Export 221 results:
Author [ Title(Desc)] Type Year
Filters: First Letter Of Title is E  [Clear All Filters]
A B C D E F G H I J K L M N O P Q R S T U V W X Y Z 
G. Giaccone, The Existence of Primary Age-Related Tauopathy Suggests that not all the Cases with Early Braak Stages of Neurofibrillary Pathology are Alzheimer's Disease., J Alzheimers Dis, 2015.
Y. Lian, Xiao, L. Dongxia, Zeng, F., Wu, X., Wang, Z., and Ren, H., The Experiences of People with Dementia and Their Caregivers in Dementia Diagnosis., J Alzheimers Dis, vol. 59, no. 4, pp. 1203-1211, 2017.
A. Katrine Bergland, Proitsi, P., Kirsebom, B. - E., Soennesyn, H., Hye, A., Larsen, A. Inge, Xu, J., Legido-Quigley, C., Rajendran, L., Fladby, T., and Aarsland, D., Exploration of Plasma Lipids in Mild Cognitive Impairment due to Alzheimer's Disease., J Alzheimers Dis, vol. 77, no. 3, pp. 1117-1127, 2020.
S. Chamberlain, Gabriel, H., Strittmatter, W., and Didsbury, J., An Exploratory Phase IIa Study of the PPAR delta/gamma Agonist T3D-959 Assessing Metabolic and Cognitive Function in Subjects with Mild to Moderate Alzheimer's Disease., J Alzheimers Dis, vol. 73, no. 3, pp. 1085-1103, 2020.
A. Stevenson, Lopez, D., Khoo, P., Kalaria, R. N., and Mukaetova-Ladinska, E. B., Exploring Erythrocytes as Blood Biomarkers for Alzheimer's Disease., J Alzheimers Dis, vol. 60, no. 3, pp. 845-857, 2017.
L. Gu and Zhang, Z., Exploring Potential Electrophysiological Biomarkers in Mild Cognitive Impairment: A Systematic Review and Meta-Analysis of Event-Related Potential Studies., J Alzheimers Dis, vol. 58, no. 4, pp. 1283-1292, 2017.
V. E Daniel, Kleiman, M. J., and Galvin, J. E., Exploring Reasons for Differential Vulnerability and Alzheimer's Disease Risk in Racial and Ethnic Minorities., J Alzheimers Dis, vol. 91, no. 1, pp. 495-506, 2023.
D. Kontaxopoulou, Beratis, I. N., Fragkiadaki, S., Pavlou, D., Andronas, N., Yannis, G., Economou, A., Papanicolaou, A. C., and Papageorgiou, S. G., Exploring the Profile of Incidental Memory in Patients with Amnestic Mild Cognitive Impairment and Mild Alzheimer's Disease., J Alzheimers Dis, vol. 65, no. 2, pp. 617-627, 2018.
V. Giannouli, Stamovlasis, D., and Tsolaki, M., Exploring the Role of Cognitive Factors in a New Instrument for Elders' Financial Capacity Assessment., J Alzheimers Dis, vol. 62, no. 4, pp. 1579-1594, 2018.
C. Tammy Kim, Myung, W., Lewis, M., Lee, H., Kim, S. Estella, Lee, K., Lee, C., Choi, J., Kim, H., Carroll, B. J., and Kim, D. Kwan, Exposure to General Anesthesia and Risk of Dementia: A Nationwide Population-Based Cohort Study., J Alzheimers Dis, vol. 63, no. 1, pp. 395-405, 2018.
S. L. Gray, Anderson, M. L., Hanlon, J. T., Dublin, S., Walker, R. L., Hubbard, R. A., Yu, O., Montine, T. J., Crane, P. K., Sonnen, J. A., C Keene, D., and Larson, E. B., Exposure to Strong Anticholinergic Medications and Dementia-Related Neuropathology in a Community-Based Autopsy Cohort., J Alzheimers Dis, vol. 65, no. 2, pp. 607-616, 2018.
R. Iban-Arias, Trageser, K. J., Yang, E. - J., Griggs, E., Radu, A., Naughton, S., Rahim, M. Al, Tatsunori, O., Raval, U., Palmieri, J., Huang, Z., Chen, L. - C., and Pasinetti, G. Maria, Exposure to World Trade Center Dust Exacerbates Cognitive Impairment and Evokes a Central and Peripheral Pro-Inflammatory Transcriptional Profile in an Animal Model of Alzheimer's Disease., J Alzheimers Dis, vol. 91, no. 2, pp. 779-794, 2023.
