Biblio
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Author Title Type [ Year] Filters: Keyword is Alzheimer Disease [Clear All Filters]
“Associations between Use of Specific Analgesics and Concentrations of Amyloid-β 42 or Phospho-Tau in Regions of Human Cerebral Cortex.”, J Alzheimers Dis, vol. 61, no. 2, pp. 653-662, 2018.
, “Associations of Dietary Protein and Fiber Intake with Brain and Blood Amyloid-β.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1589-1598, 2018.
, “Biallelic Loss of Function of SORL1 in an Early Onset Alzheimer's Disease Patient.”, J Alzheimers Dis, vol. 62, no. 2, pp. 821-831, 2018.
, “Cardiorespiratory Fitness and White Matter Neuronal Fiber Integrity in Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 61, no. 2, pp. 729-739, 2018.
, “Cerebrospinal Fluid Amyloid-β Levels are Increased in Patients with Insomnia.”, J Alzheimers Dis, vol. 61, no. 2, pp. 645-651, 2018.
, “Cerebrospinal Fluid C-C Motif Chemokine Ligand 2 Correlates with Brain Atrophy and Cognitive Impairment in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 581-588, 2018.
, “Clinical Trials for Disease-Modifying Therapies in Alzheimer's Disease: A Primer, Lessons Learned, and a Blueprint for the Future.”, J Alzheimers Dis, vol. 64, no. s1, pp. S3-S22, 2018.
, “A Clinically-Translatable Machine Learning Algorithm for the Prediction of Alzheimer's Disease Conversion in Individuals with Mild and Premild Cognitive Impairment.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1555-1573, 2018.
, “Co-Expression of Glia Maturation Factor and Apolipoprotein E4 in Alzheimer's Disease Brain.”, J Alzheimers Dis, vol. 61, no. 2, pp. 553-560, 2018.
, “Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.”, J Alzheimers Dis, vol. 61, no. 1, pp. 265-281, 2018.
, “Cognitive Variability Predicts Incident Alzheimer's Disease and Mild Cognitive Impairment Comparable to a Cerebrospinal Fluid Biomarker.”, J Alzheimers Dis, vol. 61, no. 1, pp. 79-89, 2018.
, “Combined Socio-Behavioral Evaluation Improves the Differential Diagnosis Between the Behavioral Variant of Frontotemporal Dementia and Alzheimer's Disease: In Search of Neuropsychological Markers.”, J Alzheimers Dis, vol. 61, no. 2, pp. 761-772, 2018.
, “Comparison between FCSRT and LASSI-L to Detect Early Stage Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 103-111, 2018.
, “Complexity and Selectivity of γ-Secretase Cleavage on Multiple Substrates: Consequences in Alzheimer's Disease and Cancer.”, J Alzheimers Dis, vol. 61, no. 1, pp. 1-15, 2018.
, “Concordance Between Cerebrospinal Fluid Biomarkers with Alzheimer's Disease Pathology Between Three Independent Assay Platforms.”, J Alzheimers Dis, vol. 61, no. 1, pp. 169-183, 2018.
, “Deep Brain Stimulation of Frontal Lobe Networks to Treat Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 2, pp. 621-633, 2018.
, “Development of a High-Sensitivity Method for the Measurement of Human Nasal Aβ42, Tau, and Phosphorylated Tau.”, J Alzheimers Dis, vol. 62, no. 2, pp. 737-744, 2018.
, “The Diagnostic and Prognostic Value of a Dual-Tasking Paradigm in a Memory Clinic.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1189-1199, 2018.
, “Differences in Synaptic Dysfunction Between rTg4510 and APP/PS1 Mouse Models of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 195-208, 2018.
, “Differential Pattern of Phospholipid Profile in the Temporal Cortex from E280A-Familiar and Sporadic Alzheimer's Disease Brains.”, J Alzheimers Dis, vol. 61, no. 1, pp. 209-219, 2018.
, “Does Fatty Acid Composition in Subcutaneous Adipose Tissue Differ between Patients with Alzheimer's Disease and Cohabiting Proxies?”, J Alzheimers Dis, vol. 61, no. 2, pp. 515-519, 2018.
, “Does the Genetic Feature of the Chinese Tree Shrew (Tupaia belangeri chinensis) Support Its Potential as a Viable Model for Alzheimer's Disease Research?”, J Alzheimers Dis, vol. 61, no. 3, pp. 1015-1028, 2018.
, “Dysregulation and Dislocation of SFPQ Disturbed DNA Organization in Alzheimer's Disease and Frontotemporal Dementia.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1311-1321, 2018.
, “Early and Persistent O-GlcNAc Protein Modification in the Streptozotocin Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 237-249, 2018.
, “Early Stage Alterations in CA1 Extracellular Region Proteins Indicate Dysregulation of IL6 and Iron Homeostasis in the 5XFAD Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1399-1410, 2018.
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