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S. Martire, Fuso, A., Mosca, L., Forte, E., Correani, V., Fontana, M., Scarpa, S., Maras, B., and d'Erme, M., “Bioenergetic Impairment in Animal and Cellular Models of Alzheimer's Disease: PARP-1 Inhibition Rescues Metabolic Dysfunctions.”, J Alzheimers Dis, vol. 54, no. 1, pp. 307-24, 2016.
B. Borroni, Benussi, A., Premi, E., Alberici, A., Marcello, E., Gardoni, F., Di Luca, M., and Padovani, A., “Biological, Neuroimaging, and Neurophysiological Markers in Frontotemporal Dementia: Three Faces of the Same Coin.”, J Alzheimers Dis, vol. 62, no. 3, pp. 1113-1123, 2018.
A. Papazacharias, Lozupone, M., Barulli, M. Rosaria, Capozzo, R., Imbimbo, B. P., Veneziani, F., De Blasi, R., Nardini, M., Seripa, D., Panza, F., and Logroscino, G., “Bipolar Disorder and Frontotemporal Dementia: An Intriguing Association.”, J Alzheimers Dis, vol. 55, no. 3, pp. 973-979, 2017.
A. M. Fernandez, Hervas, R., Dominguez-Fraile, M., Garrido, V. Navarro, Gomez-Gutierrez, P., Vega, M., Vitorica, J., Perez, J. J., and Aleman, I. Torres, “Blockade of the Interaction of Calcineurin with FOXO in Astrocytes Protects Against Amyloid-β-Induced Neuronal Death.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1471-8, 2016.
A. Julian, Rioux-Bilan, A., Ragot, S., Krolak-Salmon, P., Berrut, G., Dantoine, T., Hommet, C., Hanon, O., Page, G., and Paccalin, M., “Blood Inflammatory Mediators and Cognitive Decline in Alzheimer's Disease: A Two Years Longitudinal Study.”, J Alzheimers Dis, vol. 63, no. 1, pp. 87-92, 2018.
W. Tarraf, Rodríguez, C. J., Daviglus, M. L., Lamar, M., Schneiderman, N., Gallo, L., Talavera, G. A., Kaplan, R. C., Fornage, M., Conceicao, A., and González, H. M., “Blood Pressure and Hispanic/Latino Cognitive Function: Hispanic Community Health Study/Study of Latinos Results.”, J Alzheimers Dis, vol. 59, no. 1, pp. 31-42, 2017.
N. Voyle, Baker, D., Burnham, S. C., Covin, A., Zhang, Z., Sangurdekar, D. P., Hehir, C. A. Tan, Bazenet, C., Lovestone, S., Kiddle, S., and Dobson, R. J. B., “Blood Protein Markers of Neocortical Amyloid-β Burden: A Candidate Study Using SOMAscan Technology.”, J Alzheimers Dis, 2015.
S. J. Kiddle, Voyle, N., and Dobson, R. J. B., “A Blood Test for Alzheimer's Disease: Progress, Challenges, and Recommendations.”, J Alzheimers Dis, vol. 64, no. s1, pp. S289-S297, 2018.
I. Koychev, Jansen, K., Dette, A., Shi, L., and Holling, H., “Blood-Based ATN Biomarkers of Alzheimer's Disease: A Meta-Analysis.”, J Alzheimers Dis, vol. 79, no. 1, pp. 177-195, 2021.
I. Cohen-Manheim, Doniger, G. M., Sinnreich, R., Simon, E. S., Murad, H., Pinchas-Mizrachi, R., and Kark, J. D., “Body Mass Index, Height, and Socioeconomic Position in Adolescence, Their Trajectories into Adulthood, and Cognitive Function in Midlife.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1207-1221, 2017.
H. - J. Kim, Im, H. Kyun, Kim, J., Han, J. - Y., de Leon, M., Deshpande, A., and Moon, W. - J., “Brain Atrophy of Secondary REM-Sleep Behavior Disorder in Neurodegenerative Disease.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1101-9, 2016.
H. - J. Kim, Im, H. Kyun, Kim, J., Han, J. - Y., de Leon, M., Deshpande, A., and Moon, W. - J., “Brain Atrophy of Secondary REM-Sleep Behavior Disorder in Neurodegenerative Disease.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1101-9, 2016.
E. D. Huey, Lee, S., Cheran, G., Grafman, J., and Devanand, D. P., “Brain Regions Involved in Arousal and Reward Processing are Associated with Apathy in Alzheimer's Disease and Frontotemporal Dementia.”, J Alzheimers Dis, vol. 55, no. 2, pp. 551-558, 2017.
Y. Vermeiren, Janssens, J., Aerts, T., Martin, J. - J., Sieben, A., Van Dam, D., and De Deyn, P. P., “Brain Serotonergic and Noradrenergic Deficiencies in Behavioral Variant Frontotemporal Dementia Compared to Early-Onset Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1079-96, 2016.
