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“"Captive by the Uncertainty"-Experiences with Anticipatory Guidance for People Living with Dementia and Their Caregivers at a Specialty Dementia Clinic.”, J Alzheimers Dis, vol. 86, no. 2, pp. 787-800, 2022.
, “Longitudinal Changes in Functional Brain Connectivity Predicts Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 377-89, 2016.
, “Longitudinal Changes in Functional Brain Connectivity Predicts Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 377-89, 2016.
, “Profiling of Specific Gene Expression Pathways in Peripheral Cells from Prodromal Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1289-1294, 2018.
, “Profiling of Specific Gene Expression Pathways in Peripheral Cells from Prodromal Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1289-1294, 2018.
, “SPARCL1 Accelerates Symptom Onset in Alzheimer's Disease and Influences Brain Structure and Function During Aging.”, J Alzheimers Dis, vol. 61, no. 1, pp. 401-414, 2018.
, “SPARCL1 Accelerates Symptom Onset in Alzheimer's Disease and Influences Brain Structure and Function During Aging.”, J Alzheimers Dis, vol. 61, no. 1, pp. 401-414, 2018.
, “SPARCL1 Accelerates Symptom Onset in Alzheimer's Disease and Influences Brain Structure and Function During Aging.”, J Alzheimers Dis, vol. 61, no. 1, pp. 401-414, 2018.
, “Dogs with Cognitive Dysfunction as a Spontaneous Model for Early Alzheimer's Disease: A Translational Study of Neuropathological and Inflammatory Markers.”, J Alzheimers Dis, vol. 52, no. 2, pp. 433-49, 2016.
, “Dogs with Cognitive Dysfunction as a Spontaneous Model for Early Alzheimer's Disease: A Translational Study of Neuropathological and Inflammatory Markers.”, J Alzheimers Dis, vol. 52, no. 2, pp. 433-49, 2016.
, “Increase of α-Secretase ADAM10 in Platelets Along Cognitively Healthy Aging.”, J Alzheimers Dis, vol. 50, no. 3, pp. 817-26, 2016.
, “Dietary high cholesterol and trace metals in the drinking water increase levels of ABCA1 in the rabbit hippocampus and temporal cortex.”, J Alzheimers Dis, vol. 49, no. 1, pp. 201-9, 2016.
, “Dietary high cholesterol and trace metals in the drinking water increase levels of ABCA1 in the rabbit hippocampus and temporal cortex.”, J Alzheimers Dis, vol. 49, no. 1, pp. 201-9, 2016.
, “N-truncated Aβ2-X starting with position two in sporadic Alzheimer's disease cases and two Alzheimer mouse models.”, J Alzheimers Dis, vol. 49, no. 1, pp. 101-10, 2016.
, “The Efficacy and Safety of Physiotherapy in People with Dementia: A Systematic Review.”, J Alzheimers Dis, vol. 94, no. 3, pp. 909-917, 2023.
, “Longitudinal Protein Changes in Blood Plasma Associated with the Rate of Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1105-14, 2016.
, “Longitudinal Protein Changes in Blood Plasma Associated with the Rate of Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1105-14, 2016.
, “Clinical and Demographic Factors Associated with the Cognitive and Emotional Efficacy of Regular Musical Activities in Dementia.”, J Alzheimers Dis, vol. 49, no. 3, pp. 767-81, 2016.
, “Clinical and Demographic Factors Associated with the Cognitive and Emotional Efficacy of Regular Musical Activities in Dementia.”, J Alzheimers Dis, vol. 49, no. 3, pp. 767-81, 2016.
, “Clinical and Demographic Factors Associated with the Cognitive and Emotional Efficacy of Regular Musical Activities in Dementia.”, J Alzheimers Dis, vol. 49, no. 3, pp. 767-81, 2016.
, “Altered Expression of Circulating Cdc42 in Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1477-1483, 2018.
, “Altered Expression of Circulating Cdc42 in Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1477-1483, 2018.
, “A Network of Genetic Effects on Non-Demented Cognitive Aging: Alzheimer's Genetic Risk (CLU + CR1 + PICALM) Intensifies Cognitive Aging Genetic Risk (COMT + BDNF) Selectively for APOEɛ4 Carriers.”, J Alzheimers Dis, vol. 62, no. 2, pp. 887-900, 2018.
, “A Network of Genetic Effects on Non-Demented Cognitive Aging: Alzheimer's Genetic Risk (CLU + CR1 + PICALM) Intensifies Cognitive Aging Genetic Risk (COMT + BDNF) Selectively for APOEɛ4 Carriers.”, J Alzheimers Dis, vol. 62, no. 2, pp. 887-900, 2018.
, “A Network of Genetic Effects on Non-Demented Cognitive Aging: Alzheimer's Genetic Risk (CLU + CR1 + PICALM) Intensifies Cognitive Aging Genetic Risk (COMT + BDNF) Selectively for APOEɛ4 Carriers.”, J Alzheimers Dis, vol. 62, no. 2, pp. 887-900, 2018.
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