F. J. Castora, Kerns, K. A., Pflanzer, H. K., Hitefield, N. L., Gershon, B., Shugoll, J., Shelton, M., and Coleman, R. A., Expression Changes in Mitochondrial Genes Affecting Mitochondrial Morphology, Transmembrane Potential, Fragmentation, Amyloidosis, and Neuronal Cell Death Found in Brains of Alzheimer's Disease Patients., J Alzheimers Dis, vol. 90, no. 1, pp. 119-137, 2022.
F. M. Torres-Cruz, Rodríguez-Cruz, F., Escobar-Herrera, J., Barragán-Andrade, N., Basurto-Islas, G., Ripova, D., Avila, J., and Garcia-Sierra, F., Expression of Tau Produces Aberrant Plasma Membrane Blebbing in Glial Cells Through RhoA-ROCK-Dependent F-Actin Remodeling., J Alzheimers Dis, vol. 52, no. 2, pp. 463-82, 2016.
J. E. Jakobsen, Johansen, M. G., Schmidt, M., Liu, Y., Li, R., Callesen, H., Melnikova, M., Habekost, M., Matrone, C., Bouter, Y., Bayer, T. A., Nielsen, A. Lade, Duthie, M., Fraser, P. E., Holm, I. E., and Jørgensen, A. Lund, Expression of the Alzheimer's Disease Mutations AβPP695sw and PSEN1M146I in Double-Transgenic Göttingen Minipigs., J Alzheimers Dis, vol. 53, no. 4, pp. 1617-30, 2016.
J. Eugenín, Vecchiola, A., Murgas, P., Arroyo, P., Cornejo, F., and von Bernhardi, R., Expression Pattern of Scavenger Receptors and Amyloid-β Phagocytosis of Astrocytes and Microglia in Culture are Modified by Acidosis: Implications for Alzheimer's Disease., J Alzheimers Dis, vol. 53, no. 3, pp. 857-73, 2016.
M. Reale, D'Angelo, C., Costantini, E., Di Nicola, M., Yarla, N. Sastry, Kamal, M. Amjad, Salvador, N., and Perry, G., Expression Profiling of Cytokine, Cholinergic Markers, and Amyloid-β Deposition in the APPSWE/PS1dE9 Mouse Model of Alzheimer's Disease Pathology., J Alzheimers Dis, vol. 62, no. 1, pp. 467-476, 2018.
M. N. Best, Lim, Y., Ferenc, N. N., Kim, N., Min, L., Wang, D. Bigler, Sharifi, K., Wasserman, A. E., McTavish, S. A., Siller, K. H., Jones, M. K., Jenkins, P. M., Mandell, J. W., and Bloom, G. S., Extracellular Tau Oligomers Damage the Axon Initial Segment., J Alzheimers Dis, vol. 93, no. 4, pp. 1425-1441, 2023.
E. Swanson, Breckenridge, L., McMahon, L., Som, S., McConnell, I., and Bloom, G. S., Extracellular Tau Oligomers Induce Invasion of Endogenous Tau into the Somatodendritic Compartment and Axonal Transport Dysfunction., J Alzheimers Dis, vol. 58, no. 3, pp. 803-820, 2017.
M. Varghese, Santa-Maria, I., Ho, L., Ward, L., Yemul, S., Dubner, L., Księżak-Reding, H., and Pasinetti, G. Maria, Extracellular Tau Paired Helical Filaments Differentially Affect Tau Pathogenic Mechanisms in Mitotic and Post-Mitotic Cells: Implications for Mechanisms of Tau Propagation in the Brain., J Alzheimers Dis, vol. 54, no. 2, pp. 477-96, 2016.
S. Baker, Polanco, J. Carlos, and Götz, J., Extracellular Vesicles Containing P301L Mutant Tau Accelerate Pathological Tau Phosphorylation and Oligomer Formation but Do Not Seed Mature Neurofibrillary Tangles in ALZ17 Mice., J Alzheimers Dis, vol. 54, no. 3, pp. 1207-1217, 2016.