Biblio
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Author [ Title] Type Year Filters: First Letter Of Title is E [Clear All Filters]
“Exercise-Related Physical Activity Relates to Brain Volumes in 10,125 Individuals.”, J Alzheimers Dis, vol. 97, no. 2, pp. 829-839, 2023.
, “The Existence of Primary Age-Related Tauopathy Suggests that not all the Cases with Early Braak Stages of Neurofibrillary Pathology are Alzheimer's Disease.”, J Alzheimers Dis, 2015.
, “The Experiences of People with Dementia and Their Caregivers in Dementia Diagnosis.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1203-1211, 2017.
, “Exploration of Plasma Lipids in Mild Cognitive Impairment due to Alzheimer's Disease.”, J Alzheimers Dis, vol. 77, no. 3, pp. 1117-1127, 2020.
, “An Exploratory Phase IIa Study of the PPAR delta/gamma Agonist T3D-959 Assessing Metabolic and Cognitive Function in Subjects with Mild to Moderate Alzheimer's Disease.”, J Alzheimers Dis, vol. 73, no. 3, pp. 1085-1103, 2020.
, “Exploring Erythrocytes as Blood Biomarkers for Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 3, pp. 845-857, 2017.
, “Exploring Potential Electrophysiological Biomarkers in Mild Cognitive Impairment: A Systematic Review and Meta-Analysis of Event-Related Potential Studies.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1283-1292, 2017.
, “Exploring Reasons for Differential Vulnerability and Alzheimer's Disease Risk in Racial and Ethnic Minorities.”, J Alzheimers Dis, vol. 91, no. 1, pp. 495-506, 2023.
, “Exploring the Genetic Heterogeneity of Alzheimer's Disease: Evidence for Genetic Subtypes.”, J Alzheimers Dis, vol. 100, no. 4, pp. 1209-1226, 2024.
, “Exploring the Profile of Incidental Memory in Patients with Amnestic Mild Cognitive Impairment and Mild Alzheimer's Disease.”, J Alzheimers Dis, vol. 65, no. 2, pp. 617-627, 2018.
, “Exploring the Role of Cognitive Factors in a New Instrument for Elders' Financial Capacity Assessment.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1579-1594, 2018.
, “Exposure to General Anesthesia and Risk of Dementia: A Nationwide Population-Based Cohort Study.”, J Alzheimers Dis, vol. 63, no. 1, pp. 395-405, 2018.
, “Exposure to Strong Anticholinergic Medications and Dementia-Related Neuropathology in a Community-Based Autopsy Cohort.”, J Alzheimers Dis, vol. 65, no. 2, pp. 607-616, 2018.
, “Exposure to World Trade Center Dust Exacerbates Cognitive Impairment and Evokes a Central and Peripheral Pro-Inflammatory Transcriptional Profile in an Animal Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 91, no. 2, pp. 779-794, 2023.
, “Expression Changes in Mitochondrial Genes Affecting Mitochondrial Morphology, Transmembrane Potential, Fragmentation, Amyloidosis, and Neuronal Cell Death Found in Brains of Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 90, no. 1, pp. 119-137, 2022.
, “Expression of Tau Produces Aberrant Plasma Membrane Blebbing in Glial Cells Through RhoA-ROCK-Dependent F-Actin Remodeling.”, J Alzheimers Dis, vol. 52, no. 2, pp. 463-82, 2016.
, “Expression of the Alzheimer's Disease Mutations AβPP695sw and PSEN1M146I in Double-Transgenic Göttingen Minipigs.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1617-30, 2016.
, “Expression Pattern of Scavenger Receptors and Amyloid-β Phagocytosis of Astrocytes and Microglia in Culture are Modified by Acidosis: Implications for Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 3, pp. 857-73, 2016.
, “Expression Profiling of Cytokine, Cholinergic Markers, and Amyloid-β Deposition in the APPSWE/PS1dE9 Mouse Model of Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 62, no. 1, pp. 467-476, 2018.
, “Extracellular Tau Oligomers Damage the Axon Initial Segment.”, J Alzheimers Dis, vol. 93, no. 4, pp. 1425-1441, 2023.
, “Extracellular Tau Oligomers Induce Invasion of Endogenous Tau into the Somatodendritic Compartment and Axonal Transport Dysfunction.”, J Alzheimers Dis, vol. 58, no. 3, pp. 803-820, 2017.
, “Extracellular Tau Paired Helical Filaments Differentially Affect Tau Pathogenic Mechanisms in Mitotic and Post-Mitotic Cells: Implications for Mechanisms of Tau Propagation in the Brain.”, J Alzheimers Dis, vol. 54, no. 2, pp. 477-96, 2016.
, “Extracellular Vesicles Containing P301L Mutant Tau Accelerate Pathological Tau Phosphorylation and Oligomer Formation but Do Not Seed Mature Neurofibrillary Tangles in ALZ17 Mice.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1207-1217, 2016.
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