2021.
, “Droplet Degeneration of Hippocampal and Cortical Neurons Signifies the Beginning of Neuritic Plaque Formation.”, J Alzheimers Dis, vol. 85, no. 4, pp. 1701-1720, 2021.
, “Economic Burden of Alzheimer's Disease Dementia in Japan.”, J Alzheimers Dis, vol. 81, no. 1, pp. 309-319, 2021.
, “Effects of Peroral Omega-3 Fatty Acid Supplementation on Cerebrospinal Fluid Biomarkers in Patients with Alzheimer's Disease: A Randomized Controlled Trial-The OmegAD Study.”, J Alzheimers Dis, vol. 83, no. 3, pp. 1291-1301, 2021.
, “Entorhinal Perfusion Predicts Future Memory Decline, Neurodegeneration, and White Matter Hyperintensity Progression in Older Adults.”, J Alzheimers Dis, vol. 81, no. 4, pp. 1711-1725, 2021.
, “Ethnic Differences in Dementia Risk: A Systematic Review and Meta-Analysis.”, J Alzheimers Dis, vol. 80, no. 1, pp. 337-355, 2021.
, “Five-Year Change in Body Mass Index Predicts Conversion to Mild Cognitive Impairment or Dementia Only in APOE ɛ4 Allele Carriers.”, J Alzheimers Dis, vol. 81, no. 1, pp. 189-199, 2021.
, “Gut Microbiota Changes and Their Correlation with Cognitive and Neuropsychiatric Symptoms in Alzheimer's Disease.”, J Alzheimers Dis, vol. 81, no. 2, pp. 583-595, 2021.
, “High Fat Diet Mediates Amyloid-β Cleaving Enzyme 1 Phosphorylation and SUMOylation, Enhancing Cognitive Impairment in APP/PS1 Mice.”, J Alzheimers Dis, vol. 85, no. 2, pp. 863-876, 2021.
, “High Fat Diet Mediates Amyloid-β Cleaving Enzyme 1 Phosphorylation and SUMOylation, Enhancing Cognitive Impairment in APP/PS1 Mice.”, J Alzheimers Dis, vol. 85, no. 2, pp. 863-876, 2021.
, “Hippocampal Functional Connectivity and Memory Performance After Exercise Intervention in Older Adults with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 82, no. 3, pp. 1015-1031, 2021.
, “In vivo Characterization of Biochemical Variants of Amyloid-β in Subjects with Idiopathic Normal Pressure Hydrocephalus and Alzheimer's Disease Neuropathological Change.”, J Alzheimers Dis, vol. 80, no. 3, pp. 1003-1012, 2021.
, “Lipidomic Network of Mild Cognitive Impairment from the Mayo Clinic Study of Aging.”, J Alzheimers Dis, vol. 81, no. 2, pp. 533-543, 2021.
, “Loneliness, Not Social Support, Is Associated with Cognitive Decline and Dementia Across Two Longitudinal Population-Based Cohorts.”, J Alzheimers Dis, vol. 85, no. 1, pp. 295-308, 2021.
, “Loneliness, Not Social Support, Is Associated with Cognitive Decline and Dementia Across Two Longitudinal Population-Based Cohorts.”, J Alzheimers Dis, vol. 85, no. 1, pp. 295-308, 2021.
, “Longitudinal Trajectories in Cortical Thickness and Volume Atrophy: Superior Cognitive Performance Does Not Protect Against Brain Atrophy in Older Adults.”, J Alzheimers Dis, vol. 81, no. 3, pp. 1039-1052, 2021.
, “Longitudinally Increasing Elevated Asymmetric Flortaucipir Binding in a Cognitively Unimpaired Amyloid-Negative Older Individual.”, J Alzheimers Dis, vol. 85, no. 1, pp. 59-64, 2021.
, “Lung Function Impairment and the Risk of Incident Dementia: The Rotterdam Study.”, J Alzheimers Dis, vol. 82, no. 2, pp. 621-630, 2021.
, “Mitochondrial Membrane Potential Influences Amyloid-β Protein Precursor Localization and Amyloid-β Secretion.”, J Alzheimers Dis, vol. 85, no. 1, pp. 381-394, 2021.
, “Mortality After Ischemic Stroke in Patients with Alzheimer's Disease Dementia and Other Dementia Disorders.”, J Alzheimers Dis, vol. 81, no. 3, pp. 1253-1261, 2021.
, “Multiple Neurodegenerative Pathologies in an Alzheimer's Disease Patient Treated with Fornical Deep Brain Stimulation.”, J Alzheimers Dis, vol. 80, no. 4, pp. 1383-1387, 2021.
, “Mutations in the Amyloid-β Protein Precursor Reduce Mitochondrial Function and Alter Gene Expression Independent of 42-Residue Amyloid-β Peptide.”, J Alzheimers Dis, vol. 83, no. 3, pp. 1039-1049, 2021.
, “Mutations in the Amyloid-β Protein Precursor Reduce Mitochondrial Function and Alter Gene Expression Independent of 42-Residue Amyloid-β Peptide.”, J Alzheimers Dis, vol. 83, no. 3, pp. 1039-1049, 2021.
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