Biblio
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“Higher Blood Vitamin C Levels are Associated with Reduction of Apolipoprotein E E4-related Risks of Cognitive Decline in Women: The Nakajima Study.”, J Alzheimers Dis, vol. 63, no. 4, pp. 1289-1297, 2018.
, “Higher Blood Vitamin C Levels are Associated with Reduction of Apolipoprotein E E4-related Risks of Cognitive Decline in Women: The Nakajima Study.”, J Alzheimers Dis, vol. 63, no. 4, pp. 1289-1297, 2018.
, “Hippocampal Subfield Volumetry: Differential Pattern of Atrophy in Different Forms of Genetic Frontotemporal Dementia.”, J Alzheimers Dis, vol. 64, no. 2, pp. 497-504, 2018.
, “Homocysteine and Cerebral Atrophy: The Epidemiology of Dementia in Singapore Study.”, J Alzheimers Dis, vol. 62, no. 2, pp. 877-885, 2018.
, “Impact of Untimely Access to Formal Care on Costs and Quality of Life in Community Dwelling People with Dementia.”, J Alzheimers Dis, vol. 66, no. 3, pp. 1165-1174, 2018.
, “In vivo Depiction of α7 Nicotinic Receptor Loss for Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1355-1365, 2018.
, “In Vivo Visualization of Tau Accumulation, Microglial Activation, and Brain Atrophy in a Mouse Model of Tauopathy rTg4510.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1037-1052, 2018.
, “Involvement of Activation of Asparaginyl Endopeptidase in Tau Hyperphosphorylation in Repetitive Mild Traumatic Brain Injury.”, J Alzheimers Dis, vol. 64, no. 3, pp. 709-722, 2018.
, “Linking Atrial Fibrillation with Alzheimer's Disease: Epidemiological, Pathological, and Mechanistic Evidence.”, J Alzheimers Dis, vol. 62, no. 1, pp. 61-72, 2018.
, “Low Accuracy of Brief Cognitive Tests in Tracking Longitudinal Cognitive Decline in an Asian Elderly Cohort.”, J Alzheimers Dis, vol. 62, no. 1, pp. 409-416, 2018.
, “Mechanism of Tau Hyperphosphorylation Involving Lysosomal Enzyme Asparagine Endopeptidase in a Mouse Model of Brain Ischemia.”, J Alzheimers Dis, vol. 63, no. 2, pp. 821-833, 2018.
, “Mental States in Moving Shapes: Distinct Cortical and Subcortical Contributions to Theory of Mind Impairments in Dementia.”, J Alzheimers Dis, vol. 61, no. 2, pp. 521-535, 2018.
, “Metabolic Syndrome, Brain Insulin Resistance, and Alzheimer's Disease: Thioredoxin Interacting Protein (TXNIP) and Inflammasome as Core Amplifiers.”, J Alzheimers Dis, vol. 66, no. 3, pp. 857-885, 2018.
, “Microglial Activation During Pathogenesis of Tauopathy in rTg4510 Mice: Implications for the Early Diagnosis of Tauopathy.”, J Alzheimers Dis, vol. 64, no. s1, pp. S353-S359, 2018.
, “The Need to Separate Chronic Traumatic Encephalopathy Neuropathology from Clinical Features.”, J Alzheimers Dis, vol. 61, no. 1, pp. 17-28, 2018.
, “Neural Correlates of Sleep Disturbance in Alzheimer's Disease: Role of the Precuneus in Sleep Disturbance.”, J Alzheimers Dis, vol. 63, no. 3, pp. 957-964, 2018.
, “Neuro-Immuno-Gene- and Genome-Editing-Therapy for Alzheimer's Disease: Are We There Yet?”, J Alzheimers Dis, vol. 65, no. 2, pp. 321-344, 2018.
, “Neuroprotective Effects of Ferruginol, Jatrophone, and Junicedric Acid Against Amyloid-β Injury in Hippocampal Neurons.”, J Alzheimers Dis, vol. 63, no. 2, pp. 705-723, 2018.
, “New Insights into the Spontaneous Human Alzheimer's Disease-Like Model Octodon degus: Unraveling Amyloid-β Peptide Aggregation and Age-Related Amyloid Pathology.”, J Alzheimers Dis, vol. 66, no. 3, pp. 1145-1163, 2018.
, “Novel GRN Mutations in Alzheimer's Disease and Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1683-1689, 2018.
, “A Novel Therapeutic Approach to Treat Alzheimer's Disease by Neurotrophic Support During the Period of Synaptic Compensation.”, J Alzheimers Dis, vol. 62, no. 3, pp. 1211-1218, 2018.
, “Nutrition: Review on the Possible Treatment for Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 867-883, 2018.
, “Overactive BRCA1 Affects Presenilin 1 in Induced Pluripotent Stem Cell-Derived Neurons in Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 1, pp. 175-202, 2018.
, “Predicting Imminent Progression to Clinically Significant Memory Decline Using Volumetric MRI and FDG PET.”, J Alzheimers Dis, 2018.
, “Preoperative Phosphorylated Tau Concentration in the Cerebrospinal Fluid Can Predict Cognitive Function Three Years after Shunt Surgery in Patients with Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 66, no. 1, pp. 319-331, 2018.
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