Biblio
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“Cognitive Impairment Precedes and Predicts Functional Impairment in Mild Alzheimer's Disease.”, J Alzheimers Dis, vol. 47, no. 1, pp. 205-14, 2015.
, “Depression and Suicidal Ideation During Two Psychosocial Treatments in Older Adults with Major Depression and Dementia.”, J Alzheimers Dis, vol. 48, no. 2, pp. 453-62, 2015.
, “Depressive Symptoms and Biomarkers of Alzheimer's Disease in Cognitively Normal Older Adults.”, J Alzheimers Dis, 2015.
, “Diabetes is Associated with Worse Executive Function in Both Eastern and Western Populations: Shanghai Aging Study and Mayo Clinic Study of Aging.”, J Alzheimers Dis, vol. 47, no. 1, pp. 167-76, 2015.
, “Identification of Novel Tau Interactions with Endoplasmic Reticulum Proteins in Alzheimer's Disease Brain.”, J Alzheimers Dis, 2015.
, “Mapping the Progression of Atrophy in Early- and Late-Onset Alzheimer's Disease.”, J Alzheimers Dis, 2015.
, “The Multi-Target Drug M30 Shows Pro-Cognitive and Anti-Inflammatory Effects in a Rat Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 47, no. 2, pp. 373-83, 2015.
, “A Novel Plasma Based Biomarker of Alzheimer's Disease.”, J Alzheimers Dis, vol. 47, no. 3, pp. 761-71, 2015.
, “ABCA7 Genotypes Confer Alzheimer's Disease Risk by Modulating Amyloid-β Pathology.”, J Alzheimers Dis, vol. 52, no. 2, pp. 693-703, 2016.
, “Activation of p53 in Down Syndrome and in the Ts65Dn Mouse Brain is Associated with a Pro-Apoptotic Phenotype.”, J Alzheimers Dis, vol. 52, no. 1, pp. 359-71, 2016.
, “Active Cigarette Smoking in Cognitively-Normal Elders and Probable Alzheimer's Disease is Associated with Elevated Cerebrospinal Fluid Oxidative Stress Biomarkers.”, J Alzheimers Dis, vol. 54, no. 1, pp. 99-107, 2016.
, “Active Cigarette Smoking in Cognitively-Normal Elders and Probable Alzheimer's Disease is Associated with Elevated Cerebrospinal Fluid Oxidative Stress Biomarkers.”, J Alzheimers Dis, vol. 54, no. 1, pp. 99-107, 2016.
, “Activities of Daily Living and Depressive Symptoms in Patients with Subjective Cognitive Decline, Mild Cognitive Impairment, and Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1043-50, 2016.
, “Added Prognostic Value of Cerebrospinal Fluid Biomarkers in Predicting Decline in Memory Clinic Patients in a Prospective Cohort.”, J Alzheimers Dis, vol. 52, no. 3, pp. 875-85, 2016.
, “Adenosine Type A2A Receptor in Peripheral Cell from Patients with Alzheimer's Disease, Vascular Dementia, and Idiopathic Normal Pressure Hydrocephalus: A New/Old Potential Target.”, J Alzheimers Dis, vol. 54, no. 2, pp. 417-25, 2016.
, “Adenosine Type A2A Receptor in Peripheral Cell from Patients with Alzheimer's Disease, Vascular Dementia, and Idiopathic Normal Pressure Hydrocephalus: A New/Old Potential Target.”, J Alzheimers Dis, vol. 54, no. 2, pp. 417-25, 2016.
, “Age and its association with low insulin and high amyloid-β peptides in blood.”, J Alzheimers Dis, vol. 49, no. 1, pp. 129-37, 2016.
, “Age-Dependent Regulation of the Blood-Brain Barrier Influx/Efflux Equilibrium of Amyloid-β Peptide in a Mouse Model of Alzheimer's Disease (3xTg-AD).”, J Alzheimers Dis, vol. 49, no. 2, pp. 287-300, 2016.
, “Age-Related Changes in the Synaptic Density of Amyloid-β Protein Precursor and Secretases in the Human Cerebral Cortex.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1209-14, 2016.
, “Alcohol Consumption and Incident Dementia: Evidence from the Sydney Memory and Ageing Study.”, J Alzheimers Dis, vol. 52, no. 2, pp. 529-38, 2016.
, “Alterations in the Levels of Amyloid-β, Phospholipid Hydroperoxide, and Plasmalogen in the Blood of Patients with Alzheimer's Disease: Possible Interactions between Amyloid-β and These Lipids.”, J Alzheimers Dis, vol. 50, no. 2, pp. 527-37, 2016.
, “Altered Topology in Information Processing of a Narrated Story in Older Adults with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 53, no. 2, pp. 517-33, 2016.
, “Alzheimer's Disease: Assessing the Role of Spirochetes, Biofilms, the Immune System, and Amyloid-β with Regard to Potential Treatment and Prevention.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1271-6, 2016.
, “Amyloid-β Increases Activity of Proteasomes Capped with 19S and 11S Regulators.”, J Alzheimers Dis, vol. 54, no. 2, pp. 763-76, 2016.
, “Amyloid-β Increases Activity of Proteasomes Capped with 19S and 11S Regulators.”, J Alzheimers Dis, vol. 54, no. 2, pp. 763-76, 2016.
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