Y. Huang, Driedonks, T. A. P., Cheng, L., Rajapaksha, H., Routenberg, D. A., Nagaraj, R., Redding, J., Arab, T., Powell, B. H., Pletniková, O., Troncoso, J. C., Zheng, L., Hill, A. F., Mahairaki, V., and Witwer, K. W., “Brain Tissue-Derived Extracellular Vesicles in Alzheimer's Disease Display Altered Key Protein Levels Including Cell Type-Specific Markers.”, J Alzheimers Dis, vol. 90, no. 3, pp. 1057-1072, 2022.
N. Boublay, Bouet, R., Dorey, J. - M., Padovan, C., Makaroff, Z., Federico, D., Gallice, I., Barrellon, M. - O., Robert, P., Moreaud, O., Rouch, I., and Krolak-Salmon, P., “Brain Volume Predicts Behavioral and Psychological Symptoms in Alzheimer's Disease.”, J Alzheimers Dis, vol. 73, no. 4, pp. 1343-1353, 2020.
F. Guillot, Kemppainen, S., Lavasseur, G., Miettinen, P. O., Laroche, S., Tanila, H., and Davis, S., “Brain-Specific Basal and Novelty-Induced Alternations in PI3K-Akt and MAPK/ERK Signaling in a Middle-Aged AβPP/PS1 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1157-73, 2016.
A. Zhu, Wu, Z., Zhong, X., Ni, J., Li, Y., Meng, J., Du, C., Zhao, X., Nakanishi, H., and Wu, S., “Brazilian Green Propolis Prevents Cognitive Decline into Mild Cognitive Impairment in Elderly People Living at High Altitude.”, J Alzheimers Dis, vol. 63, no. 2, pp. 551-560, 2018.
H. - F. Wang, Wan, Y., Hao, X. - K., Cao, L., Zhu, X. - C., Jiang, T., Tan, M. - S., Tan, L., Zhang, D. - Q., Tan, L., and Yu, J. - T., “Bridging Integrator 1 (BIN1) Genotypes Mediate Alzheimer's Disease Risk by Altering Neuronal Degeneration.”, J Alzheimers Dis, vol. 52, no. 1, pp. 179-90, 2016.
R. E. Curiel, Crocco, E., Rosado, M., Duara, R., Greig, M. T., Raffo, A., and Loewenstein, D. A., “A Brief Computerized Paired Associate Test for the Detection of Mild Cognitive Impairment in Community-Dwelling Older Adults.”, J Alzheimers Dis, vol. 54, no. 2, pp. 793-9, 2016.
L. De Beaumont, Pelleieux, S., Lamarre-Théroux, L., Dea, D., and Poirier, J., “Butyrylcholinesterase K and Apolipoprotein E-ɛ4 Reduce the Age of Onset of Alzheimer's Disease, Accelerate Cognitive Decline, and Modulate Donepezil Response in Mild Cognitively Impaired Subjects.”, J Alzheimers Dis, vol. 54, no. 3, pp. 913-922, 2016.
L. De Beaumont, Pelleieux, S., Lamarre-Théroux, L., Dea, D., and Poirier, J., “Butyrylcholinesterase K and Apolipoprotein E-ɛ4 Reduce the Age of Onset of Alzheimer's Disease, Accelerate Cognitive Decline, and Modulate Donepezil Response in Mild Cognitively Impaired Subjects.”, J Alzheimers Dis, vol. 54, no. 3, pp. 913-922, 2016.
C
A. R. Haeusler, Donnelly, C. J., Periz, G., Simko, E. A. J., Shaw, P. G., Kim, M. - S., Maragakis, N. J., Troncoso, J. C., Pandey, A., Sattler, R., Rothstein, J. D., and Wang, J., “C9orf72 nucleotide repeat structures initiate molecular cascades of disease.”, Nature, vol. 507, no. 7491, pp. 195-200, 2014.
P. Maresova, Dolejš, J., and Kuca, K., “Call for a Uniform Strategy of Collecting Alzheimer's Disease Costs: A Review and Meta-Analysis.”, J Alzheimers Dis, vol. 63, no. 1, pp. 227-238, 2018.
F. P. M. Oliveira, Moreira, A. Paula, de Mendonça, A., Verdelho, A., Xavier, C., Barroca, D., Rio, J., Cardoso, E., Cruz, Â., Abrunhosa, A., and Castelo-Branco, M., “Can 11C-PiB-PET Relative Delivery R1 or 11C-PiB-PET Perfusion Replace 18F-FDG-PET in the Assessment of Brain Neurodegeneration?”, J Alzheimers Dis, vol. 65, no. 1, pp. 89-97, 2018.

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